Open access peer-reviewed Edited Volume

Trending Topics in Multiple Sclerosis

Multiple sclerosis (MS) is a chronic inflammatory disease characterized by progressive demyelination and neurodegeneration of the central nervous system (CNS), constituting the most common demyelinating disease of the CNS in humans. Although intensive research over many decades has unveiled many pathophysiological mechanisms in the development of MS, the cause is still unknown. Nevertheless, it does seem clear that genetic susceptibility and environmental factors play crucial roles. Trending Topics in Multiple Sclerosis is a book that provides an insight into some of the main problems currently debated in this area of research, focusing on topics that deal with genetic and environmental risk factors, pathophysiological mechanisms, neurocognitive findings, and neuroprotective strategies.

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Trending Topics in Multiple SclerosisEdited by Alina Gonzalez-Quevedo

Published: September 8th 2016

DOI: 10.5772/61417

ISBN: 978-953-51-2657-7

Print ISBN: 978-953-51-2656-0

eBook (PDF) ISBN: 978-953-51-7310-6

Copyright year: 2016

Books open for chapter submissions

20942 Total Chapter Downloads

13 Crossref Citations

8 Web of Science Citations

45 Dimensions Citations

chaptersDownloads

Open access peer-reviewed

1. Genetic and Biochemical Factors Related to the Risk and Disability Progression in Multiple Sclerosis

By Daniel Čierny, Jozef Michalik, Ema Kantorová, Egon Kurča and Ján Lehotský

1452

Open access peer-reviewed

2. The Gateway Reflex, a Novel Neuro‐immune Interaction, is Critical for the Development of Mouse Multiple Sclerosis (MS) Models

By Daisuke Kamimura, Yasunobu Arima, Andrea Stofkova, Naoki Nishikawa, Kotaro Higuchi, Takuto Ohki and Masaaki Murakami

1399

Open access peer-reviewed

3. Intrathecal Immunoglobulin Synthesis in MS—A Complete Reappraisal

By Mickael Bonnan

2889

Open access peer-reviewed

4. Autoimmune Processes in Multiple Sclerosis: Production of Harmful Catalytic Antibodies Associated with Significant Changes in the Hematopoietic Stem Cell Differentiation and Proliferation

By Georgy A. Nevinsky

1214

Open access peer-reviewed

5. Multiple Sclerosis and Its Relationship with Oxidative Stress, Glutathione Redox System, ATPase System, and Membrane Fluidity

By Genaro G. Ortiz, Fermín P. Pacheco‐Moisés, Erandis D. Torres‐ Sánchez, Tanya E. Sorto‐Gómez, Mario Mireles‐Ramírez, Alfredo León‐Gil, Héctor González‐Usigli, Luis J. Flores‐Alvarado, Erika D. González‐Renovato, Angelica L. Sánchez‐López, Margarita Cid‐ Hernández and Irma E. Velázquez‐Brizuela

1720

Open access peer-reviewed

6. Pediatric Multiple Sclerosis

By Ozgul Ekmekci

1474

Open access peer-reviewed

7. Association Between Multiple Sclerosis Risk and Human Immunodeficiency Virus Infection: Insights and Challenges

By Ehtishamul Haq, Insha Zahoor and Mushfiquddin Khan

1695

Open access peer-reviewed

8. The Role of Over-Nutrition and Obesity in Multiple Sclerosis

By Ema Kantorová, Egon Kurča, Daniel Čierny, Dušan Dobrota and Štefan Sivák

1352

Open access peer-reviewed

9. Cognitive Deficits and Neuropsychological Assessment in Multiple Sclerosis

By Alena Javůrková, Denisa Zimová, Katarína Tomašovičová and Jaroslava Raudenská

1818

Open access peer-reviewed

10. Social Cognition Impairments in Patients with Multiple Sclerosis and Comparison with Imaging Studies, Disease Duration and Grade of Disability

By Valentina Ignatova, Lyudmila Todorova and Jivko Surchev

1278

Open access peer-reviewed

11. Sex Hormones and Multiple Sclerosis

By Anastasiya G. Trenova

1469

Open access peer-reviewed

12. Neuroprotection and Recovery in Multiple Sclerosis

By Dafin F. Muresanu, Maria Balea, Olivia Rosu, Anca Buzoianu and Dana Slavoaca

1477

Open access peer-reviewed

13. Neuroprotection: A New Therapeutic Approach of Relapsing Remitting Multiple Sclerosis

By Juan Espinosa-Parrilla, Marco Pugliese, Nicole Mahy and Manuel J Rodríguez

1705

Edited Volume and chapters are indexed in

  • Worldcat
  • OpenAIRE
  • Google Scholar
  • AZ ebsco
  • Base
  • CNKI

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