Worldwide prevalence of Behçet’s disease [24].
\\n\\n
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Barely three months into the new year and we are happy to announce a monumental milestone reached - 150 million downloads.
\n\nThis achievement solidifies IntechOpen’s place as a pioneer in Open Access publishing and the home to some of the most relevant scientific research available through Open Access.
\n\nWe are so proud to have worked with so many bright minds throughout the years who have helped us spread knowledge through the power of Open Access and we look forward to continuing to support some of the greatest thinkers of our day.
\n\nThank you for making IntechOpen your place of learning, sharing, and discovery, and here’s to 150 million more!
\n\n\n\n\n'}],latestNews:[{slug:"webinar-introduction-to-open-science-wednesday-18-may-1-pm-cest-20220518",title:"Webinar: Introduction to Open Science | Wednesday 18 May, 1 PM CEST"},{slug:"step-in-the-right-direction-intechopen-launches-a-portfolio-of-open-science-journals-20220414",title:"Step in the Right Direction: IntechOpen Launches a Portfolio of Open Science Journals"},{slug:"let-s-meet-at-london-book-fair-5-7-april-2022-olympia-london-20220321",title:"Let’s meet at London Book Fair, 5-7 April 2022, Olympia London"},{slug:"50-books-published-as-part-of-intechopen-and-knowledge-unlatched-ku-collaboration-20220316",title:"50 Books published as part of IntechOpen and Knowledge Unlatched (KU) Collaboration"},{slug:"intechopen-joins-the-united-nations-sustainable-development-goals-publishers-compact-20221702",title:"IntechOpen joins the United Nations Sustainable Development Goals Publishers Compact"},{slug:"intechopen-signs-exclusive-representation-agreement-with-lsr-libros-servicios-y-representaciones-s-a-de-c-v-20211123",title:"IntechOpen Signs Exclusive Representation Agreement with LSR Libros Servicios y Representaciones S.A. de C.V"},{slug:"intechopen-expands-partnership-with-research4life-20211110",title:"IntechOpen Expands Partnership with Research4Life"},{slug:"introducing-intechopen-book-series-a-new-publishing-format-for-oa-books-20210915",title:"Introducing IntechOpen Book Series - A New Publishing Format for OA Books"}]},book:{item:{type:"book",id:"3279",leadTitle:null,fullTitle:"Sarcoidosis",title:"Sarcoidosis",subtitle:null,reviewType:"peer-reviewed",abstract:"More than 130 years have passed since the first description of sarcoid lesions by Sir Jonathan Hutchinson, but the cause of this systemic granulomatous disease is still unknown. Sarcoidosis remains a somewhat mysterious disease because many patients may enter spontaneous remission but some patients are chronically affected or relapse after intervention. Advances in recent sarcoidosis researches have added new insights to our understanding of complexity of sarcoidosis as introduced in this textbook, which includes a new concept of etiology as an endogenous infection caused by hypersensitivity to indigenous bacteria, updated immunopathogenesis and genetic factors, new directions in therapy, and new diagnostic approaches with EBUS-TBNA and FDG-PET. 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Coordinator for Post Graduate programs of the Faculty of Business (March 2006-April 2014), Chairperson of Center for Globalization and Sustainability Research (CGSR) (March2009-April2014), Multimedia University (MMU), Melaka Campus, Malaysia, member of Research and Development, research grants panel and the Institute of Postgraduate Studies (IPS) Coordination Committee (ICC) MMU. He is currently teaching Advanced Research Methodology, Entrepreneurship and Commercialization, and Economics for Managers at the postgraduate level and economic subjects at the undergraduate level.\nHis research interests include development economics, productivity analysis, knowledge-based economy, productivity and environment (green productivity), Bioeconomy, Islamic finance and microfinance, economic growth, (environment, tourism) and Entrepreneurship. He is the book\\'s author (Green Productivity: Applications in Malaysia’s Manufacturing) in 2012. 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Behçet’s disease (BD) is described as a severely debilitating chronic, recurrent, multisystem vascular inflammatory disorder characterised by the triad of oral and genital ulceration and ocular lesions. This exemplary presentation would have been probably first identified by Hippocrates [1, 2]. Even though Behçet’s disease was first identified by the father of medicine in the fifth century BC, there were no literature representations for this entity till 1930, and then a Greek ophthalmologist Benediktos Adamantiades presented one of his case of relapsing eye lesions associated with genital ulceration and arthritis in a 20-year-old male; since then entity has been added with a synonym “Adamantiades-Behçet’s disease” [3, 4, 5, 6]. Further to its classic expression of orogenital ulceration and ocular lesions, the conditions seem to involve the musculoskeletal system, nervous system, gastrointestinal tract, vascular beds, urogenital tract and cardiopulmonary system, so Behçet’s disease have a high morbidity and mortality particularly in males with early age onset [7, 8]. The self-limiting inflammation and the relapsing episodes of clinical manifestations are the hallmark of Behçet’s disease [9]. Frequency and span of regression are uncertain and show no distinguishable pattern of onset.
\nAlthough Behçet’s disease has a worldwide distribution, it is more rampant in Turkish population, where the prevalence is about 421 cases per 100,000 and 13 to 20 cases per 100,000 population in China, Korea, Japan and the Middle East [10, 11]. Behçet’s disease is most commonly diagnosed in the second and third decades of life especially during the reproductive years and shows a female predilection among Europeans. Behçet’s disease rarely occurs prior to puberty and after the fifth decade of life. Behçet’s disease developing in early age has shown to have severe clinical manifestations and mortality. Familial occurrences have been reported in various literatures but are unusual, and the aetiology of Behçet’s disease remains cryptic [12, 13, 14, 15, 16, 17, 18]. The widely accepted hypothesis is that of an inappropriate inflammatory response provoked by an infectious agent in a genetically liable host [19].
\nDiagnosis of Behçet’s disease is based on the criteria developed by the International study group (ISG) in 1990 and International Criteria for Behçet’s Disease (ICBD) [20]. The ICBD was developed with the intention of bettering the ISG criteria that did not consist of nervous system, vascular and gastrointestinal involvement that are severe but rather infrequent complications of Behçet’s disease. A recent study conducted in the United Kingdom has compared the efficacy of ISG and ICBD diagnostic criteria among their patients with Behçet’s disease and concluded that the sensitivity was higher for ICBD than ISG criteria, but the specificity of ICBD was much lower than the ISG criteria [20, 21, 22].
\nManagement is aimed to alleviate symptoms, resolve inflammation, restrain tissue damage, dwindle frequency and severity and avoid lethal complications. A multitude of treatment modalities have been proposed for managing Behçet’s disease ranging from anti-inflammatory to immunomodulators and biologic monoclonal antibodies. Detrimental ramifications are a main concern since it requires long-term medication [8].
\nBehçet’s disease is observed to have a worldwide distribution. However, it is seen commonly among the inhabitants of the historic Silk Road, which was supposed to be an old trading route that linked Japan and China in the Far East to the Mediterranean Sea, including countries such as Turkey and Iran. Therefore, Behçet’s disease is also known by the synonym “Silk Road disease” [23]. Behçet’s disease has a highest prevalence rate in Turkey, where the estimated prevalence is about 421 per 100,000 population followed by Iran, Israel and Japan [24]. It is rarely reported in African countries and the USA [23] as well as Australia, where the prevalence remains unknown but with an annual incidence of 0.6% (Table 1) [25].
\nPrevalence per one lakh population | \nCountry | \n
---|---|
Turkey | \n421 | \n
Iran | \n80 | \n
Saudi Arabia | \n20 | \n
Iraq | \n17 | \n
Israel | \n15.2 | \n
Japan | \n13.5 | \n
France | \n7.1 | \n
USA | \n5.2 | \n
Sweden | \n4.9 | \n
Germany | \n2.26 | \n
Portugal | \n1.53 | \n
UK | \n0.64 | \n
Worldwide prevalence of Behçet’s disease [24].
Two studies conducted in Korea and Turkey in 2016 and 2017 reported the prevalence of Behçet’s disease. A study conducted in Korea estimated the prevalence of Behçet’s disease, and change in prevalence rate over time from the Korean healthcare big data that consists of medical institution claims data concerning diagnosis, prescription medicines and surgical treatment and represents the total population. The database was reviewed for patients diagnosed to have Behçet’s disease during the year 2011 and 2015. The diagnosis was based on the Korean Standard Classification of Diseases (KCD). The estimated prevalence was about 35.0 per 100,000 population which was a little more than the previously reported prevalence of 30.2 per 100,000 in Korea. The change in prevalence rate was also estimated during the time period of 2011 and 2015 and concluded that there is a creeping escalation in the annual prevalence rate between 2011 and 2015. The prevalence rate was 32.8 per 100,000 in 2011, 34.0 per 100,000 in 2012, 34.6 per 100,000 in 2013, 35.5 per 100,000 in 2014 and 35.7 per 100,000 in 2015. The prevalence would have been more since patients with milder form of the disease have not come for treatment. Females had a higher prevalence, with a female-to-male ratio of 1:0.54 in 2011 and 1:0.56 in 2015 [26].
\nAnother study conducted in Northern Turkey showed a prevalence of Behçet’s disease in 600/100,000 population who have crossed their second decade of life and is considered as the highest prevalence reported in the literature so far [27]. Nonetheless this conclusion seems to be biased since there are methodological errors. Patients were randomly selected from the 52 urban and 33 rural general practices of a city and are invited for an examination and interview by three dermatologists. As per the authors, the invited sample would well represent the whole population since all individuals are recorded in general practices. Eye examination and pathergy test were performed in suspected individuals. Individuals compatible to ISG criteria were diagnosed as Behçet’s disease. However, the total number of individuals who were invited for interview was not mentioned. Authors reported that 2325 of 2428 individuals interviewed were included in the study, of which 1290 were females and 1035 were males. Symptomatic individuals may have responded to the invitation, which might have led to a higher prevalence estimate. Authors have also reported increased prevalence of Behçet’s disease in females (860/100,000) compared to males (140/100,000) (p = 0.022) which does not correlate with the previous studies conducted in Turkey, since they concluded no significant gender predilections [27].
\nA recent meta-analysis that included 45 prevalence studies suggested that even though there was clearly a wide variation across countries, the overall prevalence of Behçet’s disease were 10.3/100,000. The estimated prevalence was 119.8/100,000 for Turkey, 31.8/100,000 for the Middle East, 4.5/100,000 for Asia and 3.3/100,000 for Europe [28]. A meta-regression analysis showed that classification criteria, study period or reference type had hardly any effect and study design was the deciding factor for Behçet’s disease prevalence in these studies. The authors have also suggested that true geographic variations and different study methods are the possible factors responsible for the large variation in Behçet’s disease prevalence across countries.
\nTwo studies conducted in Korea reported on the clinical manifestations of Behçet’s disease patients according to gender [29, 30]. The first study was carried out by a rheumatology department of a university hospital in Turkey, which assessed the prevalence between male and female with Behçet’s disease in a retrospective chart review of 329 patients [29]. The study included patients who fulfilled the ISG criteria. Data on demographic features of the patients, family history, age of onset, manifestations of Behçet’s, positive pathergy test and HLA-B51 were retrieved from patient charts. Among the 329 patients, 199 (60.4%) were males. A family history of Behçet’s disease was reported by 9.7% of the patients and a family history of oral ulcers by 22.5%. Early onset was observed in patients with a positive family history; this phenomenon is termed as genetic anticipation which had already been seen in previous studies [31]. Female patients had an early onset compared to males (23.0 vs. 25.2 years, p < 0.05). Eye lesions were more common among males (42.2 vs. 29.2%, p = 0.014). Vascular involvement was also more common among in males (44.7 vs. 15.3%, p < 0.001), whereas joint involvement and headache were common among women (70.0 vs. 49.2%, p < 0.001 for joint involvement and 60.0 vs. 36.2%, p < 0.001 for headache). Frequency of genital ulcers, nodular lesions, papulopustular lesions and nervous system involvement was similar among males and females. Pathergy test was positive in 48.7% of the patients (52.4% among male and 43.4% among female, p = 0.316). HLA-B51 was positive in 44.1% of Behçet’s disease patients, and the frequency was lower among men, but the difference was not that significant (38.8 vs. 50.6%, p = 0.106). The second study was conducted in Korea, which was a retrospective chart review of 193 patients, which analysed gender, age of onset of Behçet’s disease and HLA-B51 [30]. The study showed that there is an increased prevalence in females (67%), whereas other parameters like the mean age at onset, the mean disease duration, positive pathergy test and positive HLA-B51 were similar in both males and females. Genital ulcers, peripheral joint involvement and inflammatory back pain were more common in females, while skin lesions were more frequent in males. In total, major organ involvement was present in 39% of the patients (uveitis, 27%; nervous system involvement, 7%; vascular involvement, 4% and gastrointestinal involvement, 11%), with similar frequency in both the genders. When clinical findings were compared based on onset of disease, patients with late onset (>40 years) had more nervous system involvement than those with an early onset (15.9 vs. 4.2%, p = 0.007).
\nLike the previous study, HLA-B51 positive patients had a history of early disease onset, but less nervous system (17.2 vs. 2.5%, p = 0.02) and gastrointestinal system (20.7 vs. 2.5%, p = 0.01) involvement than patients with negative HLA-B51 [32, 33].
\nA Japanese study conducted in 2015 analysed the change in disease phenotype over time among Behçet’s disease patients treated in seven hospitals in a district in mid-Japan between 1991 and 2015. Patients fulfilled the 1987 revised diagnostic criteria of the Behçet’s disease research committee; the Ministry of Health, Labour and Welfare of Japan was included in the study [34, 35]. This criterion classified the manifestations of Behçet’s disease into major and minor symptoms. Major symptoms include recurrent aphthous oral ulcers, skin lesions, ocular inflammation and genital ulcers, and minor symptoms include arthritis, intestinal ulcers, epididymitis, vascular lesions and neuropsychiatric disease.
\nThose who have all four major symptoms during the clinical course are classified as complete, whereas those who have three major symptoms, two major and two minor symptoms, typical recurrent ocular inflammation and one or more major symptoms or typical recurrent ocular inflammation and two minor symptoms are classified as incomplete Behçet’s disease. Patients were divided into three groups as group A, diagnosed before 2000; group B, diagnosed between 2000 and 2007; and group C, diagnosed after 2008. Authors observed a decrease in the number of patients with complete disease over time, suggesting a trend for milder disease in more recent years which could be due to usage of newer medicaments and awareness about the disease. The male-to-female ratio and the age of onset of disease were not changed over time, whereas the frequency of genital ulcers and HLA-B51 positivity decreased and gastrointestinal involvement frequency increased [34, 35].
\nAdditionally, individuals migrated from countries with high prevalence of Behçet’s disease to countries with less prevalence rate were shown to have less risk of developing disease [36, 37]. Prevalence of Behçet’s disease among Turkish individuals in Germany is about 21/100,000, which, although higher than that of the German population, remains inferior to prevalence in Turkey [38].
\nAlthough Behçet’s disease is rare in Africans, two series have been published recently based on studies conducted in Nigeria and Dakar from the year 2007 to 2011. Based on the observation by Italian authors, the prevalence of Behçet’s disease was found to be higher among immigrants when they analysed its prevalence among patients of Italian and non-Italian origin living in these areas.
\nA study conducted in the Netherlands estimated the prevalence of Behçet’s disease in patients with diverse ethnic origins residing in the Rotterdam area showed a prevalence of 1/100,000 population among Dutch Caucasians, 39/100,000 among Moroccans and 71/100,000 among Turks, suggesting that the prevalence among Moroccans is the same as in their countries of origin [39, 40].
\nPeculiar geographical distribution, familial aggregation, polymorphisms in genes that control immune responses and correlation with HLA-B51 class I antigen are the four factors that are considered to contribute to Behçet’s disease susceptibility. Ohno in 1973 described the association of BD with HLA-B5 [39]. They demonstrated that HLA-B5 includes HLA-B51 and HLA-B52 as it has a heterogeneous composition. In major histocompatibility locus, HLA-B51 and HLA-B5701 were associated with the pathogenesis of the disease, mainly among populace alongside of ancient Silk Road. Although associations with HLA-A and HLA-C have been described, they are non-specific and require confirmation. Other MHC genes such as TNF and MHC class I genes (MICA) are under study; the exact mechanism has not been fully understood. The association with HLA-B51 appears to be important in neutrophil activation. However, the presence of HLA-B51 alone is not ample to explain the manifestations of Behçet’s disease. A case series of Iranian patients showed association with HLA-B35, HLA-B51, HLA-B52 and HLA-Bw4. Supplementary studies suggest associations with HLA-B15, HLA-B27, HLA-B57 and HLA-A26 [40].
\nResearches focused on single-nucleotide polymorphisms (SNPs) revealed that SNP which is in the HLA-B region between HLA-B and MICA genes is responsible for the relationship between HLA-B51 and Behçet’s disease. The epistatic interactions with endoplasmic reticulum-associated aminopeptidase 1 (ERAP-1) has also been studied, and the authors suggested that the interaction of ERAP-1 and HLA-B has also been consistent, and these cytokines were apparently high in the peripheral blood mononuclear cells of patients with Behçet’s disease compared to the control group. Another conclusion was the unbalanced suppressor of cytokine signalling expression in patients with Behçet’s disease compared to controls [41].
\nA Chinese study stated that the polymorphisms of NOS3/rs1799983, a nitric oxide synthase gene, were found to be associated with the disease and the involvement of CD16 and CD11c in Behçet’s disease susceptibility. Mutations in the familial Mediterranean fever (MEFV) gene were suggested by a meta-analysis of 8 studies which analysed 2538 patients and 2792 healthy controls. Researches also suggested the associations of Toll-like receptors 7 (TLR7) and other nucleic acid-sensing genes of innate immunity-like inflammatory pathways such as IFI16 (a dsDNA cytosolic sensor and mediator of the AIM2-dependent inflammatory pathway) found to influence Behçet’s disease susceptibility [39]. Epigenetic studies with inverted repeat sequences (IRS) demonstrated that methylation level of IRS elements may influence the pathogenesis of the Behçet’s disease. Researches were done on genes related to apoptosis, but the results were not conclusive [41].
\nA multitude of factors that indicate the interaction between environmental, innate and adaptive immunity in the development of the disease has been already discussed in the Genetics section. Various researches have studied about the role of bacterial and viral agents in the development of Behçet’s disease, but the results were inconclusive.
Matzinger suggested the danger model, which occurs due to a continuous autoimmune cascade resulting from signals emitted by the affected cells of the host which will override the external stimulus. T cells and other antigen-presenting cells (APC) would command the process, which would be perpetuated on a favourable genetic terrain. Damage by a non-self-entity would trigger permanent aggression by activating an uncontrolled adaptive response. Because of their similarity to other pathogenic proteins, heat shock proteins (HSP60) could be involved. This adaptive reaction to external stimuli would persist in the permanent pathogenic presence via autoantigens that would activate dendritic T cells and B cells [40]. Thus, overexpression of proinflammatory cytokines especially Th1 and Th17 and probably association with genetic susceptibility may be the contributing factor for the increased inflammatory reaction in Behçet’s disease. Stimulated lymphocytes would activate neutrophils and endothelial cells, and HSPs would probably trigger innate and adaptive immune responses. Hypersensitivity to
Many studies have demonstrated the presence of IFN-gamma and IL-12 in peripheral blood of Behçet’s disease patients suggesting involvement of TH1 cells. Elevated IL-1, IL-6, IL-18, TNF-alpha and chemokines would reflect the activation of innate and adaptive immune systems. Neutrophils are hyperactivated with increased phagocytosis, chemotaxis, superoxide and lysosomal enzyme production. Lymphocytes have also shown abnormal functions such as clonal expansion of autoreactive T cells specific for heat shock protein 60 peptides. In addition, gamma-delta T cells are abundant in the blood and mucosal lesions of the affected individuals. Researchers have concluded that the interaction between T cells, neutrophils and antigen-presenting cells probably contributes to the pathogenesis of Behçet’s disease [40].
\nBehçet’s disease is a form of systemic vasculitis which affect almost all vascularized systems (Table 2) and is characterised by episodes of relapses and remissions [9, 43, 44].
\nManifestation | \nPrevalence | \nPrognosis | \nComments | \n
---|---|---|---|
Oral ulcers | \n47–83% | \nFavourable | \nAppear in all patients during the disease course | \n
Genital ulcers | \n57–93% | \nFavourable | \nLesion will leave a scar | \n
Ocular lesions | \n30–70% | \nPoor | \nMore frequent in males. Has high morbidity | \n
Cutaneous lesions | \n38–99% | \nFavourable | \nErythema nodosum more frequent in females | \n
Joint involvement | \n45–60% | \nFavourable | \nNondeforming and nonerosive | \n
Cardiovascular involvement | \n7–49% | \nPoor | \nMore frequent in males, high morbidity and mortality | \n
Neurological involvement | \n5–10% | \nPoor | \nMore frequent in males, high morbidity and mortality | \n
Gastrointestinal involvement | \n3–26% | \nPoor | \nMore frequent among Japanese | \n
Manifestations of Behçet’s disease.
The triad of oral aphthae, genital ulcers and ocular lesions specifies the disease pattern even though Behçet’s disease has other multisystem involvement and several clinical manifestations.
\nChildren exhibit more frequent perianal ulceration and more severe course of chorioretinitis and less frequent genital ulcers and vascular involvement [44, 45].
\nA meta-analysis of 18 articles which was performed by Horie et al. to assess the association between HLA-B51 and the ocular manifestations of Behçet’s disease among various ethnic group studies from Middle and Far East suggested that there was a strong association between HLA-B51 expression and ocular involvement, whereas studies from North Africa and Europe showed no association [46].
\nAccorinti et al. investigated demographic and clinical trends of 385 Behçet’s disease patients with uveitis seen over 44 years in a referral unit at Sapienza University in Rome, Italy. The cohort was divided into cohort 1, which included the patients seen from 1968 to 1992, and cohort 2 included the patients seen during 1993 and 2011. Compared to the cohort 1, the cohort 2 has more female patients, more patients with milder disease (more frequent isolated anterior uveitis, less frequent hypopyon) and more immunosuppressive use. Ocular complications such as optic atrophy, maculopathy and retinal neovascularisation and retinal detachment were significantly more common in cohort 1. The visual acuity at final evaluation was better in the second cohort. When considered both the cohorts, more severe findings were observed in males. From the above findings, the authors concluded that timely and aggressive management reduces ocular complications and facilitates better prognosis [47].
\nEven though uveitis is the common ocular pathology in Behçet’s disease, it is not a common cause of uveitis. The highest incidence of uveitis is reported by Mishima S from Japan [48], where it constitutes about 25%, but it is much rarer in other countries such as Brazil, where the incidence is 2% [49], and India, where the incidence is 1% reported by Sen DK from a study conducted in 94 Indian children [50]. The classical feature of Behçet’s disease is recurrent, acute anterior uveitis associated with hypopyon. Other ocular manifestations seen in Behçet’s disease include necrotizing vascular lesions involving the retina and the optic nerve. These manifestations may be unnoticed because of severity of the anterior segment reaction and commonly include macular oedema, retinal periphlebitis and periarteritis, thrombosis of the vessels and retinal or vitreous haemorrhages which are frequently observed in males with Behçet’s disease [51, 52].
\nRecurrent oral ulcers are one of the earliest manifestations of Behçet’s disease in around 47–86% of patients and are seen in almost all patients during their clinical course [53]. Other symptoms may at times take years to appear after the onset of disease. Oral ulcerative lesions usually manifest as a round- or oval-shaped ulcer with discrete erythematous border with a greyish-white pseudomembrane or a central yellowish fibrinous floor and grow rapidly from a flat ulcer to a deep sore [9]. They may occur as single ulcers or as numerous [9, 53]. Oral ulcers most commonly affect the gingival and buccal mucosa, tongue and lips yet may also appear in the soft and hard palates, pharynx and tonsils [9]. Minor ulcers which are less than 1 cm in diameter heal without scarring over a period of 1 to 2 weeks, whereas major ulcers which are more than 1 cm in diameter are more painful and heal within 2–6 weeks. Herpetiform ulcers occurring in recurrent crops of small ulcers that are 0.2–0.3 cm in diameter are painful and may coalesce to form large ulcers. Treatment is usually symptomatic, and prognosis of oral ulcerations is favourable [18, 53].
\nThe most commonly seen cutaneous manifestations are erythema nodosum and pseudofolliculitis, in which erythema nodosum is manifested as painful multiple subcutaneous nodules that vary in size and colour, whereas pseudofolliculitis (pustulosis) appears as a dome-shaped sterile pustule on a round reddish edematous base that looks like acne vulgaris [54]. Although these are commonly seen on the lower extremities, it can be seen throughout the body [55]. The pathergy reaction is included in the criteria for the diagnosis of Behçet’s disease. Although more than 50% of patients from Turkey and Japan had a positive pathergy test, it is rarely observed in patients from Northern Europe, the USA and Australia [56, 57]. It used to be an important diagnostic criterion for Behçet’s disease; however, the frequency of the pathergy phenomenon was reported to decrease during the recent years [58]. Subcutaneous thrombophlebitis is often confused with lesions of erythema nodosum; the former is manifested as tender erythematous nodules with a linear arrangement in most case. The lesions may relocate depending on the vascular segment involved [59].
\nCardiac involvement is a rare finding in patients with Behçet’s disease. Behçet’s disease affects arteries, veins as well as the heart. Cardiovascular features have been reported to affect 7–49% of patients, more frequently in males. They occur 3–16 years after the onset of Behçet’s disease. Cardiac involvement in Behçet’s disease patients was first described by Mirone et al. in 1958 as a case of paroxysmal fibrillation and heart block. In 1963, Oshima et al. described a case of myocardial infarction complicated by incomplete right bundle-branch block [60]. Since then different types of cardiovascular lesions, including pericarditis, atrial thrombus, complex ventricular arrhythmia, myocardial infarction, heart block and sudden death, have been reported in association with Behçet’s disease [61]. Dong Soo et al. in 1998 reported a case of superior vena cava syndrome caused by Behçet’s disease in a 40-year-old man with recurrent oral aphthous ulcers and skin rashes on the anterior chest wall [62].
\nNeurological involvement in Behçet’s disease, the neuro-Behçet’s disease, occurs very rarely and only in 5–10% of patients. It is frequently seen in males [63]. It usually occurs around 5 years after the onset of the disease and is associated with long-term morbidity and mortality [9]. Neuro-Behçet’s disease can be parenchymal, nonparenchymal or mixed brain disease, in which the parenchymal brain disease affects the brainstem and/or basal ganglia and is correlated with a poor prognosis, whereas nonparenchymal also known as vasculo-Behçet’s disease or angio-Behçet’s disease is characterised by subsets of cerebral venous thromboses, dural venous sinus thrombosis, arterial vasculitis and aseptic meningitis and comprises the most devastating symptom category of Behçet’s disease with high mortality [63, 64]. A mixed parenchymal and nonparenchymal disease has also been reported but is very rare. Infrequent clinical manifestations like epilepsy, stroke, brain tumour-like neuro-Behçet’s disease, acute meningeal syndrome, movement disorders, optic neuropathy, spinal cord involvement and asymptomatic and subclinical neurological involvement have also been reported [65, 66]. Meningitis, neurological deficits including motor disturbances and brainstem symptoms and psychiatric symptoms including personality changes develop in patients with neuro-Behçet’s disease are considered as a classic finding [18, 63]. These symptoms are associated with disease exacerbations and gradually cause disability and unfortunately are irreversible [18]. At late stages, dementia develops in approximately one-third of patients with neuro-Behçet’s disease.
\nGastrointestinal involvement was observed in 3–26% of patients and varies among different populations [38, 39]. It is much more frequent in Japan than in the Middle East and the Mediterranean region [1, 2, 3]. Mucosal inflammation and ulceration can occur anywhere in the gastrointestinal tract but typically in the ileocecal region. Other rarely involved sites include the oesophagus, ascending colon and transverse colon [18, 19, 67]. Clinical symptoms include anorexia, vomiting, dyspepsia, diarrhoea, melena, abdominal pain and, less frequently, perforation requiring surgical intervention [68]. Due to the similarity in intestinal and extraintestinal symptoms, it is tough to differentiate Behçet’s disease from inflammatory bowel disease; however, the presence of granulomata can be used to confirm inflammatory bowel disease [66].
\nJoint involvement is common in patients with Behçet’s disease seen in almost half of patients. It presents as an initial manifestation seen long before the commencement of other features of BD [55]. Erosive, non-deforming oligoarthritis typically involving the knees, ankles and wrists is seen in most of the Behçet’s disease patients. Rarely, it can present as sacroiliitis or erosive arthritis. Myopathy has also been reported by Arkin in 1980 [69, 70, 71, 72].
\nVasculitis is one of the classic signs in Behçet’s disease, and the typical finding is deep vein thrombosis. Thrombophlebitis occurs generally in the first year after onset of Behçet’s disease, and relapses are frequent. Vasculitis can affect both the veins and arteries and capillaries although venous involvement is more common [55]. Venous thrombosis may occur at any site and can even involve large vessels like the inferior vena cava, the superior vena cava and the pulmonary artery. [55]. Arterial involvement is seen in 3–5% of cases [73]. Aneurysm formation is found to be the foremost manifestation, and the affected patients can be asymptomatic [74]. Vascular surgery is mandatory, but relapse is a frequent concern [55].
\nPulmonary manifestations are rare in patients with Behçet’s disease. Haemoptysis is the only reported manifestation, but it can be massive and fatal [74].
\nThe diagnosis of Behçet’s disease relies purely on its clinical manifestation since it lacks specific biological test, so diagnosis with clinical symptoms is challenging, especially due to non-concomitant symptoms. Hence different diagnostic criteria have been developed for the determination of Behçet’s disease. In 1990, an ISG for Behçet’s disease was formed by a group of researchers [21]. The International Study Group developed a set of classification criteria which is designed for research studies, which in many instances is used for diagnosing. The features were defined as the presence of specific symptoms as seen in Table 3. Oral ulceration along with any other two symptoms is said to be diagnostic [75]. The capability of the ISG criteria as a diagnostic tool has been questioned [56]. An international team which includes 27 countries was formed to evaluate and reassess the ISG criteria, and a new ICBD was developed that include the major criteria consisting of oral aphthosis, genital aphthosis and ocular lesions which were each given 2 points, whereas 1 point was assigned to the skin lesion, neurological manifestation, vascular manifestation and positive pathergy test (which is optional and not included in the primary criteria due to geographic variation and its declining sensitivity and increased specificity) [58, 75]. Higher sensitivity of the ICBD was reported as it aided in earlier diagnosis and hence earlier treatment and thereby better prognosis [75]. However, the delay between the onsets of major manifestations may be decades needs to be considered [76]. Inflammatory markers such erythrocyte sedimentation rate (ESR) and C-reactive protein have no longer been recognised as accurate or specific for the disease activity (Table 4).
\nRecurrent oral ulcerations | \nMinor aphthous, major aphthous or herpetiform ulceration observed by physician or patient, which recurred at least three times in one 12-month period | \n
Plus any two of the following | \n|
Recurrent genital ulceration | \nAphthous ulceration or scarring observed by physician or patient | \n
Eye lesion | \nAnterior uveitis, posterior uveitis or cells in vitreous on slit lamp examination or retinal vasculitis observed by ophthalmologist | \n
Skin lesions | \nErythema nodosum observed by physician or patient, pseudofolliculitis or papulopustular lesions or acneiform nodules observed by physician in post adolescent patients not on corticosteroid treatment | \n
Positive pathergy test | \nRead by a physician at 24–48 hours | \n
The international study group criteria for Behçet’s disease [21].
Symptoms | \nPoints | \n
---|---|
Oral aphthosis | \n2 | \n
Genital aphthosis | \n2 | \n
Ocular lesion | \n2 | \n
Skin lesion | \n1 | \n
Neurological manifestation | \n1 | \n
Vascular manifestation | \n1 | \n
Positive pathergy test | \n1b | \n
International criteria for Behçet’s disease [75].
Investigations for Behçet’s disease include routine total blood count; renal, liver and bone profile; inflammatory markers, including C-reactive protein and erythrocyte sedimentation rate; urine analysis; chest X-ray; coeliac screen; stool sample; autoimmune screen; coagulation profile and antiphospholipid antibodies; mouth and genital ulcer swab and culture; and occasionally eye swab. Other investigations that may be required include oral biopsy and direct immunofluorescence to exclude orofacial granulomatosis and bullous dermatosis. Vulval biopsy is indicated to exclude lichen sclerosis).
\nDoppler studies, CT or MRI brain and spinal cord, magnetic resonance venography, computerised tomographic venography, magnetic resonance angiography, computerised tomography angiography to evaluate neurologic and vascular disease; CSF studies, electro encephalogram, electromyography, nerve conduction study, 18 F fluoro2-deoxyglucose positron emission tomography, with CT/MRI localisation (for early inflammation in large vessel vasculitis). Musculoskeletal BD—synovial fluid analysis, X-ray, ultrasound or MRI to evaluate joints. Skin biopsy and immunofluorescence for cutaneous involvement. Cardiac BD—electrocardiogram, echocardiogram in case of cardio vascular involvement chest CT to assess mediastinal diseases, fibrosing mediastinitis, aneurysms, pleural effusions, complications of venous thrombosis and collaterals. Stool sample for faecal calprotectin, endoscopy and biopsy if there is Gastrointestinal involvement. Ocular BD—optical coherence tomography, visual evoked potentials, fluorescein angiogram, Schirmer’s test, intraocular fluid culture to exclude infections in Behçets patients with ocular involvement. Cystoscopy and kidney, urinary bladder CT for urological disease [7].
\nThe multisystem involvement in Behçet’s systems alarms the requirement of a teamwork from different medical specialties such as oral medicine, ophthalmology, dermatology, rheumatology, neurology, cardiology and gastroenterology for its management. The main objective of management of Behçet’s disease is to induce and maintain remission and improve the patient’s quality of life, preventing irreversible damage and exacerbation of mucocutaneous and articular disease [77, 78]. Since Behçet’s disease has plethoric expressions, treatment plan may vary depending on severity, organ affected, age of disease onset, disease duration and frequency of recurrences [59].
\nThe European League Against Rheumatism (EULAR) formed a committee in 2008 to develop an evidence-based recommendation for the treatment modalities of Behçet’s disease [78]. Suggestions regarding ocular and mucocutaneous diseases and arthritis are primarily evidence-based, but recommendations on neurological, vascular and gastrointestinal involvement relied on observational studies, retrospective analyses and expert opinion [78].
\nTreatment of Behçet’s disease is mainly based on the suppression of the inflammatory attack using immunomodulatory agents such as corticosteroids, azathioprine or interferon 𝛼 [78]. The primary line of management of mucocutaneous lesions in Behçet’s disease is colchicine (1 mg/day). NSAID is usually sufficient for joint manifestations [54]. Clinicians should keep in mind the neurotoxicity of cyclosporine A while prescribing it to any patient with neurologic problems for treating vascular involvement and refractory eye disease [54]. Biologic agents are the only option if the disease seems to be resistant to treatments [78]. Suggested management options are summarised in Tables 5 and 6.
\nManifestations | \nTreatments | \n
---|---|
Mild mucocutaneous lesions | \nTopical steroids | \n
Moderate to severe mucocutaneous lesions | \nInitial: systemic steroid, colchicine | \n
Refractory case: azathioprine, interferon𝛼 | \n|
Ocular lesions | \nInitial: systemic steroid + azathioprine | \n
Refractory case: first cyclosporine A + steroid + azathioprine Second interferon 𝛼+/− systemic steroid | \n|
Arthritis | \n(First) colchicine nonsteroidal anti-inflammatory drug, (second) azathioprine, interferon 𝛼 | \n
Vascular: deep venous thrombosis | \nAzathioprine, cyclosporine A, cyclophosphamide (larger vessels) | \n
Vascular: arterial aneurysm | \nCyclophosphamide + systemic steroid | \n
Neurological | \nCyclophosphamide (drug of choice) | \n
Gastrointestinal | \nSystemic steroid, sulfasalazine, azathioprine | \n
Disease-modifying drugs (DMDs) | \nDosage | \n
---|---|
Azathioprine | \n2–3 mg/kg/day | \n
Mycophenolate mofetil | \n2–3 g/day | \n
Methotrexate | \n20–25 mg/week | \n
Tacrolimus | \n4–8 mg/day | \n
Ciclosporin | \n2–5 mg/kg/day | \n
Sulfasalazine | \n2–3 g/day | \n
Dapsone | \n2–3 mg/kg/day | \n
Thalidomide (exceptional use) | \n50–300 mg/day | \n
Colchicine | \n0.5–2 mg/day | \n
Prednisolone | \nVariable dose (depending on indication and stage of treatment) | \n
Cyclophosphamide | \n15 mg/kg (vasculitis regimens) | \n
Infliximab | \n5 mg/kg at 0, 2 and 6 weeks then once every 8 weeks | \n
Adalimumab | \n40 mg every 2 weeks | \n
Etanercept | \n50 mg/week | \n
Certolizumab | \n400 mg at 0, 2 and 4 weeks then once every 4 week | \n
Rituximab | \n1 g at 0 and 2 weeks | \n
Interferon α | \nVarious regimens for Roferon A and pegylated interferon α 2b | \n
Alemtuzumab | \n3 mg (day 1), 10 mg (day 3), 30 mg (day 5), 30 mg (day 8), 30 mg (day 10) and 30 mg (day 12) | \n
Disease-modifying drugs used in Behçet’s disease [7].
Behçet’s disease is a chronic, relapsing vasculitis with multisystem involvement with considerable morbidity and mortality. There exists a delay in the diagnosis of Behçet’s disease since it lacks a specific diagnostic test and biomarkers. Another area to be focussed on is to develop a more effective disease activity score. Researches are on the go to explore more about the disease’s pathophysiology which may aid in early diagnosing and effective treatment planning.
\nWe thank Prof Dr. Sobha Kuriakose, Dr. Sarika S. Kamal, Dr. Sumal V. Raj, Dr. Nithin V. S and Dr. Deena C. Thomas, Dr. Alaka Subodh and Adithi S. A for critically analysing this manuscript.
\nIn the urban context, the importance of transport stems from the fact that it contributes to the productivity of workers and competitiveness of firms. It widens labour markets and makes them inclusive. It saves travel time and costs to reach valued destinations – for work, education, shopping and leisure. Urban transport investments augment agglomeration economies by enhancing access to the economic mass, reducing congestion and channelizing residential and non-residential development in desired directions. They balance the location of jobs, housing and common facilities. Urban transport plays an important role in the working of cities, enhancing their efficiency, facilitating economic growth, generating value enhancements to finance planned urban development, and creating livable, competitive and sustainable cities.
In the last four decades, the issues of urban transport have come into sharp focus in many developed countries around the world due to the problems of their automobile-dependent model of urban development. They have been subject to high levels of traffic congestion, air pollution, accidents, damages to ecosystems and neighborhoods, segregation and adverse impacts on the quality of life in cities.
Rapid motorization has worsened traffic conditions, aggravated congestion and pollution levels in several cities around the globe. Apart from environmental concerns, traffic congestion is also detrimental to the economic health of cities by adding to the wastage of time and fuel and increasing the levels of emissions. It hampers productivity by delaying and hindering the movement of goods, raw materials as well as people.
The proliferation of personalized vehicles, lack of investment in public transport and implementation of a spatial planning model that promoted dispersed, automobile-centric development have been the primary factors behind the urban transport problems in countries. The search for ways of making urban communities provide a better quality of life and promoting sustainable cities led to the emergence of an urban design paradigm called ‘New Urbanism’ in North America and Western Europe in the 1970s and 1980s. This was followed by a theory of urban planning and transportation called ‘Smart Growth’. This theory is founded on the following principles of urban design: (i) mixed land use; (ii) compact design; (iii) increased densities; (iv) housing opportunities and choices; (v) walkable and accessible neighborhoods; (vi) multiple transportation mode choices; (vii) neighborhood centres to foster social interaction; (viii) preservation of open space, farm land, natural beauty and critical environmental areas; (ix) strengthening of and directing development towards existing communities; (x) making development decisions predictable, fair, and cost-effective; and (xi) community and stakeholders’ consultation in development decisions [1]. Transit-oriented Development (TOD), a key element of Smart Growth, aims at concentrating development around one or more transit stations or within a transit corridor.
TOD aims at compact, high density and mixed use development within easy walking or biking distance from a transit station, typically about 1 kilometer. Focused around a transit node, TOD facilitates access to public transit, thereby inducing people to walk, cycle and use public transport rather than personal vehicles. The selective concentration of development acts against sprawl, promotes agglomeration economies and mitigates congestion diseconomies. It also leads to increase in property values, reflecting the benefits to residents and businesses of diverse transportation options, and resultant automobile and parking cost savings [2]. Thus, TOD assists in the mobilization of value capture finance by harnessing the windfall gains accruing to land and property-owners. The key factors that support TOD include: land use and development policies promoting dense and compact development around transit nodes and discouraging such development in the areas without good access to public transport; development of public transit and provision of quality transit services; integration of transportation and land use; and application of other mobility management strategies. These factors jointly increase the cost-effectiveness and utility of TOD for consumers as well as businesses. TOD has the potential of becoming a powerful tool for planned development of cities and rural areas in developing countries. It not only improves connectivity between regions, but also saves a lot of time and costs of workers. It augments productivity and efficiency of economic agents. The case for transit-oriented development is well-argued in research [3].
The motivation behind this research is the need for India to move from an automobile-dependent to a public transportation-led and transit-oriented model of planned urban development. The current practice of master planning in India, rooted in the 1947 Town and Country Planning Act in the United Kingdom, has neglected urban transport. The model has not facilitated transportation-land use integration, transit-oriented development and value capture financing. Land use planning and transportation planning have been pursued as independent exercises, a prime example being Delhi. Cities have thus not been able to benefit from the interaction of transport and land use for sustainable urban development and adopt a robust mechanism of financing public transit. In this context, this paper explores the theory and international practice of New Urbanism, Smart Growth and TOD. It also examines the potential of TOD to raise revenues towards financing public transportation. The objective is to draw lessons from successful practices to strategize TOD policy for cities in India. Finally, the paper analyses the existing practices in Indian cities, identifies its inadequacies and suggests corrective measures.
The study is organized as follows. Section 2 deals with the challenges of urban mobility in India. Referring to the trends of urbanization, metropiltanization and motorization, it highlights the imperative for a public transportation-based strategy of urban development in the country. Section 3 discusses the paradigmTransit Oriented Development (TOD), adopted by several developed countries to address their problems of sprawl, inefficient urban form, excessive energy consumption, greenhouse gas emission, and environmental degradation. It makes a strong case for TOD as a dominant paradigm of urban planning and development in India. Section 4 presents some examples of successful TOD strategies practiced world-wide and draws lessons for urban transport development and land use planning, in general and TOD, in particular. Section 5 focuses on financing and execution practices of TOD internationally in the overall context of urban transport development to present a range of financing instruments that Indian cities could consider to promote TOD. Section 6 presents the emerging approaches towards TOD in India, referring to case studies, including projects, policies and plans in the offing. We specially focus on financing issues. Section 7 brings out the challenges of implementing TOD in India and indicates some directions for the design of a public transportation-led, transit-oriented and value increment financing-based strategy to address India’s urbanization challenges. It also calls for an effective institutional structure for the implementation of TOD and suggests reforms in the regional and urban planning model being followed. Section 8 concludes.
Bourgeoning travel demand, rapid motorization, rise in personalized vehicles, dwindling share of public transport, congestion, degradation environmental quality, rising number of road accidents and fatalities, fragmented institutional arrangements and chronic under-investment in transport infrastructure pose major hindrances to urban mobility. These are linked to the trends and patterns of urbanization, concentration of productive economic activity, income distribution structure in cities and motorization.
Urbanization in India is characterized by rising urban population and increased density in large cities. This has led to a rapid growth in travel demand. Tables 1–4 present the trends and patterns of urbanization in India.
Year | Total Population | Rural Population | Percentage Rural | No of Cities /Towns | Urban Population | Percentage Urban |
---|---|---|---|---|---|---|
1901 | 238.4 | 212.5 | 89.2 | 1916 | 25.9 | 10.8 |
1911 | 252.1 | 226.2 | 89.7 | 1908 | 25.9 | 10.3 |
1921 | 251.3 | 223.2 | 88.8 | 2048 | 28.1 | 11.2 |
1931 | 279.0 | 245.5 | 88.0 | 2220 | 33.5 | 12.0 |
1941 | 318.7 | 274.5 | 86.1 | 2427 | 44.2 | 13.9 |
1951 | 361.1 | 298.6 | 82.7 | 3060 | 62.4 | 17.3 |
1961 | 439.2 | 360.3 | 82.0 | 2700 | 78.9 | 18.0 |
1971 | 548.2 | 439.0 | 80.1 | 3126 | 109.1 | 19.9 |
1981 | 683.3 | 523.9 | 76.7 | 4029 | 159.5 | 23.3 |
1991 | 846.3 | 628.7 | 74.3 | 4689 | 217.6 | 25.7 |
2001 | 1028.7 | 742.5 | 72.2 | 5161 | 286.1 | 27.8 |
2011 | 1210.7 | 833.5 | 68.8 | 7935 | 377.1 | 31.2 |
India: Total, rural and urban population (in million) and level of urbanization (percentage) 1901–2011.
Number of Agglomerations/Towns | Percentage of Urban Population | |||||||||||
---|---|---|---|---|---|---|---|---|---|---|---|---|
Census Year | Class I | Class II | Class III | Class IV | Class V | Class VI | Class I | Class II | Class III | Class IV | Class V | Class VI |
1901 | 24 | 43 | 130 | 391 | 744 | 479 | 26.00 | 11.29 | 15.64 | 20.83 | 20.14 | 6.10 |
1911 | 23 | 40 | 135 | 364 | 707 | 485 | 27.48 | 10.51 | 16.40 | 19.73 | 19.31 | 6.57 |
1921 | 29 | 45 | 145 | 370 | 734 | 571 | 29.70 | 10.39 | 15.92 | 18.29 | 18.67 | 7.03 |
1931 | 35 | 56 | 183 | 434 | 800 | 509 | 31.20 | 11.65 | 16.80 | 18.00 | 17.14 | 5.21 |
1941 | 49 | 74 | 242 | 498 | 920 | 407 | 38.23 | 11.42 | 16.35 | 15.78 | 15.08 | 3.14 |
1951 | 76 | 91 | 327 | 608 | 1124 | 569 | 44.63 | 9.96 | 15.72 | 13.63 | 12.97 | 3.09 |
1961 | 102 | 129 | 437 | 719 | 711 | 172 | 51.42 | 11.23 | 16.94 | 12.77 | 6.87 | 0.77 |
1971 | 148 | 173 | 558 | 827 | 623 | 147 | 57.24 | 10.92 | 16.01 | 10.94 | 4.45 | 0.44 |
1981 | 218 | 270 | 743 | 1059 | 758 | 253 | 60.37 | 11.63 | 14.33 | 9.54 | 3.58 | 0.50 |
1991 | 300 | 345 | 947 | 1167 | 740 | 197 | 65.20 | 10.95 | 13.19 | 7.77 | 2.60 | 0.29 |
2001 | 393 | 401 | 1151 | 1344 | 888 | 191 | 68.67 | 9.67 | 12.23 | 6.84 | 2.36 | 0.23 |
2011 | 468 | 474 | 1373 | 1686 | 1748 | 424 | 70.15 | 8.54 | 11.11 | 6.39 | 3.36 | 0.45 |
India: Number of agglomerations/towns and percentage of urban population by size classes of towns 1901–2011.
Note: Class I: 100,000 or more, Class II: 50,000 – 99,999, Class III: 20,000 – 49,999; Class IV: 10,000 – 19,999, Class V: 5000–9999 and Class VI: Below 5000.
Each urban agglomeration, comprising generally a number of cities, towns and outgrowths is considered as one unit.
Census year | Number | Population (in Million) | Population per city (in Million) | Percentage of urban population |
---|---|---|---|---|
1901 | 1 | 1.51 | 1.51 | 5.84 |
1911 | 2 | 2.76 | 1.38 | 10.65 |
1921 | 2 | 3.13 | 1.56 | 11.14 |
1931 | 2 | 3.41 | 1.70 | 10.18 |
1941 | 2 | 5.31 | 2.65 | 12.23 |
1951 | 5 | 11.75 | 2.35 | 18.81 |
1961 | 7 | 18.10 | 2.58 | 22.93 |
1971 | 9 | 27.83 | 3.09 | 25.51 |
1981 | 12 | 42.12 | 3.51 | 26.41 |
1991 | 23 | 70.66 | 3.07 | 32.54 |
2001 | 35 | 108.29 | 3.09 | 37.85 |
2011 | 53 | 160.70 | 3.03 | 42.61 |
India: Number of metropolitan cities and their share in urban population 1901–2011.
Sl No | States | Percentage of Urban Population | Annual Exponential Growth Rate | |||||||
---|---|---|---|---|---|---|---|---|---|---|
1971 | 1981 | 1991 | 2001 | 2011 | 1971–1981 | 1981–1991 | 1991–2001 | 2001–2011 | ||
1 | Andhra Pradesh | 19.3 | 23.3 | 26.8 | 27.3 | 33.4 | 3.9 | 3.6 | 1.4 | 3.04 |
2 | Arunachal Pradesh | 3.7 | 6.3 | 12.2 | 20.4 | 22.9 | 8.3 | 9.3 | 7.0 | 3.31 |
3 | Assam | 8.8 | 9.9 | 11.1 | 12.7 | 14.1 | 3.3 | 3.3 | 3.1 | 2.46 |
4 | Bihar | 10.0 | 12.5 | 13.2 | 10.5 | 11.3 | 4.3 | 2.7 | 2.6 | 3.03 |
5 | Chhattisgarh | NA | NA | NA | 20.1 | 23.2 | NA | NA | 3.1 | 3.49 |
6 | Delhi | 89.7 | 92.8 | 89.9 | 93.0 | 97.5 | 4.6 | 3.8 | 4.1 | 2.37 |
7 | Goa | 26.4 | 32.5 | 41.0 | 49.8 | 62.2 | 4.4 | 4.0 | 3.3 | 3.01 |
8 | Gujarat | 28.1 | 31.1 | 34.4 | 37.4 | 42.6 | 3.4 | 2.9 | 2.8 | 3.07 |
9 | Haryana | 17.7 | 22.0 | 24.8 | 29.0 | 34.9 | 4.7 | 3.6 | 4.1 | 3.68 |
10 | Himachal Pradesh | 7.0 | 7.7 | 8.7 | 9.8 | 10.0 | 3.0 | 3.1 | 2.8 | 1.45 |
11 | Jammu &Kashmir | 18.6 | 21.1 | 22.8 | 24.9 | 27.4 | 3.8 | 3.4 | 3.4 | 3.10 |
12 | Jharkhand | NA | NA | NA | 22.3 | 24.0 | NA | NA | 2.6 | 2.80 |
13 | Karnataka | 24.3 | 28.9 | 30.9 | 34.0 | 38.7 | 4.1 | 2.6 | 2.5 | 2.74 |
14 | Kerala | 16.2 | 18.8 | 26.4 | 26.0 | 47.7 | 3.2 | 4.8 | 0.7 | 6.56 |
15 | Madhya Pradesh | 16.3 | 20.3 | 23.2 | 26.7 | 27.6 | 4.5 | 3.7 | 2.7 | 2.28 |
16 | Maharashtra | 31.2 | 35.0 | 38.7 | 42.4 | 45.2 | 3.4 | 3.3 | 3.0 | 2.12 |
17 | Manipur | 13.2 | 26.4 | 27.7 | 23.9 | 32.5 | 9.7 | 3.0 | 1.2 | 3.70 |
18 | Meghalaya | 14.6 | 18.0 | 18.7 | 19.6 | 20.1 | 4.9 | 3.1 | 3.2 | 2.70 |
19 | Mizoram | 11.4 | 25.2 | 46.2 | 49.5 | 52.1 | 11.8 | 9.6 | 3.3 | 2.59 |
20 | Nagaland | 10.0 | 15.5 | 17.3 | 17.7 | 28.9 | 8.5 | 5.6 | 5.3 | 5.10 |
21 | Odisha | 8.4 | 11.8 | 13.4 | 15.0 | 16.7 | 5.2 | 3.1 | 2.6 | 2.38 |
22 | Punjab | 23.7 | 27.7 | 29.7 | 34.0 | 37.5 | 3.6 | 2.6 | 3.2 | 2.29 |
23 | Rajasthan | 17.6 | 20.9 | 22.9 | 23.4 | 24.9 | 4.5 | 3.3 | 2.7 | 2.54 |
24 | Sikkim | 9.4 | 16.2 | 9.1 | 11.1 | 25.2 | 9.6 | −3.2 | 4.8 | 9.42 |
25 | Tamil Nadu | 30.3 | 33.0 | 34.2 | 43.9 | 48.4 | 2.5 | 1.8 | 3.6 | 2.39 |
26 | Tripura | 10.4 | 11.0 | 15.3 | 17.0 | 26.2 | 3.3 | 6.2 | 2.5 | 5.66 |
27 | Uttar Pradesh | 14.0 | 18.0 | 19.9 | 20.8 | 22.3 | 4.8 | 3.3 | 2.8 | 2.53 |
28 | Uttaranchal | NA | NA | NA | 25.6 | 30.2 | NA | NA | 2.8 | 3.36 |
29 | West Bengal | 24.8 | 26.5 | 27.4 | 28.0 | 31.9 | 2.8 | 2.5 | 1.8 | 2.60 |
Union Territories | ||||||||||
1 | Andaman & Nicobar Islands. | 22.8 | 26.4 | 26.8 | 32.7 | 37.7 | 6.4 | 4.1 | 4.4 | 2.10 |
2 | Chandigarh | 90.6 | 93.6 | 89.7 | 89.8 | 97.3 | 5.9 | 3.1 | 3.4 | 2.38 |
3 | Dadra & Nagar Haveli | 0.0 | 6.7 | 8.5 | 22.9 | 46.7 | — | 5.3 | 14.6 | 11.57 |
4 | Daman & Diu | — | — | 46.9 | 36.3 | 75.2 | — | 4.9 | 1.9 | 11.59 |
5 | Lakshadweep | 0.0 | 46.3 | 56.3 | 44.5 | 78.1 | — | 4.5 | −0.8 | 6.24 |
6 | Pondicherry | 42.0 | 52.3 | 64.1 | 66.6 | 68.3 | 4.7 | 4.9 | 2.3 | 2.73 |
All India | 20.2 | 23.7 | 25.7 | 27.8 | 31.2 | 3.8 | 3.1 | 2.7 | 2.76 |
India: Level of urbanization and growth in urban population across states and union territories 1971–2011.
Note: a) The figures for the states of Uttar Pradesh, Bihar and Madhya Pradesh for the 1970s and 1980s pertain to the undivided states as existed during that time. The figures for the1990s are, however, for the new states and hence these figures are not temporally comparable.
b) In the absence of the Census data for total and urban population for the year 1981 in case of Assam, the urban and total population growth rates have been assumed to be constant during 1970s and 1980s. The same has been assumed for 1980s and 1990s for Jammu and Kashmir. The percentage of urban population has been arrived for Assam (1981) and Jammu and Kashmir (1991) based on these assumptions.
c) Goa in 1971 and 1981corresponds to Goa, Daman and Diu.
While the number of cities/towns in India increased by 3 times, urban population rose by 13 times between 1901 and 2011. This reflects the concentrated pattern of urbanization. In 2011, the number of urban agglomerations (UAs) /towns was 7935 as against 5161 in 2001. While the number of statutory towns rose from 3799 to 4041 between 2001 and 2011, the number of census towns experienced a phenomenal jump from 1362 to 3894. About 30 percent of urban population growth in the last decade is accounted for by census towns.
Table 2 presents the distribution of urban population between size classes of towns in India from 1901 to 2011. It reflects a top-heavy urban structure, highlighting the increasing density of large cities. Table 3 presents the trends in metropolitan population in India and reflects a similar trend.
There are large interstate variations in urbanization patterns in India, having differential implications for urban transport demand and strategy. Among the states, Delhi was the most urbanized in 2011, with 97.5 percent urbanization level, followed by Goa (62.2 percent), Mizoram (52.1 percent) and Tamil Nadu (48.4 percent). Table 4 presents the percentage of urban population in 1971, 1981, 1991, 2001 and 2011, and decadal annual exponential growth in urban population for 1971–81, 1981–91, 1991–2001 and 2001–11.
India’s urban population is projected to more than double between 2011 and 2050 – from 377 million to 814 million. With an estimated rural population of 860 million in 2014, the country would still have 810 million in villages in 2050 [5]. Thus, India would confront the dual challenges of urban and rural development for many decades. The country has to address not only the problems of transportation within cities, it will have to connect villages to cities and towns providing efficient transport services to rural areas.
Census of India 2011 data reveals that not only many cities, but also urban agglomerations or regions in India have a population density of more than 10,000 – with central city areas being denser than peripheries. Table 5 provides data on densities of 10 urban districts in India with the highest population density. A simple conclusion from international comparisons relating to population densities of urban regions is that the density patterns of many cities and urban districts in India overwhelmingly support a public transport-led urban development strategy.
Rank | Urban District | Area (Sq. Kms) | 2001 Census | 2011 Census | ||
---|---|---|---|---|---|---|
Population (In Lakhs) | Density | Population (In Lakhs) | Density | |||
1 | North East Delhi | 56 | 17.68 | 31,573 | 22.42 | 36,155 |
2 | Central Delhi | 23 | 6.46 | 28,104 | 5.82 | 27,730 |
3 | East Delhi | 49 | 14.64 | 29,869 | 17.09 | 27,132 |
4 | Chennai | 174 | 43.44 | 24,963 | 46.47 | 26,553 |
5 | Kolkata | 185 | 45.72 | 24,718 | 44.97 | 24,306 |
6 | Mumbai Suburban | 446 | 86.40 | 19,373 | 93.57 | 20,980 |
7 | Mumbai City | 157 | 33.38 | 21,261 | 30.85 | 19,652 |
8 | West Delhi | 131 | 21.29 | 16,251 | 25.43 | 19,563 |
9 | Hyderabad | 217 | 38.30 | 17,649 | 39.43 | 18,172 |
10 | North Delhi | 59 | 7.82 | 13,256 | 8.88 | 14,557 |
Apart from the trends and patterns of urbanization and population density, the composition of population and income distribution structure in urban India also favors the use of public transport for living and working. An overwhelming majority in cities belongs to the poor, low and lower-middle income groups. The Global Wealth Report 2015 published by Credit Suisse suggests that more than 90 percent of the adult population in India fall below the bottom of the wealth pyramid (less than $10,000). The middle class population in India, defined as those with annual wealth of about Rs.61,480 or $13,662 is estimated at 23.6 million [6]. About one-fourth of urbanites have been identifies to be below the poverty line. An equivalent number are slum dwellers. More than 65 percent of urban households lives in two rooms or less.
Table 6 presents a picture of urban poverty vis-à-vis rural poverty in India based on the Rangarajan Committee report. According to the Committee, a person spending less than Rs.1407 per month or Rs.47 a day was considered poor in cities in 2011–12. The number of urban poor was estimated at 102.5 million, accounting for 26 percent of the urban population in the same year.
Year | Poverty ratio (%) | No. of poor (million) | ||||
---|---|---|---|---|---|---|
Rural | Urban | Total | Rural | Urban | Total | |
1. 2009–10 | 39.6 | 35.1 | 38.2 | 325.9 | 128.7 | 454.6 |
2. 2011–12 | 30.9 | 26.4 | 29.5 | 260.5 | 102.5 | 363.0 |
3. Reduction | 8.7 | 8.7 | 8.7 | 65.4 | 26.2 | 91.6 |
India: Rural and urban poverty estimates 2009–10 and 2011–12.
Census 2001 estimated the urban slum population in India at 42.6 million. It reported that 41.6 percent of slum population in the country lived in metropolitan cities. Mumbai had the largest number of slum dwellers, accounting for 54 percent of the population. Census 2011 has placed the number of slum-dwellers in India at 65.5 million. It further reveals that 46 million-plus cities contain 38 percent of the slum households. 9 metropolitan cities have more than 30 percent of households in slums, with Visakhapatnam topping the list at 44.1 percent, followed by Jabalpur Cantonment Board (43.1 percent) and Greater Mumbai (41.3 percent). Among the largest municipal corporations, apart from Greater Mumbai, Kolkata and Chennai have reported more than 25 percent of households living in slums.
The trends of urbanization, patterns of population density and state of slums, poverty and housing in cities suggest that the demographic and income distribution structures of urban India are overwhelmingly suitable for a public transportation-led model of urban development. Transportation planners and traffic engineers advocate the following strategies for urban transportation depending on their peak hour per direction traffic (PHPDT) that significantly depend upon the density of commuters:
PHPDT Recommended strategy
10,000 - 15,000 Bus and Dedicated Busways
15,000 - 30,000 Light Rail Transit
> 30,000 Heavy Rail Mass Transit
Based on the above criteria and other factors, many cities in India qualify for light rail transit and heavy rail transit. The largest metropolitan cities also need high speed rail connecting them to sub-urban centres and regional towns.
The number of registered motor vehicles in India increased from 0.3 million in 1951 to 55 million in 2001 and 210 million in 2015. While the share of two wheelers rose from 8.8 percent in 1951 to 73.5 percent in 2015, the share of busses declined from 11 percent to 1 percent. Table 7 presents the trends in the number of motor vehicles and the composition of the vehicular population for the period 1951–2015.
Year | Number in Million | Composition (% of Total Vehicle Population) | ||||
---|---|---|---|---|---|---|
Two Wheelers | Cars, Jeeps and Taxis | Busses | Goods vehicles | Other vehicles | ||
1951 | 0.3 | 8.8 | 52.0 | 11.0 | 26.8 | 1.3 |
1961 | 0.7 | 13.2 | 46.6 | 8.6 | 25.3 | 6.3 |
1971 | 1.9 | 30.9 | 36.6 | 5.0 | 18.4 | 9.1 |
1981 | 5.4 | 48.6 | 21.5 | 3.0 | 10.3 | 16.6 |
1991 | 21.4 | 66.4 | 13.8 | 1.5 | 6.3 | 11.9 |
2001 | 55.0 | 70.1 | 12.8 | 1.2 | 5.4 | 10.5 |
2006 | 89.6 | 72.2 | 12.9 | 1.1 | 4.9 | 8.8 |
2011 | 141.8 | 71.8 | 13.6 | 1.1 | 5.0 | 8.5 |
2012 | 159.5 | 72.4 | 13.5 | 1.0 | 4.8 | 8.3 |
2013 | 176.0 | 72.7 | 13.6 | 1.0 | 4.7 | 8.0 |
2014 | 190.7 | 73.1 | 13.6 | 1.0 | 4.6 | 7.7 |
2015 | 210.0 | 73.5 | 13.6 | 1.0 | 4.4 | 7.5 |
Total number of registered motor vehicles in India (in million) 1951–2015.
The population of motor vehicles reported by million-plus cities in India in 2015 was 66.24 million. Among these, Delhi had the highest number at 88.51 lakhs, followed by Bengaluru (55.60 lakhs), Chennai (49.34 lakhs), Ahmedabad (34.20 lakhs), Greater Mumbai (25.71 lakhs), Surat (24.59 lakhs), Hyderabad (23.69 lakhs), Pune (23.37 lakhs), and Jaipur (22.49 lakhs). The largest number of two-wheelers in 2015 was in Delhi at 56.98 lakhs, followed by Bengaluru (38.41 lakhs), Chennai (35.16 lakhs), Ahmedabad (24.32 lakhs), Surat (19.13 lakhs); Pune (17.65 lakhs); Hyderabad (17.08 lakhs); Jaipur (16.58 lakhs) and Greater Mumbai (14.70 lakhs). Considering the quantum of cars in 2015, Delhi had 27.30 lakhs, followed by Bengaluru (10.89 lakhs), Chennai (8.60 lakhs), Greater Mumbai (7.97 lakhs), Kolkata (5.41 lakhs), Ahmedabad (5.26 lakhs), Hyderabad (4.02 lakhs) and Pune (3.75 lakhs). Table 8 shows the number and share of two wheelers and cars in the population of motor vehicles for metropolitan cities as of 31st March 2015.
Million Plus Cities | Total Number of Registered Motor Vehicles | Two Wheeler | Cars | ||
---|---|---|---|---|---|
Number | % of Total | Number | % of Total | ||
Agra | 9,05,023 | 7,41,778 | 81.96 | 76,107 | 8.41 |
Ahmedabad | 34,19,828 | 24,31,839 | 71.11 | 5,25,891 | 15.38 |
Allahabad | 8,97,035 | 7,30,758 | 81.46 | 72,779 | 8.11 |
Aurangabad | 4,26,246 | 3,35,725 | 78.76 | 19,591 | 4.60 |
Bengaluru | 55,59,730 | 38,41,139 | 69.09 | 10,88,587 | 19.58 |
Bhopal | 10,80,477 | 8,47,334 | 78.42 | 1,36,627 | 12.65 |
Chandigarh | 7,45,520 | 3,95,565 | 53.06 | 2,61,752 | 35.11 |
Chennai | 49,34,412 | 35,16,062 | 71.26 | 8,60,932 | 17.45 |
Coimbatore | 19,01,277 | 15,47,395 | 81.39 | 2,32,751 | 12.24 |
Delhi | 88,50,720 | 56,98,242 | 64.38 | 27,30,071 | 30.85 |
Dhanbad | 5,63,426 | 4,27,714 | 75.91 | 58,836 | 10.44 |
Durg-Bhillai | 7,68,922 | 6,44,138 | 83.77 | 49,569 | 6.45 |
Ghaziabad | 7,51,603 | 5,33,808 | 71.02 | 1,52,256 | 20.26 |
Greater Mumbai | 25,71,204 | 14,70,175 | 57.18 | 7,97,267 | 31.01 |
Gwalior | 6,17,681 | 4,87,259 | 78.89 | 52,685 | 8.53 |
Hyderabad | 23,68,818 | 17,07,714 | 72.09 | 4,02,334 | 16.98 |
Indore | 17,12,702 | 13,01,383 | 75.98 | 2,08,005 | 12.14 |
Jabalpur | 6,38,219 | 4,93,633 | 77.35 | 67,445 | 10.57 |
Jaipur | 22,49,240 | 16,58,006 | 73.71 | 3,05,445 | 13.58 |
Jamshedpur | 4,72,051 | 3,51,696 | 74.50 | 55,020 | 11.66 |
Jodhpur | 9,16,172 | 6,50,097 | 70.96 | 71,972 | 7.86 |
Kannur | 1,88,497 | 1,12,851 | 59.87 | 43,920 | 23.30 |
Kanpur | 14,61,530 | 11,72,577 | 80.23 | 1,47,072 | 10.06 |
Kochi | 6,05,689 | 3,36,316 | 55.53 | 1,71,063 | 28.24 |
Kolkata | 14,01,638 | 6,00,156 | 42.82 | 5,41,432 | 38.63 |
Kollam | 2,74,006 | 1,75,528 | 64.06 | 58,097 | 21.20 |
Kota | 6,54,041 | 5,12,740 | 78.40 | 51,749 | 7.91 |
Kozhikode | 4,12,304 | 2,89,801 | 70.29 | 70,539 | 17.11 |
Lucknow | 17,09,662 | 13,61,787 | 79.65 | 2,44,121 | 14.28 |
Madurai | 9,54,893 | 7,93,510 | 83.10 | 68,804 | 7.21 |
Malappuram | 2,76,765 | 1,51,351 | 54.69 | 59,297 | 21.43 |
Meerut | 5,25,235 | 4,24,975 | 80.91 | 63,148 | 12.02 |
Nagpur | 12,75,575 | 10,67,160 | 83.66 | 1,08,951 | 8.54 |
Nashik | 6,22,206 | 4,61,628 | 74.19 | 62,473 | 10.04 |
Patna | 10,18,798 | 7,05,298 | 69.23 | 1,35,638 | 13.31 |
Pune | 23,37,085 | 17,65,172 | 75.53 | 3,75,267 | 16.06 |
Raipur | 11,11,745 | 8,45,861 | 76.08 | 84,377 | 7.59 |
Rajkot | 9,79,423 | 7,87,608 | 80.42 | 93,185 | 9.51 |
Ranchi | 5,47,036 | 3,56,067 | 65.09 | 65,434 | 11.96 |
Srinagar | 2,35,614 | 1,00,291 | 42.57 | 77,043 | 32.70 |
Surat | 24,59,111 | 19,12,715 | 77.78 | 3,07,540 | 12.51 |
Trichy | 7,63,396 | 6,36,961 | 83.44 | 58,712 | 7.69 |
Thiruvananthapuram | 5,71,956 | 3,49,657 | 61.13 | 1,53,674 | 26.87 |
Thrissur | 3,55,491 | 2,26,285 | 63.65 | 72,994 | 20.53 |
Varanasi | 7,68,769 | 6,09,656 | 79.30 | 55,727 | 7.25 |
Vijayawada | 6,10,321 | 4,52,403 | 74.13 | 53,755 | 8.81 |
Vadodara | 10,41,818 | 8,03,969 | 77.17 | 1,23,509 | 11.86 |
Visakhapatnam | 7,30,872 | 5,74,135 | 78.55 | 79,592 | 10.89 |
Total | 6,62,43,782 | 4,73,97,918 | 71.55 | 1,16,53,035 | 17.59 |
Share of two wheelers and cars in total number of registered motor vehicles in million plus cities of India as on 31st March 2015.
Table 9 presents the growth of motor vehicle population in 22 metropolitan cities in India over the period 2005–15 for which data are available. As the table shows 16 out of 22 metropolitan cities recorded more than 10 percent annual growth over the period; 3 cities had an annual growth rate exceeding 20 percent.
Metropolitan City | No. of Motor Vehicles (in Thousands) | Average Annual Growth (%) | |
---|---|---|---|
2005 | 2015 | ||
Ahmedabad | 1632 | 3420 | 10.96 |
Bengaluru | 2232 | 5560 | 14.91 |
Bhopal | 428 | 1080 | 15.23 |
Chennai | 2167 | 4934 | 12.77 |
Coimbatore | 682 | 1901 | 17.87 |
Delhi | 4186 | 8851 | 11.14 |
Greater Mumbai | 1295 | 2571 | 9.85 |
Hyderabad | 1433 | 2369 | 6.53 |
Indore | 705 | 1713 | 14.30 |
Jaipur | 923 | 2249 | 14.37 |
Kanpur | 425 | 1462 | 24.40 |
Kochi | 166 | 606 | 26.51 |
Kolkata | 911 | 1402 | 5.39 |
Lucknow | 615 | 1710 | 17.80 |
Madurai | 330 | 955 | 18.94 |
Nagpur | 770 | 1276 | 6.57 |
Patna | 378 | 1019 | 16.96 |
Pune | 827 | 2337 | 18.26 |
Surat | 692 | 2459 | 25.53 |
Varanasi | 366 | 769 | 11.01 |
Vadodara | 586 | 1042 | 7.78 |
Visakhapatnam | 435 | 731 | 6.80 |
Growth in number of registered motor vehicles in select metropolitan cities 2005–2015.
The car-penetration rate defined as the number of cars per 1000 persons is very small in India compared to that in developed countries and several developing countries. Table 10 compares data on Gross National Income (GNI) and vehicular penetration rates for select countries with those for India.
Country | GNI per capita (US$) for 2013 | Number per 1000 persons | ||
---|---|---|---|---|
Passengers Cars | Total Vehicles | Two-wheelers | ||
Developed Countries | ||||
United States | 53,470 | 360 | 783 | 27 |
United Kingdom | 41,680 | 455 | 517 | 19 |
Japan | 46,330 | 466 | 598 | 81 |
Germany | 47,270 | 544 | 603 | 50 |
Australia | 63,390 | 562 | 711 | 32 |
Developing Countries | ||||
Mexico | 9940 | 203 | 285 | 15 |
Malaysia | 10,430 | 358* | 396* | 356 |
South Africa | 7190 | 110** | 162** | 6 |
Brazil | 11,690 | 227 | 290 | 108 |
China | 6560 | 76 | 93 | 70 |
South Korea | 25,920 | 300 | 386 | 42 |
India | 1570 | 19 | 167 | 123 |
Vehicular penetration rates in select developed and developing countries 2013.
Data relates to 2012.
Date relates to 2011.
The data in the above table suggest that with the rise in GNI, following structural transformation and economic growth, the vehicular penetration rate, with attendant problems of congestion, pollution, noise and carbon emissions in cities, will lead to increased demand for road space and public transport, including rail-based transit.
Ironically, many of India’s urban mobility problems can be traced to the lack of an appropriate planning model and public transport development strategy rooted in the economics of cities. In particular, cities have not exploited the links between agglomeration externalities and transportation in their spatial planning and development models. Land use planning and transportation planning have been pursued as disjointed exercises in India. Cities had land use planners, but no transport planners. As a result, they have not been able to harness the power of city externalities to guide transport-land use integration and local economic development, address congestion and raise resources to finance public transport. The trends of urbanization, metropolitanization and motorization; patterns of population composition and densities in cities; abysmal state of urban transport with no robust model of financing in sight; emerging energy security and environmental concerns; andthe demands of inclusive economic growth in India call for exploring the principles of New Urbanism, Smart Growth and TOD for restructuring urban planning.
New Urbanism and Smart Growth emerged in the last four decades in the United States, Europe and other developed countries in response to their problems of urban sprawl, a consequence of automobile-dependency. They are rooted in a search for alternatives to low-density, single-use and spread-out patterns of urban expansion, increasing traffic congestion and air pollution, and adversely impacting the environment and quality of life.
New Urbanism is a design-oriented with architectural roots. Promoted by architects, it is focused on neighborhood design. Smart Growth is policy-oriented with environmental roots. Spearheaded by planners, it is centered on promoting guided development. Smart Growth is not so much concerned with urban design as it is with growth promotion. It elevates the discourse on urban planning from growth control to issues of how and where growth should be accommodated. It calls for public subsidies for growth, such as infrastructure facilities and land use incentives. Both New Urbanism and Smart Growth advocate TOD.
Transit Oriented Development (TOD) owes its origin to the paradigms of New Urbanism and Smart Growth. It is an urban planning and development approach aimed at creating vibrant, livable and sustainable communities by concentrating growth around one or more transit stations or within a transit corridor. It emphasizes compact, walkable, mixed-use communities with access to high quality transit services within a walking distance. TOD principles are not new; they were introduced by many cities in North America and Australia into their planning models after World War II. However, TOD as a specific policy paradigm has taken root only in the last twenty years.
The concentration of development based on a TOD approach acts against urban sprawl and uneconomic extension of costly infrastructure, catalyzes external economies of agglomeration, mitigates congestion diseconomies, and assists in the mobilization of resources through increases in land and property values and other tax bases. TOD enables lower-stress living without complete dependence on a car for mobility. It is environment-friendly and inclusive. The poor, who do not own automobiles benefit significantly when included under a TOD scheme. As an instrument of inclusive regional and urban planning, TOD promotes the inclusion of the poor in the urban development process. The economic, social and environmental benefits of TOD are briefly presented below:
The economic benefits of TOD include reduced congestion, agglomeration economies, resource mobilization for financing infrastructure, reduced costs of development, efficiency of investment, etc.
Reduced Congestion: TOD reduces the need to travel and, thus, reduces congestion and stress levels.
Agglomeration Economies: TOD, if designed properly, can augment agglomeration economies by enhancing access to the economic mass and facilitating the collocation of productive economic activities in nodes with potential to engineer growth. These economies lead to benefits of backward and forward linkages, market access, sharing of common infrastructure facilities and resources, specialized labour pooling, human capital accumulation, knowledge spillovers and networking. They lead to economies of sharing, matching and learning; they promote specialization, diversity and competition.
Increased Revenue Yields: Properties around transit hubs are accorded higher values. These higher property values could be converted into revenue for the government through value capture levies.
Efficiency of Investment: TOD directly fosters patronage for growth and helps to optimize existing transit and connectivity infrastructure. It maximizes the efficiency and carrying capacity of the transportation network.
TOD: Social Benefits.
Affordable housing and public transport are key enablers of social inclusion. They increase the accessibility to jobs, health care, education, recreation and socio-cultural interactions.
TOD: Environmental Benefits.
Public transport can help to reduce the proliferation of personal vehicles and thus, reduce the level of emissions. This reduction could be quite significant, especially during the peak hours.
The success of TOD depends on its design. Box 1 presents some key principles to guide TOD designing.
Transit-oriented development: design principles.
1. Multimodal Transit Station.
Transit is the focus of TOD.Transit facilities should not be designed in isolation, rather it should connect the neighborhoods. Further, it should include a mix of modes like two wheelers, car, bicycles, BRT, LRT and NMT.
2. Interconnected Streets.
Such a pattern not only decreases congestion but also encourages mixed use development along with enhanced travel choices.
3. Mixed Use Development.
A compact structure involving diverse land use pattern can benefit residents as well as workers to meet their daily requirements including work, shopping and leisure.
4. Walkability.
In order to encourage walking it is important to design a pedestrian-friendly structure. Such a structure must include sidewalks, shaded pedestrian routes, benches to rest and safe crossing points at transit stations.
5. Compact Development.
In order to be successful, the structure needs to be compact. The extent of neighborhoods around transit nodes is based on a comfortable walking distance from edge to centre (approximately 400 to 800 meters in radius).
6. Street-facing Buildings.
Streets can be better defined by placing the buildings near them. Street front retail should be provided to humanize the building wall and activate the sidewalk.
7. Urban Place-making.
A successful TOD design works on developing public spaces in the neighborhood. It is important for improving social interaction and strengthening community bonds and participation.
8. Neighborhood High Street.
Retail streets provide the goods and services of daily life, activate the street, reduce auto reliance, and increase ownership and safety of the pedestrian realm.
9. Streetscape Design.
A beautified street pattern equipped with pedestrian utilities improves the desire to walk and makes it pleasant while shortening the sense of distance.
10. Bicycle-friendly Streets / Parking.
Bicyles are environment friendly and efficient alternatives to automobiles. Bike lanes, bike routes, and secure parking make the bicycle an easy option.
11. Urban Parks & Plazas with Minimized Ecological Footprint.
Open spaces enable public interaction and promote healthy communities.
12. A Well-designed Transit Station for a High Quality User Experience.
The design of the transit station is at the heart of a successful TOD structure. Its design is critical for enhancing customer attraction and ensuring seamless and efficient accessibility to consumers.
13. Reduced Parking Standards.
Reducing parking standards provides increased site area for alternative public amenities.
14. Safety & Security.
Ensuring safety and security of transit users especially pedestrians, not only improves the transit experience but also enhances transit ridership.
15. Market Acceptance and Successful Implementation.
A vibrant and transit supportive space which attracts several jobs and residents is critical for a TOD programme. Flexible strategies along with designs which cater to the needs of the surrounding neighborhood can ensure a successful TOD.
While the principles of Smart Growth and TOD originated in developed countries in response to their problems of sprawl, the paradigms make good sense for developing countries like India. However, TOD policies have not been implemented in an appreciable way in India. Only recently Delhi and Haryana have brought out planning guidelines for TOD, calling for the integration of transportation and land use. Bengaluru, Mumbai, Pimpri-Chinchwad, Ahmedabad. Hyderabad, Naya Raipur and Bhubaneswar have embarked on programmes to promote transit-oriented planning and development. TOD presents significant opportunities to India to make the country’s urbanization process efficient, inclusive and sustainable. However, the execution of TOD and financing of transit investments are key challenges for Indian cities. Apart from the principles of sustainable development, successful international practices of transport-land use and integrationapproaches to financing public transport investments can guide the design of TOD in India. Section 4 refers to some oft-cited examples of successful international practices of TOD. Section 5 presents the broad approaches to financing of public transport, including transit to guide Indian cities to draw lessons for TOD.
TOD is emerging as a preferred paradigm to plan cities, localities and urban extensions and renew old cities and derelict areas within cities in many countries. Some of the successful TOD models practiced internationally that can provide lessons for Indian cities for the integration of transportation and land use are discussed in this section.
Hong Kong is internationally known for its successful integration of rail transit investments and urban development. The integrated “rail-property” development model (R + P), plays a vital role in managing and financing railway expansion, advancing high-quality urban designs, creating “one-stop” settings for “live-work-shop-play”, guiding regional urban growth, and more. As with all good public- private partnerships, this occurs in a win-win fashion – i.e., the railway corporation reaps financial benefits and society at-large benefits from more sustainable, transit-oriented patterns of development. Maritime Square Residential-Retail Development atop Tsing Yi Station provides a good example of Hong Kong TOD. Maritime Square features hierarchically integrated uses. Shopping mall extends from the ground floor to the 3rd level. Station concourse sits on the 1st floor, with rail lines and platforms above and ancillary/logistical functions (like public transport/bus interchange and parking) at or below. Above the 4th and 5th floor residential parking lies a podium garden and above this, high-rise, luxury residential towers [10].
The Hong Kong Government derives a major proportion of its revenues from land, including premium on new land and modification of existing leases, property taxes, stamp duty, rents, etc. [11]. The Hong Kong MTR has generated many benefits to the community. These include travel time saving, employment gains, environmental health benefits, property value increases and so on. The network obviously generates enormous external benefits as it passes through the densely populated districts, commercial and employment centres and carries large passenger loads.
Bogota, the capital of Colombia, has some of the most progressive public investment initiatives in developing countries, including the first-class TransMilenio BRT; integrated TDM measures; the transit-linked social housing Metrovivienda program; the Alameda Porvenir, the world’s longest pedestrian way; and other public projects that incorporate good urban design and innovative financing schemes [12].Bogota’s TransMilenio is one of the world’s most successful examples of Bus Rapid Transit (BRT) [13]. It is characterized by dedicated main trunk routes for high speed busses, physically separated from the rest of traffic [14]. The bus stations are well-connected with systematic feeder services. The integrated approach of Bogota addressing affordable housing and affordable transport simultaneously, has improved the access to work, leisure, recreation, shopping.
Curitiba’s bus system is composed of a hierarchical system of services. Minibusses routed through residential neighborhoods feed passengers to conventional busses on circumferential routes around the central city and on inter-district routes. The backbone of the system is composed of the Bus Rapid Transit, operating on the five main arteries leading into the centre of the city like spokes on a wheel hub [15]. Along each of the five arteries there is a trinary road system, comprised of middle express bus lane with vehicle lanes on each side for local auto traffic and parking.
Curitiba’s Master Plan integrated transportation with land use planning. It limited central area growth, while encouraging commercial growth along the transport arteries radiating out from the city centre. The city centre was partly closed to vehicular traffic, and pedestrian streets were created. Rush hour in Curitiba has heavy commuter movements in both directions along the public transportation arteries.
Danish Town Planning Institute created the “Egnsplan” or the Finger Plan in 1947. It was based on a TOD principle, with mixed land use and high-density areas around the centre [16]. Shopping malls, offices, recreational centres and housing were all planned in pedestrian areas with good bicycle facilities such as cycle lanes and parking and a good connection to public transport. The design includes five fingers or corridors of urban development along the suburban areas which are connected through railway lines and would directly connect the areas to Copenhagen Central Business District (CBD). The neighborhoods around the transit stations were planned to be developed in a TOD fashion with high density housing and amenities. The approach aimed at an ordered and integrated ‘green’ growth and was developed at the time of extensive and rapid urban development. There were spaces left for the use of farmland and recreational purposes between each finger, known as “green wedges”. A ring road was planned at the end of each finger which linked the Copenhagen harbor and inner city to industrial locations. Most of the land was developed by the end of the 1960s and the two southern-most fingers were extended.
Orestadtownship is one of the best examples of successful TOD following the Finger Plan. It combines economic activities, housing and amenities – jobs, housing, retail, leisure and education – all based on TOD. It helped Copenhagen to remain competitive and release pressure on CBD.
Unlike the international cities with global best practices on TOD, Indian cities have grossly neglected transportation planning, public transport investments and transport-land use integration for long. Key issues of financing public transit and development integrated with such transit are typically ignored in public discourses. As a result, a coherent strategy for financing public transport has not emerged in India. Section 5 refers to international practices for financing of transit oriented development in the broader concept of financing public transport to guide Indian cities.
The financing of TOD cannot be artificially divorced from the broader issues of financing urban transport and cities. Both planning and economic considerations are important for designing a financing strategy. The approaches to financing of various types of public transport infrastructure, including TOD internationally include the following methods:
Equity, including public-private partnerships (PPP), special purpose vehicles, infrastructure debt funds, investment funds, infrastructure financing companies.
Debt tools, including private debt, commercial bank debt, take-out financing, bond financing – infrastructure bonds, municipal bonds (revenue and general obligation), green bonds, etc.
Foreign Direct Investment and Foreign Portfolio Investment.
Grant financing, combining central and state grants with local government resource mobilization and using public funds to leverage market resources and PPP.
Direct fees, including user fees, utility fees, benefit charges and congestion pricing.
Using land as a resource - value capture and impact instruments such as land and property taxes, land value tax, land value increment tax, betterment levy, developer exactions, impact fees, special assessment districts, land readjustment, town planning scheme, joint development, land monetization including the lease and sale of land and air rights with enhanced Floor Space Index and value-enhancing land use changes in TOD zones, tax increment financing, etc.
Bullet Bonds and Pooled Finance Fund Scheme.
Land value capture (LVC) instruments take many forms and can be classified into two major types: (i) tax- or fee-based and (ii) non-tax- or non-fee-based, also called “development-based LVC.” Tax- or fee-based instruments capture land value increases through, for example, land and property taxes, betterment charges, special assessments, and tax increment financing. In contrast, development-based LVC instruments capture these increments through land-related transactions such as selling or leasing land, development rights and air rights; making land readjustments; and redeveloping urban areas [17]. If adapted well to local contexts, development-based LVC instruments can be an effective finance and planning mechanism for cities in India.
The issues of financing public transit and TOD are intricately connected. However, as Indian cities are struggling to finance the development of mass rapid transit and bus rapid transit systems, not many have focused on TOD funding linked to LVC and non-LVC instruments. Based on international experience, a combination of financing instruments needs to be considered for adoption in India. These have to be suitably customized to fit the context of cities. A summary of various potential options for funding public transport, including transit is presented in the table below (Table 11).
Sl. No. | Name | Description | Advantages | Disadvantages |
---|---|---|---|---|
1. | Fare increases | Increase fares or change fare structure to increase revenues | Widely applied. Is a user fee (considered equitable) | Discourage transit use. Is regressive. |
2. | Discounted bulk passes | Discounted passes sold to groups based on their ridership | Increases revenue and transit ridership | Increases transit service costs and so may provide little net revenue |
3. | Property taxes | Increase local property taxes | Widely applied. Distributes burden widely. | Supports no other objectives. Is considered regressive. |
4. | Sales taxes | A special local sales tax | Distributes burden widely. | Supports no other objectives. Is regressive. |
5. | Income tax | Special income tax for transit or transportation | Progressive with respect to income. Relatively stable. | May be difficult to implement. |
6. | Fuel taxes | An additional fuel tax in the region | Widely Applied. Reduces vehicle traffic and fuel use | Is considered regressive. |
7. | Vehicle fees | An additional fee for vehicles registered in the region | Applied in some jurisdictions. Charges motorists for costs. | Does not affect vehicle use. |
8. | Utility levy | A levy to all utility accounts in the region | Easy to apply. Distributes burden widely. | Is small, regressive and support no other objectives. |
9. | Employee levy | A levy on each employee within a designated area or jurisdiction | Charges for commuters. | Requires administration. Encourage sprawl if in city centers. |
10. | Road tolls | Tolls on some roads or bridges | Reduces traffic congestion. | Costly to implement. Can encourage sprawl if only applied in city centers. |
11. | Vehicle-Km tax | A distance-based fee on vehicles registered in the region | Reduces vehicle traffic. | Costly to implement. |
12. | Parking taxes | Special tax on commercial parking transactions | Is applied in other cities. | Discourages parking pricing and downtown development. |
13. | Parking levy | Special property tax on parking spaces throughout the region. | Large potential. Distributes burden widely supports strategic goals. | Costly to implement. Opposed by suburban property owners. |
14. | Expanded parking pricing | Increase when and where public parking facilities (e.g. on-street parking) are priced | Moderate to large potential. Distributes burden widely. Reduces parking & traffic problems. | Requires parking meters and enforcement, and imposes transaction costs. |
15. | Development or transport impact fees | A fee on new development to help finance infrastructure, including transit improvements. | Charges beneficiaries. | Limited potential. |
16. | Land value capture | Special taxes on property that benefit from the transit service | Large potential. Charges beneficiaries. | May be costly to implement. May discourage TOD. |
17. | Station rents | Collect revenues from public private development at stations | Charges beneficiaries. | Limited potential. |
18. | Station air rights | Sell the rights to build over transit stations. | Charges beneficiaries. | Limited potential. |
19. | Advertising | Additional advertising on vehicles and stations. | Already used. | Limited potential. Sometimes unattractive. |
International experience suggests that no one size fits all. But it makes clear that public transit and TOD impact on local, regional and national economies and lead to enhanced tax bases of all governments. Thus, if they are financed by borrowed funds with repayment linked to a value creation, capture and recycling strategy, cities in India can hope to get out of their vicious circles and traverse on a path of planned development. Future tax increments can finance current investment programmes which augments tax bases.
Some state governments and urban local bodies in India have resorted to novel initiatives to plan and implement projects aimed at improving urban mobility following the TOD principle and Smart Growth framework. Some case studies are presented below.
The city has decided to develop and implement an integrated public transit system including:
A Suburban Rail Transit System to connect the city with its industrial suburbs such as Kalol, Naroda, Mehmedabad, etc.
A Metro Rail System to cater to the high intensity movement between Ahmedabad and Gandhinagar.
A Bus Rapid Transit System (BRTS) to cater to major mobility needs of the city.
A regular bus system to support BRTS.
Decentralized Regional Bus & Rail Terminal.
Integrate different form of transport, i.e., BRT with other regional and urban transport systems, with bicycles and pedestrian facilities.
Integration of Land Use -Transport elements like increased FSI along BRTS corridor.
The primary objective of the integrated public transit initiative in Ahmedabad is to make the city more accessible – with physical, social and economic accessibility.
Ahmedabad city has developed a Bus Rapid Transit System under the Jawaharlal Nehru National Urban Renewal Mission (JNNURM) with the name “Janmarg” or “the people’s way” and the slogan ‘Accessible Ahmedabad’.Janmarg boasts of an innovative plan anddesign which includes pedestrian only sections and one-way bus lane etc.
Bangalore Metro Rail Corporation Limited (BMRCL), a joint venture of Government of India and Government of Karnataka is a Special Purpose Vehicle entrusted with the responsibility of implementation of the Bengaluru Metro Rail Project. “Namma Metro” is an environment friendly initiative as it aims at reducing carbon emissions in the city. The project has an East–West corridor - 18.10 km long, starting from Baiyappanahalli in the East and terminating at Mysore Road terminal in the West and a 24.20 km North–South corridor commencing at Nagasandra in the North and terminating at Puttenahalli in the South.
In connection with the construction of Bengaluru Mass Rapid Transit System, the Government of Karnataka has introduced a number of innovative measures to create a dedicated resource pool, including special cess to capture land value increments due to transit [19]. The Government has taken up several value capture instruments like development ceases, chess on additional FAR, Transferable Development Right (TDR) etc. to finance Bengaluru mass rapid transit system.
The Delhi Metro system serves Delhi and its satellite cities of Faridabad, Gurgaon, Noida and Ghaziabad in National Capital Region in India. Delhi Metro is the world’s 12th largest metro system in terms of both length and number of stations. The network consists of five color-coded regular lines and the faster Airport Express line, with a total length of 213 kilometers serving 160 stations (including 6 on Airport Express line) [20]. The system has a mix of underground, at-grade, and elevated stations using both broad-gauge and standard-gauge. The metro generated an average daily ridership of 2.661 million passengers.
Delhi Metro has been instrumental in reducing vehicular congestion on the roads. According to a study, Delhi Metro has helped in removing about 3.9 lakh vehicles from the streets of Delhi. The Delhi Metro Rail Corporation has been certified by the United Nations as the first metro rail and rail-based system in the world to get “carbon credits for reducing greenhouse gas emissions” and helping in reducing pollution levels in the city by 630,000 tonnes every year, thus helping in reducing global warming [21].
The Delhi TOD Policy 2013 has provided for significant increases in FSI in transit influence zones to promote intensive development so that TOD can be self-financing adopting a land value capture method and even be surplus-generating. The Delhi Development Authority has proposed to take up TOD to build the East Delhi Hub as a signature destination. This includes the development of 75 acres of land in Karkardooma with FSI raised to a maximum of 4 and maximum density of 2000 persons per hectare on the basis of a TOD model. The project is being taken up on a partnership with the National Building Construction Company (NBCC). Some parcels of land are under development, but Delhi is far from achieving the TOD Policy objectives of inclusive development.
The salient features of the Delhi TOD Policy are:
Development/redevelopment in TOD zone will be incentivized by providing significantly higher FAR of 4.0 on the entire amalgamated plot being developed/redeveloped.
Additional FAR may be availed only through Transferable Development Rights (TDR), for schemes larger than 1 hectare.
Entire approved layout plan of a scheme will be included in the influence zone if more than 50 percent of the plan area falls in the influence zone.
It will be mandatory to use a minimum of 30 percent of overall FAR for residential use, a minimum of 10 percent of FAR for commercial use and a minimum of 10 percent of FAR for community facilities. Utilization of the remaining 50 percent FAR shall be as per the land use category designated in the Zonal Plan.
There shall be a mix of housing types for a wide range of income brackets within communities with shared public spaces/greens/recreational facilities/amenities, which will minimize gentrification and create more community-oriented developments.
The mandatory residential component covering 30 percent FAR shall wholly comprise of units of 65 m2 area or less. Out of the half of the FAR, i.e. 15 percent of the total FAR, has to be used for units of size ranging between 32 and 40 m2. Over and above this, an additional mandatory FAR of 15 percent, i.e. FAR of 0.6 (out of 4.0) has to be utilized for Economically Weaker Sections (EWS). The size of EWS units will range between 32 and 40 m2.
20 percent of land shall be used for roads/circulation areas. 20 percent area for green open space shall be kept open for general public use at all times. Further, 10 percent of green area may be for exclusive use.
MRTS agencies are exempted from providing the mandatory 30 percent residential component which is part of the TOD norms applicable to all other developer entities (DEs).
Hyderabad has gone for a metro rail transit based on project report prepared by Delhi Metro Rail Corporation (DMRC) which identified 269 acres of land requirement. Originally the project was conceived as a government-funded project. However, subsequently the city went for a metro based on a PPP mode, adopting a Design, Build, Finance, Operate and Transfer (DBFOT) format. Hyderabad is currently implementing the world’s largest elevated metro rail project in PPP mode with L&T as concessionaire.
The revenue model of the concessionaire is: 55 percent passenger fare, 40 percent property development and 5 percent advertisement and parking fees. Thus, the project’s revenue is partially fare based. But, a significant portion of the revenue is also non-fare based. The concessionaire hugely relied on the potential of development of property or air space above and around transit stations. It has been provided with some valuable government land at vantage transit stations and is undertaking commercial exploitation of property with engineering innovations. It cannot sell property but can enjoy the rentals during the concession period of 35 years.
Mumbai Metro Line 1 – Versova-Andheri-Ghatkopar Mass Rapid Transit System is the first metro project awarded in the country on a PPP basis. It has provided the much needed connectivity in the financial capital of India linking the East and the West. It has ensured connectivity to Western and Central Railways. Providing modern, fast, clean and caring infrastructure, the Line has carried 100 million commuters in the first year of operation. It has reduced the journey time between Versova and Ghatkopar from 71 minutes to 21 minutes.
While Indian cities have making efforts to promote TOD with new policies, projects and plans emerging, especially in the context of Smart Cities Mission, a study of the Indian initiatives so far suggests robust approaches to financing and execution of TOD have not emerged. The approaches to financing transit also vary considerably as shown in Table 12 below.
Funding Approach | Financing Pattern | Practicing Metro Rail |
---|---|---|
Government-funded | 50–50 Central Government: State Government | Delhi, Bengaluru, Chennai, Kochi, Nagpur |
100 percent State Government | Jaipur, Lucknow (initially) | |
100 percent Central Government | Kolkata (North –South) Kolkata (East–West) | |
Public-Private Partnerships | Private provisioning of operation and maintenance | Delhi Airport Express (initially) – Reliance Infra |
PPP-BOT model (Design, Build, Finance, Operate, Transfer) | Hyderabad Metro (Government of India Viability Gap Funding – 10%, L&T – 20% equity and 70% debt)) Mumbai Metro Line 1 (RInfra −69% MMRDA – 26%, Veolia – 5%) | |
Private Funding | Complete private funding | Gurgaon Rapid Metro Phase I – Equity 75%, DLF 25% Phase II – Senior Debt from Banks/ Financial Institutions – 70%, Sponsor’s Contribution – 30% |
Emerging approaches to financing public transit in India.
A key lesson from the initiatives of Indian cities towards financing transit and TOD, when compared to international best cases, is that urban policy, spatial planning, city development strategy, city financing framework, transit orientation, zoning, land use and development control regulations and institutional framework to integrate land use and transportation planning, raise resources and execute TOD need to be part of a holistic model of integrated urban development and should not be undertaken disjointedly.
The existing institutional framework in Indian cities is not adequate to tackle the issues associated with planning, financing and implementing TOD. The starting point for successfully implementing TOD in India is to devise an appropriate institutional framework along with clarity in financing mechanisms. Considering the investment and planning efforts demanded by TOD, an effective, extensive and robust institutional framework needs to be put in place. This framework is required at all three levels of governance: centre, state and local. The design, implementation of TOD and enforcement of urban transport pricing and regulatory measures require special attention. Proper co-ordination must be ensured between the several agencies involved at the different levels in order to prevent potential conflicts and delays.
The draft National Transit Oriented Development Policy paper takes into account the above internationally recognized principles and implementation guidelines for TOD. Keeping in view the international best practices and national debate and discussion on TOD as an instrument of sustainable and planned urban development, Box 2 provides some broad directions for executing TOD in cities and towns of India.
Implementing TOD in India.
TOD focuses on compact, mixed use development around transit corridors - metro rail, BRTS etc. International best practices have demonstrated that though transit system facilitates transit-oriented development, improving accessibility and creating walkable communities is equally important. Thus, to achieve the goals of TOD, the planning and development principles mentioned earlier in the study need to be adopted. The principles should also be supported by TOD-support policy tools such as right size infrastructure, technology integration, station area planning, land value capture, safety and security, universal accessibility etc. The following key aspects need to be considered for translating TOD principles and policies into practice in India:
1. Approach for TOD Implementation:
1.1 Influence Zone:
Influence zone of any transit corridor or station is the area in its immediate surrounding. It is intended to be developed into a compact, high density structure with mixed land use to cater to the residents’ basic needs. It is generally up to a radius of nearly 500–1000 mt of the transit station.
1.2 High Density Compact Development:
TOD calls for the densification of the influence zone. This can be done by providing higher Floor Area Ratio (FAR)/ Floor Space Index (FSI) and higher job and population density in the influence areas.To ensure sustainable and financially viable development, the minimum FAR should be 300–500 percent, and can be higher, depending on the city size.
1.3 Mixed Use Development:
Mixed land use in the TOD zone reduces the need to travel for work, shopping, leisure, education etc. The basic necessities of the residents can be provided within walking distance.
1.4 Mandatory and Inclusive Housing:
The cities should have minimum percentage (30 percent or higher) of allowed FAR for affordable housing in all development/ redevelopment in the influence zones. Housing in the influence zones should have a mix of all economic groups/ sections. The development control regulation should cater housing for EWS as well as LIG and MIG to give an opportunity to the people who depend on public transport for daily commuting to live in walkable neighborhoods.
1.5 Multimodal Integration:
An integrated multimodal network is required for availing various facilities in the influence zone. Seamless physical connectivity, integrated information system and fare integration can provide easy first and last mile connectivity.
1.6 Focus on pedestrians, cyclists and NMT users:
The influence zone should address the needs of pedestrians and NMT users. Sidewalks and amenities like benches, lighting, shops and information signage etc. should be developed.
1.7 Street Oriented Buildings and Vibrant Public Spaces:
Buildings should face the streets so as to define them better. Buildings should be oriented towards facing the pedestrian facilities. Public spaces should be developed to improvesocial interaction and strengthen community bonds and participation.
1.8 Managed Parking:
Use of private vehicles can be discouraged by reducing availability of parking spaces in influence zones and making it expensive. On-street parking should be prohibited within 100 mt of the transit station, except for freight delivery and pick-up or drop-off of the differently abled.
2. Value Capture Financing (VCF) for TOD:
The investment in the transit system as well as increase in FAR and provision for mixed use development would result in increase in value of land within the influence zone. Land Value Capture can be used as a mechanism to finance the required upgradation of infrastructure and amenities within the influence zone and expansion of the public transport system.
3. Statutory Framework:
TOD policy should be notified as part of the Master Plan/ Development Plan of the city whose vision should be resonated by all the stakeholders, especially those involved in infrastructure development and preparation of development plans. The policy document should clearly outline the importance of the high capacity transit network in the city’s development.
4. Coordination and Implementation:
Successful implementation of TOD requires the various agencies involved in planning, design and financing to work in coordination with each other. UMTAs need to be operationalized and strengthened.
5. Communication and Outreach:
It is important to create awareness about TOD so as to increase its use. Multiple agencies including both private and public stakeholders must have a collective approach for successful implementation of TOD.
Table 13 presents a summary of steps to convert the concepts of TOD to micro level implementation and undertake rapid transit station area and transit corridor development in India:
Existing Land Use | |
---|---|
a. Existing Land Use | • Development of existing corridor and station-area. • Making existing station-area pedestrian friendly, including access for persons with disabilities. • Improving parking supply in existing corridor and station-areas. |
Transit Supportive Plans and Policies | |
a. Growth Management | • Concentration of development around established activity centres and regional transit. • Greater employment opportunities should be provided close to transit stations. • Managing and conserving land. |
b. Transit Supportive Corridor Policies | • Development of station-area and increasing transit corridor. • Plans and policies aiming to increase transit-friendliness of transit stations, corridors and areas. • Designs to improve pedestrian amenities, including facilities for the differently abled commuters. • Parking policies. |
c. Supportive Zoning Regulations near Transit Stations | • Policies and regulations which encourage development around transit stations. • Zoning ordinances that provide enhanced accessibility topedestrians and encourage transit-oriented characteristics of stations. • Zoning allowances to mitigate traffic and reduce parking. • Ensuring provision of affordable housing units close to transit stations. |
d. Tools to implement land use policies | • Outreach to government agencies and the community in support of land use planning. • Regulatory and financial incentives to promote transit supportive development. • Higher FSI should be allowed along transit corridors. • Efforts to engage the development community in station area planning and transit-supportive development. |
Summary of steps to execute TOD: station area and corridor development.
India is going to experience a multifold rise in the demand for urban transport in the coming years. A strategic approach is required to ensure that the growth momentum is maintained without adversely impacting the quality of environment to urban dwellers. A holistic planning mechanism consolidating urban transport and land use planning is essential for Indian cities, especially metropolitan cities, so that synergies between urban form and functions can be channelized. This will further augment the productivity and efficiency of cities. The present challenges of congestion, pollution, accidents, sprawl etc. can be mitigated by investing in public modes of transport and optimizing multimodal mobility patterns. Urban transport influences the spatial organization of cities. So, urban transport must be approached in a holistic manner integrating pricing, financing regulation and comprehensive land use.
This chapter suggests that TOD is a necessity in India in view of the urbanization, metropolitanization and motorization trends in cities, the numbers and densities therein, income distribution patterns and considerations of sustainability. The country cannot afford auto-centric, sprawling, energy-intensive and an expensive process of urbanization. India needs to move to a public-transportation led, transit-oriented, mixed use, and value capture financing-focused strategy of planned urban development with public transportation investment and transport-land use integration as the key drivers. This also calls for a robust financing strategy. Successful TOD policy requires a robust and integrated framework bestowed with financial independence, responsiveness and competence. TOD policy can be successful by ensuring transparency and accountability towards the users while augmenting the accessibility of Indian cities.
We express our sincere thanks to the Housing and Urban Development Corporation (HUDCO), New Delhi for funding support to complete this research under the HUDCO Chair program.
We would like to thank Dr. Prasanna Kumar Mohanty, Chair Professor, Land, Housing, Transport and Urban Economics, University of Hyderabad, Member, Central Board of Directors, Reserve Bank of India and National Housing Bank, India, Ex. Chief Secretary, Undivided Andhra Pradesh, India for his valuable and constructive comments on this paper.
The authors declare no conflict of interest.
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Integrity - We are consistent and dependable, always striving for precision and accuracy in the true spirit of science.
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Then, key parameters of magnetic NPs representing their structural and magnetic properties are summarized with link to the relevant methods of characterization. Peculiar features of magnetism in real systems of the NPs at atomic, single‐particle, and mesoscopic level, respectively, are also discussed. Finally, the significant part of the chapter is devoted to the discussion of the structural and magnetic properties of the NPs in the context of the relevant preparation routes. 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Considering the energy consumption in real applications, lightweight EMI shielding materials has been attentive in this field of research. In this chapter, first of all, the EM theory will be briefly discussed. Secondly, the EMI shielding performance and corresponding mechanisms of three categories of lightweight materials, such as polymer-based composites, foams and aerogels, are reviewed. Finally, the summary and conclusions of this field will be addressed.",book:{id:"8653",slug:"electromagnetic-materials-and-devices",title:"Electromagnetic Materials and Devices",fullTitle:"Electromagnetic Materials and Devices"},signatures:"Liying Zhang, Shuguang Bi and Ming Liu",authors:[{id:"268866",title:"Prof.",name:"Liying",middleName:null,surname:"Zhang",slug:"liying-zhang",fullName:"Liying Zhang"},{id:"268868",title:"Prof.",name:"Shuguang",middleName:null,surname:"Bi",slug:"shuguang-bi",fullName:"Shuguang Bi"},{id:"268869",title:"Dr.",name:"Ming",middleName:null,surname:"Liu",slug:"ming-liu",fullName:"Ming Liu"}]},{id:"53689",title:"Optimizing Processing Conditions to Produce Cobalt Ferrite Nanoparticles of Desired Size and Magnetic Properties",slug:"optimizing-processing-conditions-to-produce-cobalt-ferrite-nanoparticles-of-desired-size-and-magneti",totalDownloads:1986,totalCrossrefCites:3,totalDimensionsCites:8,abstract:"The excellent chemical stability, good mechanical hardness, and a large positive first order magnetocrystalline anisotropy constant of cobalt ferrite (CoFe2O4) make it a promising candidate for magneto‐optical recording media. For practical applications, the capability to control particle size at the nanoscale is required in addition to precise control of the composition and structure of CoFe2O4. It has been well established that a fine tuning in cobalt ferrite nanocrystal size within the magnetic single domain region would lead to the achievement of extremely high coercivity values at room temperature. The development of a size‐sensitive phase separation method for cobalt ferrite that is based on a selective dissolution of the superparamagnetic fraction and subsequent size‐sensitive magnetic separation of single‐domain nanoparticles is presented. The attained room temperature coercivity value (11.9 kOe) was mainly attributed to the enlargement of the average crystal size within the single domain region coupled with the removal of the superparamagnetic fraction. The strong influence of crystal size, ferrite composition, and cation distribution in the ferrite lattice on the corresponding magnetic properties at the nanoscale was also confirmed. 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In this chapter, we review the various circumstances under which this unusual phenomenon occurs. We explain how partial MDM was first observed in rough Co/Pt multilayers with perpendicular magnetization as a result of structural defects. We then show how 100 % MDM was achieved, even in smooth ferromagnetic films, by coupling Co/Pd multilayers to an antiferromagnetic IrMn template via exchange interactions. We describe how high MDM, extending throughout nearly the entirety of the magnetization process, is obtained when zero-field-cooling the material below its blocking temperature where exchange couplings occur. We also review the persistence of MDM through field cycling and while warming the material all the way up to the blocking temperature. Additionally, we discuss the spatial dependence of MDM, highlighting intriguing oscillatory behaviors suggesting magnetic correlations and rotational symmetries at the nanoscopic scales. Finally, we review the dependence of MDM on cooling conditions, revealing how MDM can be fully controlled, turned on and off, by adjusting the magnitude of the cooling field.",book:{id:"6198",slug:"magnetism-and-magnetic-materials",title:"Magnetism and Magnetic Materials",fullTitle:"Magnetism and Magnetic Materials"},signatures:"Karine Chesnel",authors:[{id:"208623",title:"Prof.",name:"Karine",middleName:null,surname:"Chesnel",slug:"karine-chesnel",fullName:"Karine Chesnel"}]}],onlineFirstChaptersFilter:{topicId:"940",limit:6,offset:0},onlineFirstChaptersCollection:[],onlineFirstChaptersTotal:0},preDownload:{success:null,errors:{}},subscriptionForm:{success:null,errors:{}},aboutIntechopen:{},privacyPolicy:{},peerReviewing:{},howOpenAccessPublishingWithIntechopenWorks:{},sponsorshipBooks:{sponsorshipBooks:[],offset:8,limit:8,total:0},allSeries:{pteSeriesList:[{id:"14",title:"Artificial Intelligence",numberOfPublishedBooks:9,numberOfPublishedChapters:89,numberOfOpenTopics:6,numberOfUpcomingTopics:0,issn:"2633-1403",doi:"10.5772/intechopen.79920",isOpenForSubmission:!0},{id:"7",title:"Biomedical Engineering",numberOfPublishedBooks:12,numberOfPublishedChapters:104,numberOfOpenTopics:3,numberOfUpcomingTopics:0,issn:"2631-5343",doi:"10.5772/intechopen.71985",isOpenForSubmission:!0}],lsSeriesList:[{id:"11",title:"Biochemistry",numberOfPublishedBooks:32,numberOfPublishedChapters:318,numberOfOpenTopics:4,numberOfUpcomingTopics:0,issn:"2632-0983",doi:"10.5772/intechopen.72877",isOpenForSubmission:!0},{id:"25",title:"Environmental Sciences",numberOfPublishedBooks:1,numberOfPublishedChapters:12,numberOfOpenTopics:4,numberOfUpcomingTopics:0,issn:"2754-6713",doi:"10.5772/intechopen.100362",isOpenForSubmission:!0},{id:"10",title:"Physiology",numberOfPublishedBooks:11,numberOfPublishedChapters:141,numberOfOpenTopics:4,numberOfUpcomingTopics:0,issn:"2631-8261",doi:"10.5772/intechopen.72796",isOpenForSubmission:!0}],hsSeriesList:[{id:"3",title:"Dentistry",numberOfPublishedBooks:8,numberOfPublishedChapters:129,numberOfOpenTopics:2,numberOfUpcomingTopics:0,issn:"2631-6218",doi:"10.5772/intechopen.71199",isOpenForSubmission:!0},{id:"6",title:"Infectious Diseases",numberOfPublishedBooks:13,numberOfPublishedChapters:113,numberOfOpenTopics:3,numberOfUpcomingTopics:1,issn:"2631-6188",doi:"10.5772/intechopen.71852",isOpenForSubmission:!0},{id:"13",title:"Veterinary Medicine and Science",numberOfPublishedBooks:11,numberOfPublishedChapters:106,numberOfOpenTopics:3,numberOfUpcomingTopics:0,issn:"2632-0517",doi:"10.5772/intechopen.73681",isOpenForSubmission:!0}],sshSeriesList:[{id:"22",title:"Business, Management and Economics",numberOfPublishedBooks:1,numberOfPublishedChapters:19,numberOfOpenTopics:3,numberOfUpcomingTopics:0,issn:"2753-894X",doi:"10.5772/intechopen.100359",isOpenForSubmission:!0},{id:"23",title:"Education and Human Development",numberOfPublishedBooks:0,numberOfPublishedChapters:5,numberOfOpenTopics:1,numberOfUpcomingTopics:1,issn:null,doi:"10.5772/intechopen.100360",isOpenForSubmission:!0},{id:"24",title:"Sustainable Development",numberOfPublishedBooks:0,numberOfPublishedChapters:15,numberOfOpenTopics:5,numberOfUpcomingTopics:0,issn:null,doi:"10.5772/intechopen.100361",isOpenForSubmission:!0}],testimonialsList:[{id:"6",text:"It is great to work with the IntechOpen to produce a worthwhile collection of research that also becomes a great educational resource and guide for future research endeavors.",author:{id:"259298",name:"Edward",surname:"Narayan",institutionString:null,profilePictureURL:"https://mts.intechopen.com/storage/users/259298/images/system/259298.jpeg",slug:"edward-narayan",institution:{id:"3",name:"University of Queensland",country:{id:null,name:"Australia"}}}},{id:"13",text:"The collaboration with and support of the technical staff of IntechOpen is fantastic. 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