Gonzalo Emiliano Aranda Abreu

Universidad VeracruzanaMexico

Professor Dr. Gonzalo Emiliano Aranda Abreu (Centro de Investigaciones Cerebrales, Universidad Veracruzana, Xalapa, Veracruz, Mexico) was born in 1968 in Ciudad del Carmen, Campeche, Mexico. He received his BSc (Experimental Biology) degree from the Universidad Autónoma Metropolitana, Iztapalapa, Mexico, in 1992; MSc (Molecular Biology and Genetics) degree from the Centro de Investigación y Estudios Avanzados, IPN, Mexico, in 1996; and PhD (Neurobiology) degree from Weizmann Institute of Science, Rehovot, Israel, in 2001. He has more than 20 years of experience in Alzheimer’s disease research. He has written articles on tau mRNA transport in the neuronal axon and Alzheimer’s disease, as well as book chapters where the main theme is brain rehabilitation. He is the president and member of the Southeastern Mexican Chapter of the Society for Neuroscience, USA. He is involved in the training of doctoral students. He teaches molecular and cellular neurobiology, and bioinformatics to PhD degree students. Dr. Gonzalo Aranda\'s research is focused on Alzheimer\'s disease. He has extensive experience in mechanisms for locating mRNAs in the axon. He is also interested in the relationship that exists between neuroinflammation mechanisms and neurodegenerative diseases such as Alzheimer\'s disease. He is trying to find a therapy that can help Alzheimer\'s patients have a better quality of life.

2books edited

2chapters authored

Latest work with IntechOpen by Gonzalo Emiliano Aranda Abreu

"Mechanisms of Neuroinflammation" book explains how the neuronal cells become swollen at the moment of the blood-brain barrier disruption and how they lose their immunological isolation. A cascade of cytokines and immune cells from the bloodstream enters the nervous system, inflaming neurons and activating the glia. This produces a neuroinflammatory process that can generate different neurodegenerative diseases. Better understanding of mechanisms that are activated at the time when the damage to the brain occurs could lead to the development of suitable therapies that revert the neuronal inflammation and thus prevent further damage to the nervous system.

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