Kaneez Fatima Shad

University of Technology Sydney Australia

Professor Kaneez Fatima Shad, an Australian neuroscientist with a medical background, received her Ph.D. in 1994 from the Faculty of Medicine, UNSW, Australia, followed by postdoctoral research at the Allegheny University of Health Sciences, Philadelphia, USA. She has taught medical and biological sciences at various universities in Australia, the USA, UAE, Bahrain, Pakistan, and Brunei. During this period, she has been engaged in research, obtaining local and international grants totaling over 3.5 million US$ and translating them into products such as rapid diagnostic tests for stroke and other vascular disorders such as schizophrenia. She has published over seventy articles in refereed journals, edited nine books, written ten book chapters, presented at over one hundred two international conferences, and mentored thirty-four postgraduate students. She is a mentor and a protocol development specialist. For further information, see the home page www.fatimashad.com.

Kaneez Fatima Shad

9books edited

10chapters authored

Latest work with IntechOpen by Kaneez Fatima Shad

According to the World Health Organization, nearly 80% of people with epilepsy live in low- and middle-income countries and could live seizure-free if properly diagnosed and treated. Among the structural and genetic triggers that may lead to epilepsy are head trauma, abnormal synaptic connectivity, receptor subunits anomalies, and atypical ionic channel function. Developing brains are specifically susceptible to seizures for many reasons, for example, the presence of enhanced excitation due to the early production of excitatory neurotransmitters, and the fact that the inhibitory neurotransmitter GABA causes excitation early in life. These studies help to clarify why the very young brain is exceptionally prone to seizures. The chapters presented in this book describe how structural, genetic, infectious, and metabolic abnormalities can lead to epileptogenesis. Nevertheless, many underlying disease mechanisms that can lead to epilepsy remain unknown.

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