Comparison of BDS, IPC, and SCC.
\\n\\n
Released this past November, the list is based on data collected from the Web of Science and highlights some of the world’s most influential scientific minds by naming the researchers whose publications over the previous decade have included a high number of Highly Cited Papers placing them among the top 1% most-cited.
\\n\\nWe wish to congratulate all of the researchers named and especially our authors on this amazing accomplishment! We are happy and proud to share in their success!
Note: Edited in March 2021
\\n"}]',published:!0,mainMedia:{caption:"Highly Cited",originalUrl:"/media/original/117"}},components:[{type:"htmlEditorComponent",content:'IntechOpen is proud to announce that 191 of our authors have made the Clarivate™ Highly Cited Researchers List for 2020, ranking them among the top 1% most-cited.
\n\nThroughout the years, the list has named a total of 261 IntechOpen authors as Highly Cited. Of those researchers, 69 have been featured on the list multiple times.
\n\n\n\nReleased this past November, the list is based on data collected from the Web of Science and highlights some of the world’s most influential scientific minds by naming the researchers whose publications over the previous decade have included a high number of Highly Cited Papers placing them among the top 1% most-cited.
\n\nWe wish to congratulate all of the researchers named and especially our authors on this amazing accomplishment! We are happy and proud to share in their success!
Note: Edited in March 2021
\n'}],latestNews:[{slug:"intechopen-supports-asapbio-s-new-initiative-publish-your-reviews-20220729",title:"IntechOpen Supports ASAPbio’s New Initiative Publish Your Reviews"},{slug:"webinar-introduction-to-open-science-wednesday-18-may-1-pm-cest-20220518",title:"Webinar: Introduction to Open Science | Wednesday 18 May, 1 PM CEST"},{slug:"step-in-the-right-direction-intechopen-launches-a-portfolio-of-open-science-journals-20220414",title:"Step in the Right Direction: IntechOpen Launches a Portfolio of Open Science Journals"},{slug:"let-s-meet-at-london-book-fair-5-7-april-2022-olympia-london-20220321",title:"Let’s meet at London Book Fair, 5-7 April 2022, Olympia London"},{slug:"50-books-published-as-part-of-intechopen-and-knowledge-unlatched-ku-collaboration-20220316",title:"50 Books published as part of IntechOpen and Knowledge Unlatched (KU) Collaboration"},{slug:"intechopen-joins-the-united-nations-sustainable-development-goals-publishers-compact-20221702",title:"IntechOpen joins the United Nations Sustainable Development Goals Publishers Compact"},{slug:"intechopen-signs-exclusive-representation-agreement-with-lsr-libros-servicios-y-representaciones-s-a-de-c-v-20211123",title:"IntechOpen Signs Exclusive Representation Agreement with LSR Libros Servicios y Representaciones S.A. de C.V"},{slug:"intechopen-expands-partnership-with-research4life-20211110",title:"IntechOpen Expands Partnership with Research4Life"}]},book:{item:{type:"book",id:"5806",leadTitle:null,fullTitle:"Senescence - Physiology or Pathology",title:"Senescence",subtitle:"Physiology or Pathology",reviewType:"peer-reviewed",abstract:"In the second half of the twentieth century, life expectancy was prolonged, and the number of elderly people increased. The effect of population aging increases in the frequency of neurodegenerative diseases such as Alzheimer's and Parkinson's diseases, epilepsy, and stroke. Also, a higher incidence of infections, autoimmune diseases, and malignant cancers is observed in elderly people. The aging process is difficult to define. Are physiological changes in elderly people controlled by specific genes? Is aging process a pathophysiology affecting different organs with different severity? Finding answers to these questions may help prevent age-related diseases and improve the quality of life of old people. This book was made as a compendium on contemporary challenges in senescence.",isbn:"978-953-51-3462-6",printIsbn:"978-953-51-3461-9",pdfIsbn:"978-953-51-4678-0",doi:"10.5772/65533",price:119,priceEur:129,priceUsd:155,slug:"senescence-physiology-or-pathology",numberOfPages:166,isOpenForSubmission:!1,isInWos:null,isInBkci:!1,hash:"a8b68766b3057a8d6b4d30695e00f576",bookSignature:"Jolanta Dorszewska and Wojciech Kozubski",publishedDate:"August 30th 2017",coverURL:"https://cdn.intechopen.com/books/images_new/5806.jpg",numberOfDownloads:11126,numberOfWosCitations:18,numberOfCrossrefCitations:19,numberOfCrossrefCitationsByBook:0,numberOfDimensionsCitations:33,numberOfDimensionsCitationsByBook:0,hasAltmetrics:1,numberOfTotalCitations:70,isAvailableForWebshopOrdering:!0,dateEndFirstStepPublish:"November 14th 2016",dateEndSecondStepPublish:"December 5th 2016",dateEndThirdStepPublish:"March 3rd 2017",dateEndFourthStepPublish:"June 1st 2017",dateEndFifthStepPublish:"July 31st 2017",currentStepOfPublishingProcess:5,indexedIn:"1,2,3,4,5,6",editedByType:"Edited by",kuFlag:!1,featuredMarkup:null,editors:[{id:"31962",title:"Dr.",name:"Jolanta",middleName:null,surname:"Dorszewska",slug:"jolanta-dorszewska",fullName:"Jolanta Dorszewska",profilePictureURL:"https://mts.intechopen.com/storage/users/31962/images/4731_n.jpg",biography:"Editor, Professor Jolanta Dorszewska is Chief of Laboratory of Neurobiology, Department of Neurology, Poznan University of Medical Sciences (PUMS), Poznan, Poland. Prof. Dorszewska graduated from PUMS, (M.Sc., Pharmacy, 1987), Ph.D. degree obtained at PUMS, (1996), D.Sc. in Medical Sciences at PUMS, (2004) and Full Prof., (2016). Between the years 1999 and 2000 she worked as a Research Scientist at the Institute for Basic Research in Developmental Disabilities, New York, USA.\r\nProf. Dorszewska is an author and co-author of about 100 papers (e.g. Oncotarget, Curr. Alzheimer Res., Seizure) mainly concerning the pathophysiology of Parkinson’s and Alzheimer’s diseases as well as epilepsy and migraine. She is also a co-author and co-editor of books on genetic and biochemical factors in neurological diseases. \r\nProf. Dorszewska was also a Guest Editor of two Theme Issue in Current Genomics (2014, 2013), and a member of Editorial Board in Advances in Alzheimer’s Disease and Austin Alzheimer’s and Parkinson’s Disease (USA). Prof. Dorszewska is a member of the Commission of Neurochemistry of Neurological Sciences, Polish Academy of Science and Polish Association of Neuropathologists, as well as International Association of Neuropathologists.",institutionString:null,position:null,outsideEditionCount:0,totalCites:0,totalAuthoredChapters:"10",totalChapterViews:"0",totalEditedBooks:"3",institution:{name:"Poznan University of Medical Sciences",institutionURL:null,country:{name:"Poland"}}}],equalEditorOne:null,equalEditorTwo:null,equalEditorThree:null,coeditorOne:{id:"83372",title:"Prof.",name:"Wojciech",middleName:null,surname:"Kozubski",slug:"wojciech-kozubski",fullName:"Wojciech Kozubski",profilePictureURL:"https://mts.intechopen.com/storage/users/83372/images/system/83372.jpg",biography:"Prof. Wojciech Kozubski, MD, PhD is the Head of the Department of Neurology, University of Medical Sciences in Poznan, Poland.\nHe graduated from Medical School in Lodz in 1980. In 1983 he received his PhD and in 2002, his professorship.\nFrom 1987 to 1991, he was awarded a scholarship from the Academic Unit of Neuroscience, University of London, Department of Neurology, University of Tel-Aviv and the Department of Neurology, University of Trondheim.\nHe is an author and co-author of over 300 papers concerning the migraine and related headaches, stroke, and dementia. He is the editor of the handbook of clinical neurology for neurologists, the handbook for medical students, monographs on brain tumours, affective diseases of nervous system and therapy in neurology. \nFrom 2011 to 2014, he was the President of the Polish Neurological Society.",institutionString:"Poznań University of Medical Sciences",position:null,outsideEditionCount:0,totalCites:0,totalAuthoredChapters:"8",totalChapterViews:"0",totalEditedBooks:"1",institution:{name:"Poznan University of Medical Sciences",institutionURL:null,country:{name:"Poland"}}},coeditorTwo:null,coeditorThree:null,coeditorFour:null,coeditorFive:null,topics:[{id:"419",title:"Microbial Genetics",slug:"biochemistry-genetics-and-molecular-biology-microbiology-microbial-genetics"}],chapters:[{id:"56406",title:"Introductory Chapter: Molecular Basis of Senescence",doi:"10.5772/intechopen.70214",slug:"introductory-chapter-molecular-basis-of-senescence",totalDownloads:1098,totalCrossrefCites:0,totalDimensionsCites:0,hasAltmetrics:0,abstract:null,signatures:"Jolanta Dorszewska and Wojciech Kozubski",downloadPdfUrl:"/chapter/pdf-download/56406",previewPdfUrl:"/chapter/pdf-preview/56406",authors:[{id:"31962",title:"Dr.",name:"Jolanta",surname:"Dorszewska",slug:"jolanta-dorszewska",fullName:"Jolanta Dorszewska"}],corrections:null},{id:"55982",title:"Genetic Factors Associated with Longevity in Humans",doi:"10.5772/intechopen.69637",slug:"genetic-factors-associated-with-longevity-in-humans",totalDownloads:1387,totalCrossrefCites:1,totalDimensionsCites:1,hasAltmetrics:0,abstract:"Introduction: Life expectancy and the rate of survival into old age have risen dramatically throughout the past century. The positive ageing outcomes may be due to a variety of factors including healthy lifestyle behaviors, but it is clear that longevity has a genetic basis, with heritability estimate of 20–35%. In this contest, it was emerged that human longevity seems strongly influenced by gender defined as the combination between biological sexual characteristics and factors related to behavior, social role, lifestyle and life experiences. Body—research methods: Successful ageing seems to be related to gene involved in different pathways of regulation, such as immune-inflammatory responses and oxidative stress. The aims of the present review are to discuss recent findings and highlight the genetic basis of longevity. For these reasons we are aimed to describe the most important underpinning which is the gender differences in longevity between males and females. Conclusion—key results: It appears clear that longevity may represent a complex polygenic trait that is influenced by the interaction of multiple genetic variants, as was demonstrated by several genetic studies conducted in the last years. Furthermore, epigenetic and environmental factors actin on the longevity phenotype.",signatures:"Sara Bozzini and Colomba Falcone",downloadPdfUrl:"/chapter/pdf-download/55982",previewPdfUrl:"/chapter/pdf-preview/55982",authors:[{id:"201831",title:"Prof.",name:"Colomba",surname:"Falcone",slug:"colomba-falcone",fullName:"Colomba Falcone"},{id:"201834",title:"Dr.",name:"Sara",surname:"Bozzini",slug:"sara-bozzini",fullName:"Sara Bozzini"}],corrections:null},{id:"55008",title:"Sunflower Leaf Senescence: A Complex Genetic Process with Economic Impact on Crop Production",doi:"10.5772/intechopen.68588",slug:"sunflower-leaf-senescence-a-complex-genetic-process-with-economic-impact-on-crop-production",totalDownloads:1415,totalCrossrefCites:1,totalDimensionsCites:1,hasAltmetrics:1,abstract:"Leaf senescence is a complex process controlled by multiple genetic and environmental variables. In different crops, a delay in leaf senescence has an important impact on grain yield trough the maintenance of the photosynthetic leaf area during the reproductive stage. In sunflower (Helianthus annuus L.), the fourth largest oil crop worldwide, senescence reduces the capacity of plants to maintain their green leaf area for longer periods, especially during the grain filling phase, leading to important economic losses.",signatures:"Sebastián Moschen, Agustín I. López Gialdi, Norma Paniego, Paula\nFernandez and Ruth Amelia Heinz",downloadPdfUrl:"/chapter/pdf-download/55008",previewPdfUrl:"/chapter/pdf-preview/55008",authors:[{id:"199155",title:"Dr.",name:"Ruth",surname:"Heinz",slug:"ruth-heinz",fullName:"Ruth Heinz"},{id:"199163",title:"Dr.",name:"Sebastian",surname:"Moschen",slug:"sebastian-moschen",fullName:"Sebastian Moschen"},{id:"199164",title:"Dr.",name:"Paula",surname:"Fernandez",slug:"paula-fernandez",fullName:"Paula Fernandez"},{id:"199165",title:"Dr.",name:"Norma",surname:"Paniego",slug:"norma-paniego",fullName:"Norma Paniego"},{id:"199191",title:"BSc.",name:"Agustín",surname:"López Gialdi",slug:"agustin-lopez-gialdi",fullName:"Agustín López Gialdi"}],corrections:null},{id:"55125",title:"Accelerated Senescence of Cancer Stem Cells: A Failure to Thrive or a Route to Survival?",doi:"10.5772/intechopen.68582",slug:"accelerated-senescence-of-cancer-stem-cells-a-failure-to-thrive-or-a-route-to-survival-",totalDownloads:1657,totalCrossrefCites:8,totalDimensionsCites:14,hasAltmetrics:0,abstract:"Accelerated senescence of cancer stem cells (CSCs) represents an adaptive response allowing withstand cell death. TP53, the pivotal tumor suppressor plays an important role in this process by inducing a prolonged dual state with senescence and self-renewal as potential outcomes. Molecularly, this is achieved by activating both OCT4A (POU5F1) and p21CIP1. OCT4A suppresses the excessive activity of p21 preventing the immediate precipitation of apoptosis or terminal senescence. It persists as long as sufficient cellular energy remains; generated through autophagy, itself sequestrating p16INK4A in the cytoplasm. As such, autophagic capacity is the bottleneck of these TP53-dependent senescence reversal processes, as well terminal senescence will follow if DNA damage is not ultimately repaired. In TP53 mutants the CSC-like state is boosted by stressed cells overcoming the tetraploidy barrier. These cells acquire additional DNA repair capacity through mitotic slippage and entrance to a sequence of ploidy cycles, allowing repair and sorting DNA damage, ultimately facilitating the genesis of mitotically competent daughter cells following final depolyploidisation. Again, autophagy is required to fuel this process. More detailed knowledge of these arcane processes anticipates the provision of anti-cancer drug targets, such as AURORA B kinase and Survivin, which ensure mitotic slippage and the continuity of ploidy cycles.",signatures:"Jekaterina Erenpreisa, Kristine Salmina and Mark Steven Cragg",downloadPdfUrl:"/chapter/pdf-download/55125",previewPdfUrl:"/chapter/pdf-preview/55125",authors:[{id:"28637",title:"Dr.",name:"Jekaterina",surname:"Erenpreisa",slug:"jekaterina-erenpreisa",fullName:"Jekaterina Erenpreisa"},{id:"198907",title:"Dr.",name:"Kristine",surname:"Salmina",slug:"kristine-salmina",fullName:"Kristine Salmina"},{id:"198910",title:"Prof.",name:"Mark Steven",surname:"Cragg",slug:"mark-steven-cragg",fullName:"Mark Steven Cragg"}],corrections:null},{id:"55869",title:"Aging and Neurological Diseases",doi:"10.5772/intechopen.69499",slug:"aging-and-neurological-diseases",totalDownloads:1936,totalCrossrefCites:8,totalDimensionsCites:15,hasAltmetrics:0,abstract:"Current knowledge indicates that the aging process starts with subclinical changes at the molecular level. These include the accumulation of mutations, telomere attrition, and epigenetic alterations leading to genomic instability. Such defects multiply exponentially over time, resembling a “snowball effect,” and eventually leading to morphological and functional deterioration of the brain, including progressive neuronal loss, reduced levels of neurotransmitters, excessive inflammation, and disrupted integrity of vessels, followed by infarction and microbleeds. Additionally, the decreasing efficiency of DNA repair mechanisms increases the susceptibility to reactive oxygen species and spontaneous mutagenesis, resulting in age-related neoplasia. Moreover, the malnutrition and malabsorption seen commonly in the elderly may cause deficiency of vitamin B12 and folic acid, both necessary for homocysteine metabolism, and lead to vascular damage. Altogether, these lead to brain damage in old age and greatly increase the risk of developing diseases of the central nervous system, such as stroke, epilepsy, Parkinson’s disease, Alzheimer’s disease, and other dementias.",signatures:"Marta Kowalska, Michal Owecki, Michal Prendecki, Katarzyna Wize,\nJoanna Nowakowska, Wojciech Kozubski, Margarita Lianeri and\nJolanta Dorszewska",downloadPdfUrl:"/chapter/pdf-download/55869",previewPdfUrl:"/chapter/pdf-preview/55869",authors:[{id:"31962",title:"Dr.",name:"Jolanta",surname:"Dorszewska",slug:"jolanta-dorszewska",fullName:"Jolanta Dorszewska"},{id:"186409",title:"MSc.",name:"Michal",surname:"Prendecki",slug:"michal-prendecki",fullName:"Michal Prendecki"},{id:"186528",title:"MSc.",name:"Marta",surname:"Kowalska",slug:"marta-kowalska",fullName:"Marta Kowalska"},{id:"190030",title:"Ms.",name:"Katarzyna",surname:"Wize",slug:"katarzyna-wize",fullName:"Katarzyna Wize"},{id:"206247",title:"Mrs.",name:"Joanna",surname:"Nowakowska",slug:"joanna-nowakowska",fullName:"Joanna Nowakowska"},{id:"206248",title:"Dr.",name:"Michal",surname:"Owecki",slug:"michal-owecki",fullName:"Michal Owecki"}],corrections:null},{id:"55105",title:"Presenilins Interactome in Alzheimer’s Disease and Pathological Ageing",doi:"10.5772/intechopen.68748",slug:"presenilins-interactome-in-alzheimer-s-disease-and-pathological-ageing",totalDownloads:1259,totalCrossrefCites:1,totalDimensionsCites:2,hasAltmetrics:0,abstract:"Alzheimer’s disease (AD) is the most common type of dementia characterized by massive neuronal loss. Pathological hallmarks of the disease are overproduction of β-amyloid (Aβ) and hyperphosphorylation of tau protein accumulated into senile plaques (SPs) and neurofibrillary tangles (NFTs), respectively. SPs with cortical tau pathology are also hallmark of pathological ageing (PA). Recently, an extensive overlap has been shown between Aβ levels and profiles in PA and AD brains, suggesting that PA could be a prodromal AD phase. Presenilins are major components of the γ-secretase complex involved in Aβ production. Furthermore, presenilins interact with players of numerous signalling pathways important in the PA and AD. Integration of various modern research approaches would reinforce the role of presenilins signalling network in brain pathology. These approaches include high-throughput (epi)genetic and transcriptomic analyses, large-scale microscopic imaging studies, immunoaffinity purification or mass spectrometry. Comprehensive integration of these methods is necessary to update the definition of the role of presenilins in AD and PA. Hereby, we summarize the available data on presenilins’ functions and interactions. We believe that the systematization of the existing knowledge will stimulate further research and will help reveal the molecular nooks and crannies in Alzheimer’s disease and in pathological ageing.",signatures:"Michalina Maria Wężyk and Cezary Żekanowski",downloadPdfUrl:"/chapter/pdf-download/55105",previewPdfUrl:"/chapter/pdf-preview/55105",authors:[{id:"205453",title:"Dr.",name:"Michalina Maria",surname:"Wężyk",slug:"michalina-maria-wezyk",fullName:"Michalina Maria Wężyk"},{id:"205454",title:"Prof.",name:"Cezary",surname:"Żekanowski",slug:"cezary-zekanowski",fullName:"Cezary Żekanowski"}],corrections:null},{id:"55126",title:"Is Senescence Important in Hepatic Diseases?",doi:"10.5772/intechopen.68587",slug:"is-senescence-important-in-hepatic-diseases-",totalDownloads:1073,totalCrossrefCites:0,totalDimensionsCites:0,hasAltmetrics:0,abstract:"Aging is a condition in which a person gradually loses the ability to maintain homeostasis, due to structural alteration or dysfunction. Aging changes biological processes in many organs and tissues. The loss of regenerative capacity is the most dramatic age-associated alteration in the liver. Cellular damage, if not repaired, leads to apoptosis or senescence. The presence of permanent cell cycle arrest, the acquisition of major morphological change, and expression of senescence-associated β-galactosidase (SA-β-gal) are the characteristics of cellular senescence (CS). Interestingly, CS plays a crucial role in aging of both individual organs and the entire organism; consequently, senescent cells accumulate in organs and decline in organ function. Senescent cells have considerable influence on their microenvironment and exert both beneficial and detrimental effects through secretory associated senescent phenotype (SASP) factors. CS has attracted considerable recent interest with recognition of pathways linking aging, malignancy, and insulin resistance and the current focus on therapeutic interventions to extend healthspan. There are major implications for hepatology in the field of fibrosis and cancer, where cellular senescence of hepatocytes, cholangiocytes, stellate cells, and immune cells has been implicated in chronic liver disease progression.",signatures:"Ruth Pacheco Rivera, Jaime Arellanes Robledo and Jesús Serrano\nLuna",downloadPdfUrl:"/chapter/pdf-download/55126",previewPdfUrl:"/chapter/pdf-preview/55126",authors:[{id:"146165",title:"Prof.",name:"Jose De Jesús",surname:"Serrano-Luna",slug:"jose-de-jesus-serrano-luna",fullName:"Jose De Jesús Serrano-Luna"},{id:"199060",title:"Dr.",name:"Ruth",surname:"Pacheco-Rivera",slug:"ruth-pacheco-rivera",fullName:"Ruth Pacheco-Rivera"},{id:"199064",title:"Dr.",name:"Jaime",surname:"Arellanes-Robledo",slug:"jaime-arellanes-robledo",fullName:"Jaime Arellanes-Robledo"}],corrections:null},{id:"55948",title:"Potential Reduction in Mortality Associated with the Shifts of Population Educational Structures in the Czech Republic",doi:"10.5772/intechopen.69635",slug:"potential-reduction-in-mortality-associated-with-the-shifts-of-population-educational-structures-in-",totalDownloads:1303,totalCrossrefCites:0,totalDimensionsCites:0,hasAltmetrics:0,abstract:"Educational inequalities in mortality are large in Central and Eastern Europe. Mortality levels are particularly high among low educated men as well as women in the Czech Republic. However, differences in male mortality by educational attainment exceed those of females. Two mortality patterns are apparent when dividing the Czech classification of education into four categories—basic, vocational, secondary, and university. Males with basic education experience much higher mortality when compared to their higher educated counterparts. An anomaly in the mortality gradient is observed among women when comparing basic and vocational education. Women with basic education show a rather lower mortality level compared to their vocational counterparts. Three scenarios show how the shifts toward a higher education could contribute to the change in mortality level using temporary life expectancies between ages 30 and 80 for males and females: (a) population structure by sex, age, and education remains the same as from the census 2011; (b) 60% of males having the basic education move into the next higher category (vocational) and 60% of women with basic and vocational education move into the secondary education; and (3) sex age education‐specific mortality rates will be shifted upwards by one level.",signatures:"Jitka Rychtaříková and Klára Hulíková Tesárková",downloadPdfUrl:"/chapter/pdf-download/55948",previewPdfUrl:"/chapter/pdf-preview/55948",authors:[{id:"202636",title:"Prof.",name:"Jitka",surname:"Rychtarikova",slug:"jitka-rychtarikova",fullName:"Jitka Rychtarikova"},{id:"205750",title:"Dr.",name:"Klara",surname:"Hulikova",slug:"klara-hulikova",fullName:"Klara Hulikova"}],corrections:null}],productType:{id:"1",title:"Edited Volume",chapterContentType:"chapter",authoredCaption:"Edited by"},subseries:null,tags:null},relatedBooks:[{type:"book",id:"5277",title:"Challenges in Parkinson's Disease",subtitle:null,isOpenForSubmission:!1,hash:"ec247c295eeed9e8c8ab48a63b0b94c1",slug:"challenges-in-parkinson-s-disease",bookSignature:"Jolanta Dorszewska and Wojciech Kozubski",coverURL:"https://cdn.intechopen.com/books/images_new/5277.jpg",editedByType:"Edited by",editors:[{id:"31962",title:"Dr.",name:"Jolanta",surname:"Dorszewska",slug:"jolanta-dorszewska",fullName:"Jolanta Dorszewska"}],equalEditorOne:null,equalEditorTwo:null,equalEditorThree:null,productType:{id:"1",chapterContentType:"chapter",authoredCaption:"Edited by"}},{type:"book",id:"6614",title:"Alzheimer's Disease",subtitle:"The 21st Century Challenge",isOpenForSubmission:!1,hash:"91df6c15517737c8fb91543f870d484d",slug:"alzheimer-s-disease-the-21st-century-challenge",bookSignature:"Jolanta Dorszewska and Wojciech Kozubski",coverURL:"https://cdn.intechopen.com/books/images_new/6614.jpg",editedByType:"Edited by",editors:[{id:"31962",title:"Dr.",name:"Jolanta",surname:"Dorszewska",slug:"jolanta-dorszewska",fullName:"Jolanta Dorszewska"}],equalEditorOne:null,equalEditorTwo:null,equalEditorThree:null,productType:{id:"1",chapterContentType:"chapter",authoredCaption:"Edited by"}},{type:"book",id:"1446",title:"Senescence",subtitle:null,isOpenForSubmission:!1,hash:"7aa2772cf0b5653b6c599dba90f4c709",slug:"senescence",bookSignature:"Tetsuji Nagata",coverURL:"https://cdn.intechopen.com/books/images_new/1446.jpg",editedByType:"Edited by",editors:[{id:"93967",title:"Dr.",name:"Tetsuji",surname:"Nagata",slug:"tetsuji-nagata",fullName:"Tetsuji Nagata"}],equalEditorOne:null,equalEditorTwo:null,equalEditorThree:null,productType:{id:"1",chapterContentType:"chapter",authoredCaption:"Edited by"}},{type:"book",id:"3509",title:"Gene Therapy",subtitle:"Tools and Potential Applications",isOpenForSubmission:!1,hash:"0fd8b4898c201b4a9f8e597cbcf4d968",slug:"gene-therapy-tools-and-potential-applications",bookSignature:"Francisco Martin Molina",coverURL:"https://cdn.intechopen.com/books/images_new/3509.jpg",editedByType:"Edited by",editors:[{id:"32294",title:"Dr.",name:"Francisco",surname:"Martín-Molina",slug:"francisco-martin-molina",fullName:"Francisco Martín-Molina"}],equalEditorOne:null,equalEditorTwo:null,equalEditorThree:null,productType:{id:"1",chapterContentType:"chapter",authoredCaption:"Edited by"}},{type:"book",id:"1406",title:"Antimicrobial Agents",subtitle:null,isOpenForSubmission:!1,hash:"716194563847e4c8e0f4a7c07ff858ed",slug:"antimicrobial-agents",bookSignature:"Varaprasad Bobbarala",coverURL:"https://cdn.intechopen.com/books/images_new/1406.jpg",editedByType:"Edited by",editors:[{id:"90574",title:"Dr.",name:"Varaprasad",surname:"Bobbarala",slug:"varaprasad-bobbarala",fullName:"Varaprasad Bobbarala"}],equalEditorOne:null,equalEditorTwo:null,equalEditorThree:null,productType:{id:"1",chapterContentType:"chapter",authoredCaption:"Edited by"}},{type:"book",id:"364",title:"Gene Duplication",subtitle:null,isOpenForSubmission:!1,hash:"79e1de88c46f703c92c157b80d886221",slug:"gene-duplication",bookSignature:"Felix Friedberg",coverURL:"https://cdn.intechopen.com/books/images_new/364.jpg",editedByType:"Edited by",editors:[{id:"62782",title:"Prof.",name:"Felix",surname:"Friedberg",slug:"felix-friedberg",fullName:"Felix Friedberg"}],equalEditorOne:null,equalEditorTwo:null,equalEditorThree:null,productType:{id:"1",chapterContentType:"chapter",authoredCaption:"Edited by"}},{type:"book",id:"5090",title:"RNA Interference",subtitle:null,isOpenForSubmission:!1,hash:"9edcfa43c752e926f9e51ecb610e34db",slug:"rna-interference",bookSignature:"Ibrokhim Y. 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For example, there are two types of AC-AC converters, which convert fixed AC voltage and frequency into variable voltage with variable frequency. Figure 1 shows structure of AC-AC converter topologies [1]:
DC link
Direct link
AC-AC power converter topologies.
DC link converter or AC-DC-AC converter has been implemented at industries since 1902 because of its special features. For example, voltage source inverter has following merits:
Easy voltage supply control is possible for VSI.
Low harmonics content exist.
The main demerits of AC-DC-AC converter or DC link converters are as follows: (1) they are not suitable for transients operations because voltage across the large capacitor or large inductor in the circuit cannot be instantly changed [2]; (2) bulky, more weight, and costly. These limitations are overcome by direct AC-AC converters such as cycloconverters and matrix converters [2, 3]. This chapter is about matrix converter [4, 5], and its application is especially for aerospace. The matrix converter is preferred for cycloconverter [6] because of no limitations with respect to obtaining output frequency. The reason is that cycloconverter is limited to offer output frequency of one-third of its input frequency.
\nM. Venturini and A. Alesina invented the matrix converter technology in 1981 [4], and this paper described the fundamentals of matrix converter such as PWM to generate nine pulses with maximum voltage transfer ratio of 0.5 [4, 5]. The main advantages of MC are good sinusoidal input/output waveforms and inherent regeneration capability. The same authors improved the PWM algorithm to get 0.866 voltage transfer ratio with good sinusoidal output waveforms in 1986 [5]. After that, a lot of papers discussed different kinds of modulation schemes for MC [4, 5, 7]. The MC has severe problem with commutating bidirectional switches (BDS); but in 1992, four-step commutation [8, 9] was introduced. In 2001, Yaskawa Electric in Japan made 5.5-kW and 11-kW matrix converters, and now it is developing higher rating of matrix converters such as 22 kW and 45 kW [10] and selling for lift applications. Because of potential advantages of the Matrix Converter, this has been considered for commercial, industrial [11] and aerospace applications [12].
\nThe MC is especially suitable for aerospace applications because of it capability to provide wide range of unrestricted output frequencies which is imposed by it switching frequency.
\nThe aim of More Electric Aircraft (MEA) is to support green technology by replacing other powers usage of aircraft with electrical power usage. The conventional aircraft requires mainly four powers such as electrical power, pneumatic power, hydraulic power, and mechanical power. The concept of MEA is to replace other powers with electrical power using green technologies. This chapter is focused on green technology for aerospace applications such as aircraft surface actuation control systems. The reason is that regenerative power from the MC drive causes stability problems at aircraft power supply. Overcoming this limitation of MC drive is vital. For example, the host drum drive motor (HDDM) regenerates power when the tanker aircraft (TA) refueling hose trails and winds at air. Figure 2 shows circuit of the tanker aircraft with regeneration control circuit (RCC), which is used to dissipate regenerative power of MC drive using proposed methods.
\nTanker aircraft with RCC.
The host drum drive system (HDDS), which is controlled by Refuelling Control Unit (RCU) and Aeronautical Radio Incorporated Commands (ARINC), controls refueling hose and has three units such as motor control unit (MCU), dump resistor pack (DRP), and two motors. The schematic circuit of HDDS of TA is depicted in Figure 3.
\nSchematic of host drum drive system of TA.
Regeneration occurs only whenever refueling hose winds and trails and this action is commanded by MCU with RCU. It means that the MCU supervises direction of motors based on input from RCU commands. Hence, HDDS must dissipate the regenerated power; otherwise, it can cause below mentioned problems:
The input supply to HDDS will be increased during regeneration.
There is possibility of deactivation of HDDS system because of instability in the input supply of HDDS.
The HDDS is using DC link converter, which is not favorable to transient operations of HDDS drive, and this system is bulky and more weight, which are not desirable characteristics for aircraft. The MC drive is proposed to address above problems. However, inherent regeneration capability of matrix converter limits itself being used for above application because bidirectional switches directly fed back to regenerated power to aircraft input power supply without requiring any additional power electronic components.
\nAccording to aerospace power quality specifications, this regeneration onto aircraft input power supply must be limited. For this reason, avoidance of regeneration is vital for aircraft surface actuation systems of aircraft. Hence, to avoid regeneration in the matrix converter drive, three novel methods are proposed:
Bidirectional switch (BDS) method
Input power clamp (IPC) method
Standard clamp circuit (SCC) method
To detect regeneration in MC drive, two novel techniques are proposed:
Power comparison (PC) technique
Input voltage reference (IVR) technique
Each and every method has its own regeneration control circuit (RCC). For example, RCC for BDS method consists of three bidirectional switches (BDS) in series with three resistors, and this setup is connected across small input filter of MC drive. The RCC for IPC method requires one conventional uncontrolled six-pulse rectifier and a unidirectional switch (UDS) in series with a resistor, and this setup is connected in between input power supply and small input filter of MC. SCC method does not require additional components, which means that no separate RCC is required to do the same action.
\nThe simulation results prove that the matrix converter is a suitable alternative to conventional HDDS converter topologies. The BDS method is experimentally adopted to verify the proposed concept by laboratory prototype matrix converter, which is built at Smiths Aerospace laboratory (later called GE Aviation laboratory) in University of Nottingham. This MEA project strongly supports the green environment by adopting abovementioned green technologies to obtain reduced aircraft emissions.
\nMatrix converter consists of nine bidirectional switches arranged in 3 × 3 as shown in Figure 4. This is called all silicon solution [13]. The input phases (A, B, C) can be connected to output phases (a, b, c) for any switching period of time using bidirectional switches. The switches are controlled in such a way that the average output voltage is a sinusoidal waveform of the desired frequency and amplitude.
\nThe general structure of the conventional MC.
The matrix converter consists of nine bidirectional switches with 29 (512) possible switching states. However, only 27 switching states can be used because two basic rules have to be followed [13].
No short circuit of two inputs
Never open circuit the outputs
Because of the above rules and also inductive loads nature of MC drive, each output line must always be connected to an input line. Under these basic rules, space vector modulation (SVM) for MC drive has 27 switching states.
\nThe space vector modulation (SVM) is defined as type of pulse width modulation (PWM) to generate gate drive signal to trigger the bidirectional switches (BDS) in MC [14]. SVM is also preferable to control and analyze machines with vector control (VC) or field oriented control of machines and allows visualization of the spatial and time relationships between the resultant current and flux vectors (or space phasors) in various reference frames.
\nDecoupling flux and torque is feature of VC to overcome sluggish torque response of Induction Motor (IM) to work like a separately excited DC machine. To achieve an independent control of the flux and torque, the direct axis (
Field orientation: Ψr is aligned with d-axis.
Both faster current control loop and speed control loop outputs [12] provide the reference voltages (Vsd, Vsq) and hence (Va, Vb, Vc) to SVM to get the stable operation of MC drive as shown in Figure 6.
\nClosed-loop indirect field-oriented vector control (IFOVC) scheme.
Two novel techniques [18] are used to identify regeneration when step is applied to reverse the MC drive. These are (1) power comparison technique (PC) and (2) input voltage reference (IVR) technique. These techniques are responsible for generating pulses for regeneration control circuit (RCC) or electrical braking circuit (EBC) whenever regeneration is detected in the MC drive. In PC, output power is used as reference; hence, it is called PC technique similarly in IVR technique, the voltage across the small input filter capacitor and output power both are used as reference; hence, it is called IVR technique. The IVR technique is similar to and derived from conventional dynamic braking technique.
\nTo calculate the absolute value of output power of MC drive to achieve power comparison (PC) technique, the torque producing current (i*sq) and measured rotor speed (ωre) are sensed. Figure 7 shows the gate drive signal, which is generated for RCC of input power clamp (IPC) method. Here power dissipation through a resistor in the regeneration control circuit (RCC) is directly proportional to the duty cycle of unidirectional switch (UDS), as in Eq. (1),
\nBlock diagram of the power comparison (PC) technique for IPC method.
where D = duty cycle of the unidirectional switch and Pdis = power dissipation through the resistor.
\nThe duty cycle calculation requires the maximum electrical braking power (Pmb) to be calculated, as shown in Eq. (2). The duty cycle of the switches is then less than or equal to unity under all operating conditions.
\nwhere Tme = electromagnetic torque and ωmre = speed of MC drive.
\nThe gate drive signals for RCC switches are generated by using field programmable gate array (FPGA) with digital signal processor (DSP). Here FPGA that receives input parameters (ωre, Te, i*sq) from sensors is fed into DSP, which does all mathematical calculations to generate gate drive signal as shown in Figure 7, and again fed back to FPGA that is sending gate drive signal to the gate drive of UDS. The duty of UDS/BDS is linearly varying with respect to output negative power. The MC drive is not capable to output whole of regenerated power because of it losses such as friction, windage, iron, switching and conduction losses. Because of above reason, the braking resistor dissipates less than the actually regenerated power. As written in Eqs. (3) and (4), the design of braking resistor (Rb) relies on maximum regenerative power during regeneration.
\nwhere the braking current (Ib) and input power (Pin,max) are directly proportional to the input voltage. The braking resistor design also depends on the braking time, thermal capacity of the resistor, and heat sink. And the current rating of UDS/BDS in RCC must be higher than the braking current.
\nThe voltage across small input filter capacitor is measured and compared to the MC supply voltage to generate gate drive signal for RCC of IPC method as shown in Figure 8.
\nBlock diagram of the input voltage reference (IVR) technique for IPC method.
The IVR technique can be used to detect the regeneration in the matrix converter for electrical braking methods. The duty cycle variation is directly proportional to the increase in the line to line voltage across the input filter capacitor of the matrix converter under regeneration with respect to the output power (Po), as shown in Eq. (5).
\nHere,
The RCC is turned on only if any voltage difference is detected between MC supply voltage and voltage across the small input filter capacitor for each sampling period. In addition, if the small input filter capacitor voltage is equal to the MC supply voltage, then the duty cycle is set to zero. Gate drive signal is generated using FPGA and DSP control platform similar to PC technique. The power dissipation through RCC happens only if the MC drive is operating under regenerative mode.
\nTo avoid regeneration in matrix converters, three novel circuit topologies are investigated:
Bidirectional switch (BDS) method
Input power clamp (IPC) method
Standard clamp circuit (SCC) method
The power circuit for the BDS method [18], regeneration control circuit (RCC) or electrical braking circuit, is shown in Figure 9. The regeneration control circuit (RCC) is introduced across the input filter capacitors (CAB, CBC, and CAC). The regeneration control circuit (RCC) is responsible for power dissipation when regeneration takes place in the MC motor drive.
\nBidirectional switch (BDS) method.
The RCC consists of three bidirectional switches (BDSAB, BDSBC, and BDSAC) in series with three resistors (RAB, RBC, and RAC) connected across the input lines, in parallel with the input filter capacitor. The schematic of the regeneration control circuit (RCC) or electrical braking circuit (EPC) is depicted in Figure 9.
\nThe input power clamp (IPC) method [19] is used for braking the electrical energy in a matrix converter motor drive. The IPC method requires only one braking resistor and a UDS, as shown in Figure 10, when compared to the BDS method, which requires three switches in series with three resistors. Electrical braking circuit or regeneration control circuit (RCC) for the input power clamp (IPC) method is located across the input filter capacitors (
The input power clamp (IPC) method.
The main power electronic components for RCC of IPC are conventional uncontrolled six-pulse rectifier and a UDS in series with a braking resistor (R), as shown in Figure 10. This braking resistor does not have inductive property to help to achieve better electrical braking when regeneration happens in MC drive. It is believed that IPC method is the best method when compared to BDS method because it requires only fewer power semiconductor switching components but not suitable for aerospace applications because it has electrolytic capacitor in the RCC.
\nSimilar to the BDS method and the IPC method [19], the proposed standard clamp circuit (SCC) method is using two techniques to detect the regeneration in the matrix converter drive. These are (1) power comparison (PC) technique and (2) input voltage reference technique. However, here the PC technique is only considered and the simulation results of the SCC method with PC technique are discussed. The block diagram for standard clamp circuit is shown in Figure 11. The electrical braking circuit for SCC is shown in Figure 14.
\nThe standard clamp circuit method.
Power dissipation through resistor is directly proportional to duty cycle of UDS, which is already given in Eq. (1). To prove the performance of the SCC method for electrical braking in the MC drive, a 2.2-kW vector-controlled induction motor fed by MC drive is considered.
\nWhen compared to earlier methods, called the BDS method and IPC method, no auxiliary hardware is required for SCC method for electrical braking in the matrix converter drive as shown in Table 1. The BDS method [18] has three drawbacks:
It requires three BDS in series with three resistors.
Also, it requires complex control platform, which controls six PWM for electrical braking.
This auxiliary circuit increases size, weight, and cost.
Factors | \nBDS method | \nIPC method | \nSCC method | \n
---|---|---|---|
Switches | \n3 (BDS) | \n1 (UDS) | \n1 (UDS) | \n
Resistors | \n3 | \n1 | \n1 | \n
Diodes | \n0 | \n6 | \n0 | \n
Weight, size, and cost | \nConsiderably increased | \nBit increased | \nRemains same | \n
Implementation | \nComplicated | \nNot complicated | \nSimple | \n
Comparison of BDS, IPC, and SCC.
Similarly, the IPC method has three main drawbacks:
It requires conventional uncontrolled diode rectifier and UDS with a resistor.
Separate control platform for a PWM for electrical braking is required.
This auxiliary circuit increases the size, weight, and cost.
The SCC method requires only one UDS switch in series with a resistor. Because of using SCC in the MC drive, achieving electrical braking using this method is easy and no complicated control platform is required. The SCC is considered as a safety device to protect the matrix converter under abnormal conditions such as overvoltage in the input side or output side.
\nTo predict and verify the performance of the proposed methods for avoiding regeneration in a matrix converter, a simulation study is carried out using SABER software package [12].
\nThe regeneration can be demonstrated at Vin = 240 V, q = 0.75, and fs = 10 kHz by applying step transient to reverse the speed at 1.2 s as shown in Figure 12. A step transient lasts upto 2.4s, no load speed reversal (from +188.5 rad/s to -188.5 rad/s), as shown in the Figure 13 which also shows developed torque which is direclty proportional to torque producing current (isq) of IM during VC. The torque producing current (iq) of the induction motor reaches the maximum limit of 35 A during acceleration as shown in Figure 13. Here regenerative power depends upon large inertial load (j = 0.089 kg m2) of the induction motor, which is created coupling IM with the same rating of DC motor (4 kW). The output current waveforms of the matrix converter drive is depicted in Figure 14(a), which also indicates during speed reversal, the four-quadrant operation, inherent property, of MC from motoring mode to regenerating mode is smoothly achieved. The control of dq-currents (id, iq) with no coupling effects is demonstrated. Figure 14(b) shows input phase currents (iA, iB, iC) of the matrix converter during the four-quadrant operation. The input regenerative powers (PA, PB, PC) to be dissipated using the regeneration control circuit (RCC) are shown in Figure 15(a). During regeneration, the phase opposition (180
Overview of the simulation diagram for obtaining regeneration in the MC vector-controlled induction motor.
Speed and torque of the vector-controlled IM in regeneration. Vin = 240 V, q = 0.75, and fs = 10 kHz.
(a) Output currents of the MC and (b) input phase currents of the MC.
(a) Input phase powers of the MC and (b) phase opposition at regeneration.
The generation of the required identical six pulses using PC technique for the regeneration control circuit (RCC) of BDS method is shown in Figure 16(a). Here duty cycle is linearly varying with respect to the output power of the MC drive as shown in Figure 16(b). In order to verify the regenerative energy dissipation, the input phase powers are calculated using input phase voltages and the input phase currents. The resulting input phase currents and calculated input phase power are shown in Figure 17(a) and (b), respectively. When compared to Figure 15(b), Figure 18 proves that regenerative power is dissipated using novel RCC of BDS method, hence input phase voltages (VA, VB, VC) and input phase currents (iA, iB, iC) are in phase. However, there is some input power left, as shown in Figure 17(b), because of constant losses (such as friction, windage losses, and inertial losses) in the IM and switching noises.
\n(a) Generation of the six pulses and (b) duty cycle and output power variation for regeneration control circuit of the BDS method with the PC technique. Vin = 240 V, q = 0.75, and fs = 10 kHz.
(a) Input phase currents and (b) input phase powers for BDS method.
Phase relationship between input phase voltages and currents of MC drive.
The generation of a pulse to trigger the RCC, duty cycle variation, and the output power (Po) variation during regeneration for the IPC method with the IVR technique is shown in Figure 19. Figure 20 shows input phase powers (PA, PB, PC) of MC drive after regeneration control. From simulation results, the IVR technique for both methods (IPC and SCC) is producing acceptable results to avoid regeneration with a MC drive similar to PC technique.
\nPulse for RCC, duty cycle variation, and output power for the IPC method with the IVR technique. Vin = 240 V, q = 0.75, and fs = 10 kHz.
Input phase powers for the SCC method with the IVR technique. Vin = 240 V, q = 0.75, and fs = 10 kHz.
The control platform includes both the control circuits and the interface circuits. The control circuit consists of a DSP card and an FPGA card as shown in Figure 21. The interface circuit includes encoder interface board and DSP daughter card (C6713DSK HPI), which is used to send user inputs and output waveforms plotting to troubleshoot hardware problems that are faced during hardware implementation and achieving desired output results. For example, if spike occurs while reversing the speed of the MC drive, the error data were captured and troubleshot through DSP daughter card. Figure 22(a) shows the RCC of three bidirectional switches (BDSAB, BDSBC, BDSAC) that are connected between MC input supply line voltages (VAB, VBC, VAC) and the small input filter capacitors (2).
\nLayout of control and interface circuits.
(a) Photograph of RCC for BDS method and (b) complete experimental setup.
The RCC resistors (RAB, RBC, RAC) are connected in series (1) with the bidirectional switches. The triggering pulses (3) for bidirectional switches are obtained from FPGA card. Figure 22(b) shows complete experimental setup of MC drive. The host user interface PC is used to help monitor inputs to the system (complete experimental setup of MC drive) and get output from the system. The power circuit and control circuit of the laboratory prototype matrix converter drive is highlighted by letters (C) and (B), respectively. Letters (D) and (E) indicate the 4 kW induction motor with high inertial load and the RCC resistors with their heat sink arrangement, respectively.
\nProof of BDS method for electrical braking in the MC drive by carried out experiments, at Smiths aerospace (later called GE Aviation) laboratory in PEMC Group of University of Nottingham, using a prototype rated at 7.5 kW MC fed a 4 kW IM. The field current (d-currents), torque current (q-currents), torque of IM and stator currents of IM during regeneration at speed reversal from +157 rad/s to −157 rad/s [Vin = 200 V, fs = 12.5 kHz, q = 0.75] are shown in Figure 23(a) and Figure 23 (b), respectively. The input phase voltages (VA, VB), input phase currents (iA, iB), and three phase input power (using two-wattmeter method) during regeneration and after avoiding regeneration are shown in Figure 24(a) and (b), respectively. The above experimental results (from Figure 24(a) and (b)) clearly show that the regenerative (negative) power is dissipated through the RCC.
\n(a) Torque, dq-currents and (b) stator currents of the motor.
(a) During regeneration and (b) after avoiding regeneration.
The matrix converter (MC) technology has been preferred since 1989 than other direct AC/AC converters or AC-DC-AC link converters because of its special features such as no DC link components, good sinusoidal input/output waveforms, inherent regeneration capability, and unrestricted output frequency. The published research work on the matrix converter focuses on the following ideas:
MC for aerospace and industrial applications
Power quality and stability of MC
Addressing commutation techniques to avoid failure of the power circuit of MC
Modulation topologies for the MC
This novel research work is dedicated to matrix converter for more electric aircraft (MEA) application. And making MC suitable for aerospace applications by avoiding inherent regeneration in it, it means eliminating unique property of four-quadrant operation of MC, in order to satisfy the aircraft power quality specifications. Until this work, no one has paid attention on this research area, which will make the matrix converter feasible for aerospace applications and some specific industrial applications. For example, at the beginning of the twenty-first century, the matrix converter has been made as a commercial product, lift, which is manufactured by Yaskawa (Japan). The Power Electronics Machines and Control (PEMC) Group at the University of Nottingham has been developing 150-kVA matrix converter for higher power applications. Even though all three methods (BDS, IPC, and SCC) can produce good results, the standard clamp circuit method with power comparison technique is preferable because no auxiliary hardware is required. Hence, the weight, size, and cost of the matrix converter are considerably reduced. Therefore, the matrix converter with SCC method is recommended to aerospace applications where regeneration into the supply is not allowed. From obtained experimental results, it is concluded that electrical braking with a matrix converter drive is feasible and matrix converter is opt for aerospace applications such as more electric aircraft.
\nThe authors would like to thank Smiths Aerospace/GE Aviation laboratory for their support to complete this research successfully.
\nOral ulcerative lesions are defects in the oral epithelia, its underlying connective tissue or both. The oral mucosa is considered among one of the susceptible areas in the human body to painful ulceration [1, 2]. An oral ulcer is not a disease itself but rather a sign of a different underlying condition, therefore it is usually challenging to diagnose the accurate etiology [1]. It is important to identify the etiologic factor to provide a complete resolution to patients rather than constantly prescribing certain medicines to suppress the symptoms [3].
Regardless of the etiology of these lesions, oral ulcerative lesions may be categorized as minor, major or herpetiform ulcerations. Minor ulcerations are usually less than 1 cm in diameter, and they most commonly present on the labial or buccal mucosa or the ventral surface of the tongue. Less common locations include the dorsum of the tongue, hard palate or the gingiva [4, 5].
Ulcerations that measure more than 1 cm in diameter are referred to as major oral ulcerations and have a lower prevalence than minor ones. Among these three types, the least common type is the herpetiform ulceration, which unlike what its name suggests, is irrelevant to herpetic stomatitis since no vesicle formation is observed in advance [5]. These type of ulcerations are multiple and usually are much smaller in diameter (1–3 mm) [4].
Another helpful classification is based on the duration of these lesions, which may aid clinicians establish a more logical stepwise progression towards an accurate diagnosis. Accordingly, an oral ulcerative lesion is diagnosed as acute if it lasts for less than two weeks, chronic if it persists for more than two weeks or recurrent if it presents with a history ulcerative episode with intermittent periods of healing [1].
A short-lived oral ulcerative lesion which resolves in less than two weeks is considered as an acute oral ulcer and is commonly referred to as an “aphtha” [1, 2]. This word itself is attributed to Hippocrates which was used to describe disorders of the mouth in general back in his time (460–370 BC) [3]. In order to accurately diagnose and evaluate patients with acute ulcerative lesions, it is crucial that the physician is aware of the broad spectrum of possibilities that may cause these lesions. It is recommended to assess the history of the lesions first, meaning to question the patient regarding any periodic episodes, in order to exclude conditions which are characterized with recurrent oral ulcerations [2].
Clinically acute oral ulcers usually have an oval shape with an erythematous periphery due to the dilation of the blood vessels. Although commonly these are painful lesions, the pain is relatively weaker when the ulcer bed is layered with a yellowish fibro-membrane [2].
Most common acute oral ulcerations may be related to trauma (i.e., traumatic ulcers), chemotherapy (i.e. chemotherapy induced ulcers), necrotizing sialometaplasia, primary herpetic gingivostomatitis, herpes zoster infection, herpangina, hand-foot mouth disease, erythema multiforme, necrotizing ulcerative gingivitis, oral hypersensitivity reactions or plasma cell stomatitis [1, 6, 7].
Irritation fibroma of the right vestibular sulcus and an acute traumatic ulceration of the maxillary frenulum due to ill-fitting dentures.
Oral hypersensitivity reaction, three days after switching to a new brand of toothpaste.
Severe epithelial sloughing and erythema on a patient with plasma cell stomatitis.
Chronic ulcerative lesions have a slower onset than acute ulcers and last for more than two weeks. Several vesiculobullous entities, lupus erythematosus, tuberculosis, some mycoses, eosinophilic ulcers and oral cancer are among the most common conditions associated with chronic oral ulcerative lesions [6].
Wickham’s striae: The characteristic white, lace-like keratotic configurations seen in lichen planus.
Erosive lichen planus with an ulcerative area covered with a yellowish white pseudomembrane.
Typical “crater like lesion with rolled borders”-look on a malignant ulcerative lesion of the hard palate.
Cause-based treatment options are more beneficial than palliative management options. Therefore it is crucial for physicians to make a sound clinical examination and take a through patient history [3]. Biopsy should be considered if an ulcerative lesion with an unknown etiology shows no signs of healing after two weeks, or if the lesions is not responsive to a treatment aimed at a probable known etiology. Ulcers of less than 5 mm diameter are advised to undergo an excisional biopsy, whereas for larger ones an incisional biopsy is suggested [6].
The author declares no conflict of interest.
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\\n"}]'},components:[{type:"htmlEditorComponent",content:'Copyright is the term used to describe the rights related to the publication and distribution of original Works. Most importantly from a publisher's perspective, copyright governs how Authors, publishers and the general public can use, publish, and distribute publications.
\n\nIntechOpen only publishes manuscripts for which it has publishing rights. This is governed by a publication agreement between the Author and IntechOpen. This agreement is accepted by the Author when the manuscript is submitted and deals with both the rights of the publisher and Author, as well as any obligations concerning a particular manuscript. However, in accepting this agreement, Authors continue to retain significant rights to use and share their publications.
\n\nHOW COPYRIGHT WORKS WITH OPEN ACCESS LICENSES?
\n\nAgreement samples are listed here for the convenience of prospective Authors:
\n\nDEFINITIONS
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\n\nAuthor - in order to be identified as an Author, three criteria must be met: (i) Substantial contribution to the conception or design of the Work, or the acquisition, analysis, or interpretation of data for the Work; (ii) Participation in drafting or revising the Work; (iii) Approval of the final version of the Work to be published.
\n\nWork - a Chapter, including Conference Papers, a Scientific Article and any and all text, graphics, images and/or other materials forming part of or accompanying the Chapter/Conference Paper.
\n\nMonograph/Compacts - a full manuscript usually written by a single Author, including any and all text, graphics, images and/or other materials.
\n\nCompilation - a collection of Works distributed in a Book that IntechOpen has selected, and for which the coordination of the preparation, arrangement and publication has been the responsibility of IntechOpen. Any Work included is accepted in its entirety in unmodified form and is published with one or more other contributions, each constituting a separate and independent Work, but which together are assembled into a collective whole.
\n\nScientific Journal – Periodical publication intended to further the progress of science.
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\n\nIntechOpen platform - IntechOpen website www.intechopen.com whose main purpose is to host Monographs in the format of Book Chapters, Long Form Monographs, Compacts, Conference Proceedings, Scientific Journals and Videos.
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\n\nTERMS
\n\nAll Works published on the IntechOpen platform and in print are licensed under a Creative Commons Attribution 3.0 Unported and Creative Commons 4.0 International License, a license which allows for the broadest possible reuse of published material.
\n\nCopyright on the individual Works belongs to the specific Author, subject to an agreement with IntechOpen. The Creative Common license is granted to all others to:
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\n\nAll Works are published under the CC BY 3.0 and CC BY 4.0 license. However, please note that book Chapters may fall under a different CC license, depending on their publication date as indicated in the table below:
\n\n\n\n
LICENSE | \n\t\t\tUSED FROM - | \n\t\t\tUP TO - | \n\t\t
\n\t\t\t Creative Commons Attribution-NonCommercial-ShareAlike 3.0 Unported (CC BY-NC-SA 3.0) \n\t\t\t | \n\t\t\t1 July 2005 (2005-07-01) | \n\t\t\t3 October 2011 (2011-10-03) | \n\t\t
\n\t\t\t Creative Commons Attribution 3.0 Unported (CC BY 3.0) \n\t\t\t | \n\t\t\t5 October 2011 (2011-10-05) | \n\t\t\tCurrently | \n\t\t
\n\t\t\t Creative Commons 4.0 International (CC BY 4.0) – for Journal Articles \n\t\t\t | \n\t\t\t15 March 2022 | \n\t\t\tCurrently | \n\t\t
The CC BY 3.0 and CC BY 4.0 license permits Works to be freely shared in any medium or format, as well as the reuse and adaptation of the original contents of Works (e.g. figures and tables created by the Authors), as long as the source Work is cited and its Authors are acknowledged in the following manner:
\n\nContent reuse:
\n\n© {year} {authors' full names}. Originally published in {short citation} under {license version} license. Available from: {DOI}
\n\nContent adaptation & reuse:
\n\n© {year} {authors' full names}. Adapted from {short citation}; originally published under {license version} license. Available from: {DOI}
\n\nReposting & sharing:
\n\nOriginally published in {full citation}. Available from: {DOI}
\n\nRepublishing – More about Attribution Policy can be found here.
\n\nThe same principles apply to Works published under the CC BY-NC-SA 3.0 license, with the caveats that (1) the content may not be used for commercial purposes, and (2) derivative works building on this content must be distributed under the same license. The restrictions contained in these license terms may, however, be waived by the copyright holder(s). Users wishing to circumvent any of the license terms are required to obtain explicit permission to do so from the copyright holder(s).
\n\nDISCLAIMER: Neither the CC BY 3.0 license, CC BY 4.0, nor any other license IntechOpen currently uses or has used before, applies to figures and tables reproduced from other works, as they may be subject to different terms of reuse. In such cases, if the copyright holder is not noted in the source of a figure or table, it is the responsibility of the User to investigate and determine the exact copyright status of any information utilised. Users requiring assistance in that regard are welcome to send an inquiry to permissions@intechopen.com.
\n\nAll rights to Books and Journals and all other compilations published on the IntechOpen platform and in print are reserved by IntechOpen.
\n\nThe copyright to Books, Journals and other compilations is subject to separate copyright from those that exist in the included Works.
\n\nAll Long Form Monographs/Compacts are licensed under the Creative Commons Attribution-NonCommercial 4.0 International (CC BY-NC 4.0) license granted to all others.
\n\nCopyright to the individual Works (Chapters) belongs to their specific Authors, subject to an agreement with IntechOpen and the Creative Common license granted to all others to:
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\n\nNonCommercial - The use of the material for commercial purposes is prohibited. Commercial rights are reserved to IntechOpen or its licensees.
\n\nNo additional restrictions that apply legal terms or technological measures that restrict others from doing anything the license permits are allowed.
\n\nThe CC BY-NC 4.0 license permits Works to be freely shared in any medium or format, as well as reuse and adaptation of the original contents of Works (e.g. figures and tables created by the Authors), as long as it is not used for commercial purposes. The source Work must be cited and its Authors acknowledged in the following manner:
\n\nContent reuse:
\n\n© {year} {authors' full names}. Originally published in {short citation} under {license version} license. Available from: {DOI}
\n\nContent adaptation & reuse:
\n\n© {year} {authors' full names}. Adapted from {short citation}; originally published under {license version} license. Available from: {DOI}
\n\nReposting & sharing:
\n\nOriginally published in {full citation}. Available from: {DOI}
\n\nAll Book cover design elements, as well as Video image graphics are subject to copyright by IntechOpen.
\n\nEvery reproduction of a front cover image must be accompanied by an appropriate Copyright Notice displayed adjacent to the image. The exact Copyright Notice depends on who the Author of a particular cover image is. Users wishing to reproduce cover images should contact permissions@intechopen.com.
\n\nAll Video Lectures under IntechOpen's production are subject to copyright and are property of IntechOpen, unless defined otherwise, and are licensed under the Attribution-NonCommercial-NoDerivatives 4.0 International (CC BY-NC-ND 4.0) license. This grants all others the right to:
\n\nShare — copy and redistribute the material in any medium or format
\n\nUnder the following terms:
\n\nUsers wishing to repost and share the Video Lectures are welcome to do so as long as they acknowledge the source in the following manner:
\n\n© {year} IntechOpen. Published under CC BY-NC-ND 4.0 license. Available from: {DOI}
\n\nUsers wishing to reuse, modify, or adapt the Video Lectures in a way not permitted by the license are welcome to contact us at permissions@intechopen.com to discuss waiving particular license terms.
\n\nAll software used on the IntechOpen platform, any used during the publishing process, and the copyright in the code constituting such software, is the property of IntechOpen or its software suppliers. As such, it may not be downloaded or copied without permission.
\n\nUnless otherwise indicated, all IntechOpen websites are the property of IntechOpen.
\n\nAll content included on IntechOpen Websites not forming part of contributed materials (such as text, images, logos, graphics, design elements, videos, sounds, pictures, trademarks, etc.), are subject to copyright and are property of, or licensed to, IntechOpen. Any other use, including the reproduction, modification, distribution, transmission, republication, display, or performance of the content on this site is strictly prohibited.
\n\nPolicy last updated: 2016-06-08
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His present research includes organic synthesis, drug discovery and development, biochemistry, nanoscience, and nanotechnology.",institutionString:"Visiting Scientist at Lipid Nanostructures Laboratory, Centre for Smart Materials, School of Natural Sciences, University of Central Lancashire",institution:null},{id:"428125",title:"Dr.",name:"Vinayak",middleName:null,surname:"Adimule",slug:"vinayak-adimule",fullName:"Vinayak Adimule",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/428125/images/system/428125.jpg",biography:"Dr. Vinayak Adimule, MSc, Ph.D., is a professor and dean of R&D, Angadi Institute of Technology and Management, India. He has 15 years of research experience as a senior research scientist and associate research scientist in R&D organizations. He has published more than fifty research articles as well as several book chapters. He has two Indian patents and two international patents to his credit. Dr. Adimule has attended, chaired, and presented papers at national and international conferences. He is a guest editor for Topics in Catalysis and other journals. He is also an editorial board member, life member, and associate member for many international societies and research institutions. His research interests include nanoelectronics, material chemistry, artificial intelligence, sensors and actuators, bio-nanomaterials, and medicinal chemistry.",institutionString:"Angadi Institute of Technology and Management",institution:null},{id:"284317",title:"Prof.",name:"Kantharaju",middleName:null,surname:"Kamanna",slug:"kantharaju-kamanna",fullName:"Kantharaju Kamanna",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/284317/images/21050_n.jpg",biography:"Prof. K. Kantharaju has received Bachelor of science (PCM), master of science (Organic Chemistry) and Doctor of Philosophy in Chemistry from Bangalore University. He worked as a Executive Research & Development @ Cadila Pharmaceuticals Ltd, Ahmedabad. He received DBT-postdoc fellow @ Molecular Biophysics Unit, Indian Institute of Science, Bangalore under the supervision of Prof. P. Balaram, later he moved to NIH-postdoc researcher at Drexel University College of Medicine, Philadelphia, USA, after his return from postdoc joined NITK-Surthakal as a Adhoc faculty at department of chemistry. Since from August 2013 working as a Associate Professor, and in 2016 promoted to Profeesor in the School of Basic Sciences: Department of Chemistry and having 20 years of teaching and research experiences.",institutionString:null,institution:{name:"Rani Channamma University, Belagavi",country:{name:"India"}}},{id:"158492",title:"Prof.",name:"Yusuf",middleName:null,surname:"Tutar",slug:"yusuf-tutar",fullName:"Yusuf Tutar",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/158492/images/system/158492.jpeg",biography:"Prof. Dr. Yusuf Tutar conducts his research at the Hamidiye Faculty of Pharmacy, Department of Basic Pharmaceutical Sciences, Division of Biochemistry, University of Health Sciences, Turkey. He is also a faculty member in the Molecular Oncology Program. He obtained his MSc and Ph.D. at Oregon State University and Texas Tech University, respectively. He pursued his postdoctoral studies at Rutgers University Medical School and the National Institutes of Health (NIH/NIDDK), USA. His research focuses on biochemistry, biophysics, genetics, molecular biology, and molecular medicine with specialization in the fields of drug design, protein structure-function, protein folding, prions, microRNA, pseudogenes, molecular cancer, epigenetics, metabolites, proteomics, genomics, protein expression, and characterization by spectroscopic and calorimetric methods.",institutionString:"University of Health Sciences",institution:null},{id:"180528",title:"Dr.",name:"Hiroyuki",middleName:null,surname:"Kagechika",slug:"hiroyuki-kagechika",fullName:"Hiroyuki Kagechika",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/180528/images/system/180528.jpg",biography:"Hiroyuki Kagechika received his bachelor’s degree and Ph.D. in Pharmaceutical Sciences from the University of Tokyo, Japan, where he served as an associate professor until 2004. He is currently a professor at the Institute of Biomaterials and Bioengineering (IBB), Tokyo Medical and Dental University (TMDU). From 2010 to 2012, he was the dean of the Graduate School of Biomedical Science. Since 2012, he has served as the vice dean of the Graduate School of Medical and Dental Sciences. He has been the director of the IBB since 2020. Dr. Kagechika’s major research interests are the medicinal chemistry of retinoids, vitamins D/K, and nuclear receptors. 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He received his post-doctoral training in oncology and cancer proteomics for two years at the Cancer Research Institute of Human Medical University in China. In 2001, he went to the University of Tennessee Health Science Center (UTHSC) in USA, where he was a post-doctoral researcher and focused on mass spectrometry and cancer proteomics. Then, he was appointed as an Assistant Professor of Neurology, UTHSC in 2005. He moved to the Cleveland Clinic in USA as a Project Scientist/Staff in 2006 where he focused on the studies of eye disease proteomics and biomarkers. He returned to UTHSC as an Assistant Professor of Neurology in the end of 2007, engaging in proteomics and biomarker studies of lung diseases and brain tumors, and initiating the studies of predictive, preventive, and personalized medicine (PPPM) in cancer. In 2010, he was promoted to Associate Professor of Neurology, UTHSC. Currently, he is a Professor at Xiangya Hospital of Central South University in China, Fellow of Royal Society of Medicine (FRSM), the European EPMA National Representative in China, Regular Member of American Association for the Advancement of Science (AAAS), European Cooperation of Science and Technology (e-COST) grant evaluator, Associate Editors of BMC Genomics, BMC Medical Genomics, EPMA Journal, and Frontiers in Endocrinology, Executive Editor-in-Chief of Med One. He has\npublished 116 peer-reviewed research articles, 16 book chapters, 2 books, and 2 US patents. 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He has published several articles in peer-reviewed journals, chapters, and edited books. His area of specialization is free radical biochemistry and autoimmune diseases.",institutionString:"Imam Abdulrahman Bin Faisal University",institution:{name:"Imam Abdulrahman Bin Faisal University",country:{name:"Saudi Arabia"}}},{id:"41865",title:"Prof.",name:"Farid A.",middleName:null,surname:"Badria",slug:"farid-a.-badria",fullName:"Farid A. Badria",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/41865/images/system/41865.jpg",biography:"Farid A. Badria, Ph.D., is the recipient of several awards, including The World Academy of Sciences (TWAS) Prize for Public Understanding of Science; the World Intellectual Property Organization (WIPO) Gold Medal for best invention; Outstanding Arab Scholar, Kuwait; and the Khwarizmi International Award, Iran. He has 250 publications, 12 books, 20 patents, and several marketed pharmaceutical products to his credit. 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He has more than sixteen years of teaching experience and has supervised numerous postgraduate and Ph.D. students. He has to his credit more than seventy papers in SCI- and SCOPUS-indexed journals, fifty-five conference proceedings, four books, six Best Paper Awards, and five projects from different government agencies. He is currently an editorial board member of eight international journals and a reviewer for more than fifty scientific journals. He received Top Reviewer and Excellent Peer Reviewer Awards from Publons in 2016 and 2017, respectively. He is also on the panel of The International Reviewer for reviewing research proposals for grants from the Royal Society. He also serves as a Publons Academy mentor and Bentham brand ambassador.",institutionString:"Punjab Technical University",institution:{name:"Punjab Technical University",country:{name:"India"}}},{id:"142388",title:"Dr.",name:"Thiago",middleName:"Gomes",surname:"Gomes Heck",slug:"thiago-gomes-heck",fullName:"Thiago Gomes Heck",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/142388/images/7259_n.jpg",biography:null,institutionString:null,institution:{name:"Universidade Regional do Noroeste do Estado do Rio Grande do Sul",country:{name:"Brazil"}}},{id:"336273",title:"Assistant Prof.",name:"Janja",middleName:null,surname:"Zupan",slug:"janja-zupan",fullName:"Janja Zupan",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/336273/images/14853_n.jpeg",biography:"Janja Zupan graduated in 2005 at the Department of Clinical Biochemistry (superviser prof. dr. Janja Marc) in the field of genetics of osteoporosis. Since November 2009 she is working as a Teaching Assistant at the Faculty of Pharmacy, Department of Clinical Biochemistry. In 2011 she completed part of her research and PhD work at Institute of Genetics and Molecular Medicine, University of Edinburgh. She finished her PhD entitled The influence of the proinflammatory cytokines on the RANK/RANKL/OPG in bone tissue of osteoporotic and osteoarthritic patients in 2012. From 2014-2016 she worked at the Institute of Biomedical Sciences, University of Aberdeen as a postdoctoral research fellow on UK Arthritis research project where she gained knowledge in mesenchymal stem cells and regenerative medicine. She returned back to University of Ljubljana, Faculty of Pharmacy in 2016. She is currently leading project entitled Mesenchymal stem cells-the keepers of tissue endogenous regenerative capacity facing up to aging of the musculoskeletal system funded by Slovenian Research Agency.",institutionString:null,institution:{name:"University of Ljubljana",country:{name:"Slovenia"}}},{id:"357453",title:"Dr.",name:"Radheshyam",middleName:null,surname:"Maurya",slug:"radheshyam-maurya",fullName:"Radheshyam Maurya",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/357453/images/16535_n.jpg",biography:null,institutionString:null,institution:{name:"University of Hyderabad",country:{name:"India"}}},{id:"418340",title:"Dr.",name:"Jyotirmoi",middleName:null,surname:"Aich",slug:"jyotirmoi-aich",fullName:"Jyotirmoi Aich",position:null,profilePictureURL:"https://s3.us-east-1.amazonaws.com/intech-files/0033Y000038Ugi5QAC/Profile_Picture_2022-04-15T07:48:28.png",biography:"Biotechnologist with 15 years of research including 6 years of teaching experience. Demonstrated record of scientific achievements through consistent publication record (H index = 13, with 874 citations) in high impact journals such as Nature Communications, Oncotarget, Annals of Oncology, PNAS, and AJRCCM, etc. Strong research professional with a post-doctorate from ACTREC where I gained experimental oncology experience in clinical settings and a doctorate from IGIB where I gained expertise in asthma pathophysiology. A well-trained biotechnologist with diverse experience on the bench across different research themes ranging from asthma to cancer and other infectious diseases. An individual with a strong commitment and innovative mindset. Have the ability to work on diverse projects such as regenerative and molecular medicine with an overall mindset of improving healthcare.",institutionString:"DY Patil Deemed to Be University",institution:null},{id:"349288",title:"Prof.",name:"Soumya",middleName:null,surname:"Basu",slug:"soumya-basu",fullName:"Soumya Basu",position:null,profilePictureURL:"https://s3.us-east-1.amazonaws.com/intech-files/0033Y000035QxIDQA0/Profile_Picture_2022-04-15T07:47:01.jpg",biography:"Soumya Basu, Ph.D., is currently working as an Associate Professor at Dr. D. Y. Patil Biotechnology and Bioinformatics Institute, Dr. D. Y. Patil Vidyapeeth, Pune, Maharashtra, India. With 16+ years of trans-disciplinary research experience in Drug Design, development, and pre-clinical validation; 20+ research article publications in journals of repute, 9+ years of teaching experience, trained with cross-disciplinary education, Dr. Basu is a life-long learner and always thrives for new challenges.\r\nHer research area is the design and synthesis of small molecule partial agonists of PPAR-γ in lung cancer. She is also using artificial intelligence and deep learning methods to understand the exosomal miRNA’s role in cancer metastasis. Dr. Basu is the recipient of many awards including the Early Career Research Award from the Department of Science and Technology, Govt. of India. She is a reviewer of many journals like Molecular Biology Reports, Frontiers in Oncology, RSC Advances, PLOS ONE, Journal of Biomolecular Structure & Dynamics, Journal of Molecular Graphics and Modelling, etc. She has edited and authored/co-authored 21 journal papers, 3 book chapters, and 15 abstracts. She is a Board of Studies member at her university. She is a life member of 'The Cytometry Society”-in India and 'All India Cell Biology Society”- in India.",institutionString:"Dr. D.Y. Patil Vidyapeeth, Pune",institution:{name:"Dr. D.Y. Patil Vidyapeeth, Pune",country:{name:"India"}}},{id:"354817",title:"Dr.",name:"Anubhab",middleName:null,surname:"Mukherjee",slug:"anubhab-mukherjee",fullName:"Anubhab Mukherjee",position:null,profilePictureURL:"https://intech-files.s3.amazonaws.com/0033Y0000365PbRQAU/ProfilePicture%202022-04-15%2005%3A11%3A18.480",biography:"A former member of Laboratory of Nanomedicine, Brigham and Women’s Hospital, Harvard University, Boston, USA, Dr. Anubhab Mukherjee is an ardent votary of science who strives to make an impact in the lives of those afflicted with cancer and other chronic/acute ailments. He completed his Ph.D. from CSIR-Indian Institute of Chemical Technology, Hyderabad, India, having been skilled with RNAi, liposomal drug delivery, preclinical cell and animal studies. He pursued post-doctoral research at College of Pharmacy, Health Science Center, Texas A & M University and was involved in another postdoctoral research at Department of Translational Neurosciences and Neurotherapeutics, John Wayne Cancer Institute, Santa Monica, California. In 2015, he worked in Harvard-MIT Health Sciences & Technology as a visiting scientist. He has substantial experience in nanotechnology-based formulation development and successfully served various Indian organizations to develop pharmaceuticals and nutraceutical products. He is an inventor in many US patents and an author in many peer-reviewed articles, book chapters and books published in various media of international repute. Dr. Mukherjee is currently serving as Principal Scientist, R&D at Esperer Onco Nutrition (EON) Pvt. Ltd. and heads the Hyderabad R&D center of the organization.",institutionString:"Esperer Onco Nutrition Pvt Ltd.",institution:null},{id:"319365",title:"Assistant Prof.",name:"Manash K.",middleName:null,surname:"Paul",slug:"manash-k.-paul",fullName:"Manash K. Paul",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/319365/images/system/319365.png",biography:"Manash K. Paul is a Principal Investigator and Scientist at the University of California Los Angeles. He has contributed significantly to the fields of stem cell biology, regenerative medicine, and lung cancer. His research focuses on various signaling processes involved in maintaining stem cell homeostasis during the injury-repair process, deciphering lung stem cell niche, pulmonary disease modeling, immuno-oncology, and drug discovery. He is currently investigating the role of extracellular vesicles in premalignant lung cell migration and detecting the metastatic phenotype of lung cancer via machine-learning-based analyses of exosomal signatures. Dr. Paul has published in more than fifty peer-reviewed international journals and is highly cited. He is the recipient of many awards, including the UCLA Vice Chancellor’s award, a senior member of the Institute of Electrical and Electronics Engineers (IEEE), and an editorial board member for several international journals.",institutionString:"University of California Los Angeles",institution:{name:"University of California Los Angeles",country:{name:"United States of America"}}},{id:"311457",title:"Dr.",name:"Júlia",middleName:null,surname:"Scherer Santos",slug:"julia-scherer-santos",fullName:"Júlia Scherer Santos",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/311457/images/system/311457.jpg",biography:"Dr. Júlia Scherer Santos works in the areas of cosmetology, nanotechnology, pharmaceutical technology, beauty, and aesthetics. Dr. Santos also has experience as a professor of graduate courses. Graduated in Pharmacy, specialization in Cosmetology and Cosmeceuticals applied to aesthetics, specialization in Aesthetic and Cosmetic Health, and a doctorate in Pharmaceutical Nanotechnology. Teaching experience in Pharmacy and Aesthetics and Cosmetics courses. She works mainly on the following subjects: nanotechnology, cosmetology, pharmaceutical technology, aesthetics.",institutionString:"Universidade Federal de Juiz de Fora",institution:{name:"Universidade Federal de Juiz de Fora",country:{name:"Brazil"}}},{id:"219081",title:"Dr.",name:"Abdulsamed",middleName:null,surname:"Kükürt",slug:"abdulsamed-kukurt",fullName:"Abdulsamed Kükürt",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/219081/images/system/219081.png",biography:"Dr. Kükürt graduated from Uludağ University in Turkey. He started his academic career as a Research Assistant in the Department of Biochemistry at Kafkas University. In 2019, he completed his Ph.D. program in the Department of Biochemistry at the Institute of Health Sciences. He is currently working at the Department of Biochemistry, Kafkas University. He has 27 published research articles in academic journals, 11 book chapters, and 37 papers. He took part in 10 academic projects. He served as a reviewer for many articles. He still serves as a member of the review board in many academic journals. He is currently working on the protective activity of phenolic compounds in disorders associated with oxidative stress and inflammation.",institutionString:null,institution:{name:"Kafkas University",country:{name:"Turkey"}}},{id:"178366",title:"Dr.",name:"Volkan",middleName:null,surname:"Gelen",slug:"volkan-gelen",fullName:"Volkan Gelen",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/178366/images/system/178366.jpg",biography:"Volkan Gelen is a Physiology specialist who received his veterinary degree from Kafkas University in 2011. Between 2011-2015, he worked as an assistant at Atatürk University, Faculty of Veterinary Medicine, Department of Physiology. In 2016, he joined Kafkas University, Faculty of Veterinary Medicine, Department of Physiology as an assistant professor. Dr. Gelen has been engaged in various academic activities at Kafkas University since 2016. There he completed 5 projects and has 3 ongoing projects. He has 60 articles published in scientific journals and 20 poster presentations in scientific congresses. His research interests include physiology, endocrine system, cancer, diabetes, cardiovascular system diseases, and isolated organ bath system studies.",institutionString:"Kafkas University",institution:{name:"Kafkas University",country:{name:"Turkey"}}},{id:"418963",title:"Dr.",name:"Augustine Ododo",middleName:"Augustine",surname:"Osagie",slug:"augustine-ododo-osagie",fullName:"Augustine Ododo Osagie",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/418963/images/16900_n.jpg",biography:"Born into the family of Osagie, a prince of the Benin Kingdom. I am currently an academic in the Department of Medical Biochemistry, University of Benin. Part of the duties are to teach undergraduate students and conduct academic research.",institutionString:null,institution:{name:"University of Benin",country:{name:"Nigeria"}}},{id:"192992",title:"Prof.",name:"Shagufta",middleName:null,surname:"Perveen",slug:"shagufta-perveen",fullName:"Shagufta Perveen",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/192992/images/system/192992.png",biography:"Prof. Shagufta Perveen is a Distinguish Professor in the Department of Pharmacognosy, College of Pharmacy, King Saud University, Riyadh, Saudi Arabia. Dr. Perveen has acted as the principal investigator of major research projects funded by the research unit of King Saud University. She has more than ninety original research papers in peer-reviewed journals of international repute to her credit. She is a fellow member of the Royal Society of Chemistry UK and the American Chemical Society of the United States.",institutionString:"King Saud University",institution:{name:"King Saud University",country:{name:"Saudi Arabia"}}},{id:"49848",title:"Dr.",name:"Wen-Long",middleName:null,surname:"Hu",slug:"wen-long-hu",fullName:"Wen-Long Hu",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/49848/images/system/49848.jpg",biography:"Wen-Long Hu is Chief of the Division of Acupuncture, Department of Chinese Medicine at Kaohsiung Chang Gung Memorial Hospital, as well as an adjunct associate professor at Fooyin University and Kaohsiung Medical University. Wen-Long is President of Taiwan Traditional Chinese Medicine Medical Association. He has 28 years of experience in clinical practice in laser acupuncture therapy and 34 years in acupuncture. He is an invited speaker for lectures and workshops in laser acupuncture at many symposiums held by medical associations. He owns the patent for herbal preparation and producing, and for the supercritical fluid-treated needle. Dr. Hu has published three books, 12 book chapters, and more than 30 papers in reputed journals, besides serving as an editorial board member of repute.",institutionString:"Kaohsiung Chang Gung Memorial Hospital",institution:{name:"Kaohsiung Chang Gung Memorial Hospital",country:{name:"Taiwan"}}},{id:"298472",title:"Prof.",name:"Andrey V.",middleName:null,surname:"Grechko",slug:"andrey-v.-grechko",fullName:"Andrey V. Grechko",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/298472/images/system/298472.png",biography:"Andrey Vyacheslavovich Grechko, Ph.D., Professor, is a Corresponding Member of the Russian Academy of Sciences. He graduated from the Semashko Moscow Medical Institute (Semashko National Research Institute of Public Health) with a degree in Medicine (1998), the Clinical Department of Dermatovenerology (2000), and received a second higher education in Psychology (2009). Professor A.V. Grechko held the position of Сhief Physician of the Central Clinical Hospital in Moscow. He worked as a professor at the faculty and was engaged in scientific research at the Medical University. Starting in 2013, he has been the initiator of the creation of the Federal Scientific and Clinical Center for Intensive Care and Rehabilitology, Moscow, Russian Federation, where he also serves as Director since 2015. He has many years of experience in research and teaching in various fields of medicine, is an author/co-author of more than 200 scientific publications, 13 patents, 15 medical books/chapters, including Chapter in Book «Metabolomics», IntechOpen, 2020 «Metabolomic Discovery of Microbiota Dysfunction as the Cause of Pathology».",institutionString:"Federal Research and Clinical Center of Intensive Care Medicine and Rehabilitology",institution:null},{id:"199461",title:"Prof.",name:"Natalia V.",middleName:null,surname:"Beloborodova",slug:"natalia-v.-beloborodova",fullName:"Natalia V. Beloborodova",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/199461/images/system/199461.jpg",biography:'Natalia Vladimirovna Beloborodova was educated at the Pirogov Russian National Research Medical University, with a degree in pediatrics in 1980, a Ph.D. in 1987, and a specialization in Clinical Microbiology from First Moscow State Medical University in 2004. She has been a Professor since 1996. Currently, she is the Head of the Laboratory of Metabolism, a division of the Federal Research and Clinical Center of Intensive Care Medicine and Rehabilitology, Moscow, Russian Federation. N.V. Beloborodova has many years of clinical experience in the field of intensive care and surgery. She studies infectious complications and sepsis. She initiated a series of interdisciplinary clinical and experimental studies based on the concept of integrating human metabolism and its microbiota. Her scientific achievements are widely known: she is the recipient of the Marie E. Coates Award \\"Best lecturer-scientist\\" Gustafsson Fund, Karolinska Institutes, Stockholm, Sweden, and the International Sepsis Forum Award, Pasteur Institute, Paris, France (2014), etc. Professor N.V. Beloborodova wrote 210 papers, five books, 10 chapters and has edited four books.',institutionString:"Federal Research and Clinical Center of Intensive Care Medicine and Rehabilitology",institution:null},{id:"354260",title:"Ph.D.",name:"Tércio Elyan",middleName:"Azevedo",surname:"Azevedo Martins",slug:"tercio-elyan-azevedo-martins",fullName:"Tércio Elyan Azevedo Martins",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/354260/images/16241_n.jpg",biography:"Graduated in Pharmacy from the Federal University of Ceará with the modality in Industrial Pharmacy, Specialist in Production and Control of Medicines from the University of São Paulo (USP), Master in Pharmaceuticals and Medicines from the University of São Paulo (USP) and Doctor of Science in the program of Pharmaceuticals and Medicines by the University of São Paulo. Professor at Universidade Paulista (UNIP) in the areas of chemistry, cosmetology and trichology. Assistant Coordinator of the Higher Course in Aesthetic and Cosmetic Technology at Universidade Paulista Campus Chácara Santo Antônio. Experience in the Pharmacy area, with emphasis on Pharmacotechnics, Pharmaceutical Technology, Research and Development of Cosmetics, acting mainly on topics such as cosmetology, antioxidant activity, aesthetics, photoprotection, cyclodextrin and thermal analysis.",institutionString:null,institution:{name:"University of Sao Paulo",country:{name:"Brazil"}}},{id:"334285",title:"Ph.D. Student",name:"Sameer",middleName:"Kumar",surname:"Jagirdar",slug:"sameer-jagirdar",fullName:"Sameer Jagirdar",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/334285/images/14691_n.jpg",biography:"I\\'m a graduate student at the center for biosystems science and engineering at the Indian Institute of Science, Bangalore, India. I am interested in studying host-pathogen interactions at the biomaterial interface.",institutionString:null,institution:{name:"Indian Institute of Science Bangalore",country:{name:"India"}}},{id:"329248",title:"Dr.",name:"Md. Faheem",middleName:null,surname:"Haider",slug:"md.-faheem-haider",fullName:"Md. Faheem Haider",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/329248/images/system/329248.jpg",biography:"Dr. Md. Faheem Haider completed his BPharm in 2012 at Integral University, Lucknow, India. In 2014, he completed his MPharm with specialization in Pharmaceutics at Babasaheb Bhimrao Ambedkar University, Lucknow, India. He received his Ph.D. degree from Jamia Hamdard University, New Delhi, India, in 2018. He was selected for the GPAT six times and his best All India Rank was 34. Currently, he is an assistant professor at Integral University. Previously he was an assistant professor at IIMT University, Meerut, India. He has experience teaching DPharm, Pharm.D, BPharm, and MPharm students. He has more than five publications in reputed journals to his credit. Dr. Faheem’s research area is the development and characterization of nanoformulation for the delivery of drugs to various organs.",institutionString:"Integral University",institution:{name:"Integral University",country:{name:"India"}}},{id:"329795",title:"Dr.",name:"Mohd Aftab",middleName:"Aftab",surname:"Siddiqui",slug:"mohd-aftab-siddiqui",fullName:"Mohd Aftab Siddiqui",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/329795/images/system/329795.png",biography:"Dr. Mohd Aftab Siddiqui is an assistant professor in the Faculty of Pharmacy, Integral University, Lucknow, India, where he obtained a Ph.D. in Pharmacology in 2020. He also obtained a BPharm and MPharm from the same university in 2013 and 2015, respectively. His area of research is the pharmacological screening of herbal drugs/natural products in liver cancer and cardiac diseases. He is a member of many professional bodies and has guided many MPharm and PharmD research projects. Dr. Siddiqui has many national and international publications and one German patent to his credit.",institutionString:"Integral University",institution:null}]}},subseries:{item:{id:"22",type:"subseries",title:"Applied Intelligence",keywords:"Machine Learning, Intelligence Algorithms, Data Science, Artificial Intelligence, Applications on Applied Intelligence",scope:"This field is the key in the current industrial revolution (Industry 4.0), where the new models and developments are based on the knowledge generation on applied intelligence. The motor of the society is the industry and the research of this topic has to be empowered in order to increase and improve the quality of our lives.",coverUrl:"https://cdn.intechopen.com/series_topics/covers/22.jpg",hasOnlineFirst:!0,hasPublishedBooks:!0,annualVolume:11418,editor:{id:"27170",title:"Prof.",name:"Carlos",middleName:"M.",surname:"Travieso-Gonzalez",slug:"carlos-travieso-gonzalez",fullName:"Carlos Travieso-Gonzalez",profilePictureURL:"https://mts.intechopen.com/storage/users/27170/images/system/27170.jpeg",biography:"Carlos M. Travieso-González received his MSc degree in Telecommunication Engineering at Polytechnic University of Catalonia (UPC), Spain in 1997, and his Ph.D. degree in 2002 at the University of Las Palmas de Gran Canaria (ULPGC-Spain). He is a full professor of signal processing and pattern recognition and is head of the Signals and Communications Department at ULPGC, teaching from 2001 on subjects on signal processing and learning theory. His research lines are biometrics, biomedical signals and images, data mining, classification system, signal and image processing, machine learning, and environmental intelligence. He has researched in 52 international and Spanish research projects, some of them as head researcher. He is co-author of 4 books, co-editor of 27 proceedings books, guest editor for 8 JCR-ISI international journals, and up to 24 book chapters. He has over 450 papers published in international journals and conferences (81 of them indexed on JCR – ISI - Web of Science). He has published seven patents in the Spanish Patent and Trademark Office. He has been a supervisor on 8 Ph.D. theses (11 more are under supervision), and 130 master theses. He is the founder of The IEEE IWOBI conference series and the president of its Steering Committee, as well as the founder of both the InnoEducaTIC and APPIS conference series. He is an evaluator of project proposals for the European Union (H2020), Medical Research Council (MRC, UK), Spanish Government (ANECA, Spain), Research National Agency (ANR, France), DAAD (Germany), Argentinian Government, and the Colombian Institutions. He has been a reviewer in different indexed international journals (<70) and conferences (<250) since 2001. He has been a member of the IASTED Technical Committee on Image Processing from 2007 and a member of the IASTED Technical Committee on Artificial Intelligence and Expert Systems from 2011. \n\nHe has held the general chair position for the following: ACM-APPIS (2020, 2021), IEEE-IWOBI (2019, 2020 and 2020), A PPIS (2018, 2019), IEEE-IWOBI (2014, 2015, 2017, 2018), InnoEducaTIC (2014, 2017), IEEE-INES (2013), NoLISP (2011), JRBP (2012), and IEEE-ICCST (2005)\n\nHe is an associate editor of the Computational Intelligence and Neuroscience Journal (Hindawi – Q2 JCR-ISI). He was vice dean from 2004 to 2010 in the Higher Technical School of Telecommunication Engineers at ULPGC and the vice dean of Graduate and Postgraduate Studies from March 2013 to November 2017. He won the “Catedra Telefonica” Awards in Modality of Knowledge Transfer, 2017, 2018, and 2019 editions, and awards in Modality of COVID Research in 2020.\n\nPublic References:\nResearcher ID http://www.researcherid.com/rid/N-5967-2014\nORCID https://orcid.org/0000-0002-4621-2768 \nScopus Author ID https://www.scopus.com/authid/detail.uri?authorId=6602376272\nScholar Google https://scholar.google.es/citations?user=G1ks9nIAAAAJ&hl=en \nResearchGate https://www.researchgate.net/profile/Carlos_Travieso",institutionString:null,institution:{name:"University of Las Palmas de Gran Canaria",institutionURL:null,country:{name:"Spain"}}},editorTwo:null,editorThree:null,series:{id:"14",title:"Artificial Intelligence",doi:"10.5772/intechopen.79920",issn:"2633-1403"},editorialBoard:[{id:"13633",title:"Prof.",name:"Abdelhamid",middleName:null,surname:"Mellouk",slug:"abdelhamid-mellouk",fullName:"Abdelhamid Mellouk",profilePictureURL:"https://mts.intechopen.com/storage/users/13633/images/1567_n.jpg",institutionString:null,institution:{name:"Paris 12 Val de Marne University",institutionURL:null,country:{name:"France"}}},{id:"109268",title:"Dr.",name:"Ali",middleName:null,surname:"Al-Ataby",slug:"ali-al-ataby",fullName:"Ali Al-Ataby",profilePictureURL:"https://mts.intechopen.com/storage/users/109268/images/7410_n.jpg",institutionString:null,institution:{name:"University of Liverpool",institutionURL:null,country:{name:"United Kingdom"}}},{id:"3807",title:"Dr.",name:"Carmelo",middleName:"Jose Albanez",surname:"Bastos-Filho",slug:"carmelo-bastos-filho",fullName:"Carmelo Bastos-Filho",profilePictureURL:"https://mts.intechopen.com/storage/users/3807/images/624_n.jpg",institutionString:null,institution:{name:"Universidade de Pernambuco",institutionURL:null,country:{name:"Brazil"}}},{id:"38850",title:"Dr.",name:"Efren",middleName:null,surname:"Gorrostieta Hurtado",slug:"efren-gorrostieta-hurtado",fullName:"Efren Gorrostieta Hurtado",profilePictureURL:"https://mts.intechopen.com/storage/users/38850/images/system/38850.jpg",institutionString:null,institution:{name:"Autonomous University of Queretaro",institutionURL:null,country:{name:"Mexico"}}},{id:"239041",title:"Dr.",name:"Yang",middleName:null,surname:"Yi",slug:"yang-yi",fullName:"Yang Yi",profilePictureURL:"https://mts.intechopen.com/storage/users/239041/images/system/239041.jpeg",institutionString:null,institution:{name:"Virginia Tech",institutionURL:null,country:{name:"United States of America"}}}]},onlineFirstChapters:{paginationCount:10,paginationItems:[{id:"82804",title:"Psychiatric Problems in HIV Care",doi:"10.5772/intechopen.106077",signatures:"Seggane Musisi and Noeline Nakasujja",slug:"psychiatric-problems-in-hiv-care",totalDownloads:2,totalCrossrefCites:0,totalDimensionsCites:0,authors:null,book:{title:"Future Opportunities and Tools for Emerging Challenges for HIV/AIDS Control",coverURL:"https://cdn.intechopen.com/books/images_new/11575.jpg",subseries:{id:"6",title:"Viral Infectious Diseases"}}},{id:"82817",title:"Perspective Chapter: Microfluidic Technologies for On-Site Detection and Quantification of Infectious Diseases - 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