DCµG’s constraints.
\\n\\n
More than half of the publishers listed alongside IntechOpen (18 out of 30) are Social Science and Humanities publishers. IntechOpen is an exception to this as a leader in not only Open Access content but Open Access content across all scientific disciplines, including Physical Sciences, Engineering and Technology, Health Sciences, Life Science, and Social Sciences and Humanities.
\\n\\nOur breakdown of titles published demonstrates this with 47% PET, 31% HS, 18% LS, and 4% SSH books published.
\\n\\n“Even though ItechOpen has shown the potential of sci-tech books using an OA approach,” other publishers “have shown little interest in OA books.”
\\n\\nAdditionally, each book published by IntechOpen contains original content and research findings.
\\n\\nWe are honored to be among such prestigious publishers and we hope to continue to spearhead that growth in our quest to promote Open Access as a true pioneer in OA book publishing.
\\n\\n\\n\\n
\\n"}]',published:!0,mainMedia:{caption:"IntechOpen Maintains",originalUrl:"/media/original/113"}},components:[{type:"htmlEditorComponent",content:'
Simba Information has released its Open Access Book Publishing 2020 - 2024 report and has again identified IntechOpen as the world’s largest Open Access book publisher by title count.
\n\nSimba Information is a leading provider for market intelligence and forecasts in the media and publishing industry. The report, published every year, provides an overview and financial outlook for the global professional e-book publishing market.
\n\nIntechOpen, De Gruyter, and Frontiers are the largest OA book publishers by title count, with IntechOpen coming in at first place with 5,101 OA books published, a good 1,782 titles ahead of the nearest competitor.
\n\nSince the first Open Access Book Publishing report published in 2016, IntechOpen has held the top stop each year.
\n\n\n\nMore than half of the publishers listed alongside IntechOpen (18 out of 30) are Social Science and Humanities publishers. IntechOpen is an exception to this as a leader in not only Open Access content but Open Access content across all scientific disciplines, including Physical Sciences, Engineering and Technology, Health Sciences, Life Science, and Social Sciences and Humanities.
\n\nOur breakdown of titles published demonstrates this with 47% PET, 31% HS, 18% LS, and 4% SSH books published.
\n\n“Even though ItechOpen has shown the potential of sci-tech books using an OA approach,” other publishers “have shown little interest in OA books.”
\n\nAdditionally, each book published by IntechOpen contains original content and research findings.
\n\nWe are honored to be among such prestigious publishers and we hope to continue to spearhead that growth in our quest to promote Open Access as a true pioneer in OA book publishing.
\n\n\n\n
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\r\n\tCardiovascular disease (CVD) is the largest contributor to death worldwide. Early studies demonstrated that high consumption of fat increased the risk for CVD and mortality. Subsequently, certain studies indicated that replacing a portion of saturated fatty acids with monounsaturated and polyunsaturated fatty acids decreased the risk for CVD. In addition, it has been shown that intake of saturated fat increases total cholesterol concentrations. However, cholesterol is found in a variety of components, such as low-density lipoprotein (LDL), very-low-density lipoprotein (VLDL), and high-density lipoprotein (HDL) particles. The consumption of saturated fat tends to increase both LDL and HDL cholesterol concentrations. Interestingly, the intake of saturated fat has been reported to increase large LDL particles, which are less atherogenic, compared to small LDL particles. To add to the complexity, low-fat, high-carbohydrate diets likely increase triglyceride and decrease HDL concentrations.
\r\n\r\n\tFurthermore, the consumption of certain food components, including fiber, phytochemicals, micronutrients, and probiotics, may partially offset the individual effects of fatty acids on CVD risk factors. Other interesting and timely health topics regarding fat and fatty acids include seizures and depression. A high-fat, low-carbohydrate diet has been used to treat individuals who experience seizures. Additionally, some authors have indicated that omega-3 fatty acids may treat depression.
\r\n\r\n\tTherefore, the aim of this book will provide recent evidence on the effects of fat and fatty acid consumption on CVD risk, seizures, and depression.
\r\n\t
Nowadays distributed generators, storage systems, and controllable loads much more actively and simultaneously contribute to an optimal operation of the electrical distribution systems in the frame of the new concepts of smart grids (SGs) and microgrids (μGs).
In this exciting context, new technologies and services are being introduced to make the electrical networks more reliable, efficient, secure, and environmentally friendly.
This chapter deals with μGs that CIGRÉ C6.22 Working Group defines as electricity distribution systems containing loads and distributed energy resources that can be operated in a controlled, coordinated way either while they are connected to the main power network (grid-connected mode), or when operate in islanding mode [1].
As well known, μGs can be AC and DC μGs on the basis of the nature of the supplied voltage [2–4]. AC μGs have the advantage of utilizing existing AC technologies as well as DC μGs seem particularly suitable for supporting the current needs because most distributed generation sources, such as photovoltaic plants, fuel cells, and storage systems, generate DC power directly. Moreover, DC grids can also guarantee high power quality levels to AC sensible loads, even though this provision requires the presence of additional DC/AC converters.
Recently, hybrid AC/DC μGs have been developed with the aim to exploit the advantages of both AC and DC solutions and, in particular, to accelerate the integration process of DC power technologies into the existing consolidated AC systems [5–10].
As well known, several static converters are installed in the hybrid μGs, either to connect renewable generation and storage systems to the AC or DC grids, or as an interface between AC and DC grids. In this context, optimization strategies are required to perform the converters optimal control with the aim of efficiently operating the whole hybrid μG. These control strategies should take into account all goals and operating constraints of both DC and AC sections.
Motivated by the above requirements, in the following sections of this chapter, we propose an optimal control strategy for a hybrid μG to be realized in an actual Italian industrial facility. In particular, the static converters of the hybrid μG are controlled with the aim to simultaneously minimize the energy costs and to compensate waveform distortions, thus ensuring an optimal technical and economical behavior of the whole μG.
The remainder of the chapter is organized as follows. The electrical system of the actual Italian industrial facility is briefly recalled in Section 2, where also the structure of the proposed hybrid microgrid is shown. Section 3 deals with the proposed control strategy. Numerical applications are shown in Section 4, Section 5 provides our conclusions, and some data of the test system under study are explained in Appendix.
The electrical industrial system under study was a hybrid AC/DC microgrid obtained by the modification of the existing LV distribution system of an actual Italian industrial facility where transformers are assembled. The simplified electrical scheme before the modification is reported in Figure 1. The existing facility’s electrical distribution system is connected to the MV grid through a 20/0.4–630 kVA transformer and includes four low-voltage feeders; each feeder is dedicated to a different manufacturing process, i.e.: (i) tanks and boxes manufactory, (ii) assembly, (iii) winding and coils and (iv) test. An automatically switched 120 kVAr capacitor bank with 10 kVAr step is installed at bus 3 to guarantee a power factor at PCC (bus 2) equal to 0.95.
Electrical simplified scheme of the existing electrical distribution system of the industrial plant under study.
The data of the lines, transformers and loads are reported in Appendix.
The modifications on the existing electrical system of Figure 1 and the applied control strategy implemented in the centralized control system (CCS) are finalized (i) to improve the continuity and quality of the energy delivered to the loads and (ii) to optimize the exchange of energy between industrial plant and MV distribution grid.
The following modifications are effected (Figure 2): (i) a DC micro grid (DCμG) is connected at AC bus 16 through an AC/DC grids interfacing converter; (ii) a Battery Energy Storage System (BESS) is installed at AC bus 3 through a DC/AC static converter; and (iii) a dispatchable micro-turbine is connected at AC bus 20 through an AC/AC static converter.1
Scheme of the hybrid AC/DC µG.
The DC micro grid (DCμG) consists of a photovoltaic (PV) plant equipped with a maximum power point tracker control system and connected to the DC grid through a DC/DC static converter and three sensitive AC loads (Folding walls island robot, Sandblasting machine and PLCs used to control the automation of the whole electromechanical process2) moved from the original electrical scheme of Figure 1.
The sensitive loads are controllable in terms of start-up times; in particular, their working time intervals are obtained by a day-ahead scheduling aimed at achieving the minimization of the costs for the electricity purchase of the hybrid AC/DC μG [12].3
The above modifications lead to a hybrid μG, which includes AC buses (ACμG) and DC buses (DCμG).
The data of the newly added components are reported in Appendix.
The proposed control strategy is based on the solution of optimization problems performed at fundamental and at harmonic frequencies, where the efficient operation of the DC and AC grids is guaranteed by a proper control of the static converters installed in the hybrid μG, either to connect micro-turbine and storage system to the AC grid, or as interface between AC and DC grids.
The control strategy allows the hybrid AC/DC μG to operate both in grid-connected and in islanding mode as described in details in the following subsections.
When the hybrid μG is in
The converter sizes have to be appropriately chosen to correctly perform both the required services.
Two different optimization models are formulated and solved by CCS to provide the reference signals for the energy cost minimization (reference signals at fundamental frequency) and for the waveform distortion compensation (reference signals at harmonic frequencies), as shown in Figure 3.
Optimal control strategy for grid-connected mode.
At the fundamental frequency, the real-time operation of the hybrid AC/DC µG is optimized by controlling the active and reactive power of the BESS, the active and reactive power of the micro-turbine and the reactive power provided by the AC/DC grids interfacing converter. The reference signals for the converters, which minimize the total energy cost, are obtained solving a non-linear constrained optimization problem, such as:
s.t.
where
Dividing the day into Nt time slots of the same duration ∆
Input and output data, the objective function and the constraints structure are listed in the following.4
The input and output data of the optimization problem are:
the state of charge of the BESS at the end of the
the forecasted powers of the PV system and of the non-controllable loads, all provided by proper forecasting tools for all the time slots from (
the power required by the controllable loads5;
the energy charge;
the charge and discharge periods of the BESS in ACµG which are fixed a-priori in function of the energy hourly tariff (typically, the discharge period is during the hours of peak energy tariff).
the active and reactive power of the BESS and the corresponding State of Charge (SOC);
the active and reactive powers of the microturbines;
the reactive power of the interfacing power converter.
All the output data are calculated from the (
The
In Eq. (4),
In Eq. (5),
The
DCµG’s constraints.
AC grid |
---|
ACµG’s constraints.
Interfacing converter | |
---|---|
Constraints on AC/DC grids interfacing converter.
With reference to the DCµG constituted by
In Eq. (6),
In particular,
In Eq. (7),
With reference to the ACµG constituted by
In Eq. (9),
at the load bus bars,
at bus 3,
at bus 20,
at bus 16,
at buses, where no generation or loads are connected, the
In Eq. (10),
In Eq. (11),
In Eq. (12), the active power
In Eq. (13),
In Eq. (14), the active power produced by micro-turbine
In Eq. (15),
In Eq. (16),
In Eq. (17), the upper value
In Eq. (18),
In Eq. (19),
With reference to AC/DC grids interfacing converter (Table 3), the equality constraints are the DC and AC voltage balance equation (Eq. (20)) and the DC and AC active power balance equation (Eq. (21)), while an inequality constraint (Eq. (22)) refers to a limit on apparent power flowing through the AC/DC grids interfacing converter.
In Eq. (20),
In Eq. (21),
In Eq. (22), the active (
At harmonic frequencies, the centralized control system furnishes reference current signals that ACµ
The Fourier transform
In Eq. (23),
The analytical Fourier transform of the vector of the injected currents
where
The matrices
where
It should be noted that the values of the element of vector
It should be also noted that the values of the weight matrix
In addition, the control strategy applied by CCS is able to disconnect the hybrid μG from the distribution network when abnormal conditions occur. We will consider the behavior of the AC/DC µG in the case of supply interruptions. In such conditions, local energy resources and loads are optimally controlled in order to guarantee high power quality levels to priority loads. As priority loads, we refer to electrical loads requiring high levels of continuity of supply.
In particular, in islanding mode, the so-called “Single Mater Operation” control strategy is adopted [24]. In fact, while in grid-connected mode all inverters installed into the hybrid μG operate in PQ mode (inverter is used to supply a given active P and reactive Q power set point), in islanding mode the BESS interface converter operates feeding the μG with predefined values of voltage and frequency (V-f control mode), as well as the remaining inverters, on the contrary, continue to operate in PQ mode.
As soon as the hybrid μG is in the islanding mode, all loads are disconnected except the priority loads whose power demand can be provided by the BESS and micro-turbine (the PV system is not considered in the power balance because its availability depends on the uncertain presence of the primary source). As an example, in the considered case study, the priority loads are assumed to be the sensitive loads connected to the DCµG. In this case, the maximum power required by the priority loads is 72 kW (corresponding to the power absorbed when all sensitive loads are working). Also in the worst conditions (long interruption arising when BESS charge is
Eventually, in islanding mode, the proposed control strategy starts to supply the priority loads. Anyway, according to a predefined list of load priorities, some of the disconnected loads can be reconnected taking into account of: (i) the available power from PV generator; (ii) the scheduled power required by sensitive loads; (iii) the maximum time fixed for the autonomous operation of the hybrid µG; and (iv) the value of the SOC of battery. The available energy for the islanding operation can be easily calculated from the knowledge of the SOC of battery, the forecasted PV power generation in the time intervals successive to the disconnection and the rated power of micro-turbine.
A simulation of the hybrid micro grid of Section 2 using
Remind that, in the grid-connected mode, the converters participating in the energy cost minimization problem are: (i) the BESS converter at bus 3; (ii) the AC/DC grids interfacing converter at bus 16; and (iii) the micro-turbine converter at bus 20. In particular, the AC/DC grids interfacing converter (micro-turbine) participates optimally exchanging only reactive (active) power with the ACµG.
The converters participating in the harmonic compensation are: (i) the AC/DC grids interfacing converter at bus 16 and (ii) the micro-turbine converter at bus 20. Note also that sizes of the converters, shown in Table A. VIII, are chosen according to the aforesaid operating conditions. Specifically, while the 120 kVA of the BESS converter is fully utilized for the energy cost minimization, only 80 kVA of the AC/DC grids interfacing converter and 30 kVA of the micro-turbine converter are addressed for the same operation. The remaining 20 kVA of the AC/DC grids interfacing converter and 10 kVA of the micro-turbine converter are used for the harmonic compensation.
The considered day was divided into 72-time slot of equal duration (20 min), and the inputs of the proposed control strategy for the energy cost minimization are:
The forecasted powers of the PV system and of the non-controllable loads, both obtained before each time slot for all the successive time slots of the considered day. As an example, Figures 4 show the forecasted power of the PV system (Figure 4a) and the forecasted active power (solid line), reactive power (dotted line) of the non-controllable load at bus 33 (Figure 4b) obtained at the first time slot;
The scheduled power profile of the controllable loads, as illustrated in Figure 5;
The energy charge reported in Figure 6 [25]. This tariff is applicable to medium and small industrial customers and for service in the common areas of multi-family complexes. It was selected since it is characterized by a large spread between on peak and off peak prices; this is a necessary condition to make it possible a fruitful use of storage systems nowadays;
The SOC of the BESS at bus 3 equal to 43.36% at the beginning of the day. The same value is required also at the end of the considered day according to scheduling procedure.
Forecasted power profile for the first time slot: (a) power production of the PV system; (b) active (solid line) and reactive (dotted line) power of the non-controllable load at bus 33.
Scheduled profile of the power of the controllable loads.
Energy charge.
The time behavior of the main quantities of interest is illustrated in the following relatively to the considered working day.
Figure 7 shows the active (solid line) and reactive (dotted line) power profiles of the BESS converter. In Figure 8, the active (solid line) and reactive (dotted line) power profiles of the AC/DC grids interfacing converter are plotted. Figure 9 shows the reactive power supplied by the controllable capacitor bank. Eventually, in Figure 10, the active (solid line) and reactive (dotted line) power profiles at the PCC are reported. The active power of the micro-turbine is not reported since it always operates at a constant value of 30 kW.
Active (solid line) and reactive (dotted line) power profiles related to the BESS converter at bus 3.
Active (solid line) and reactive (dotted line) power profiles related to the AC/DC grids interfacing converter at bus 16.
Reactive power profile of the capacitor bank at bus 3.
Active (solid line) and reactive (dotted line) power profiles at the PCC.
From the analysis of Figures 7–10, the following considerations can arise:
the µG operates practically at unitary power factor all over the day (Figure 10); the measured power factor of the industrial system (without the hybrid µG), instead, was 0.85 on average. Indeed, the forecasting error on PV power generation and on non-controllable load powers causes an operation of network slightly different to unity power factor and, as a consequence, the reactive power absorbed by the hybrid µG at the PCC is not exactly zero;
the reactive power furnished by BESS and capacitor bank is maximized in the hours corresponding to load peak power (Figures 7 and 9);
the active power provided by the BESS during the discharge from 12:00 to 14:00 is limited by the converter size (in this interval also reactive power is furnished by the converter);
the BESS discharge reduction at 16:20 and the residual BESS discharge at 17:20 are justified by the numerical deviation between the BESS set point and the corresponding simulation response (Figure 7).
Anyway, the BESS totally discharges (i.e. the SOC reaches 20%) in the interval where the tariff is higher, maximizing in this way the economic benefit.
Note also that the reactive power production inside the µG allows not only the reduction of the power losses but also the maximization of the internal active power generation, determining the minimization of the objective function. In fact, an increasing of the mere internal active power production determines a corresponding decreasing of power factor at the PCC; therefore, a production of reactive power inside the hybrid grid is needed to guarantee an adequate power factor at the PCC.
In Figure 11, a comparison among the energy charge profile (dotted line) and the active power (solid line) of BESS converter (Figure 11a) and the active power of AC/DC grids interfacing converter (Figure 11b) is reported. Figure 12 shows a comparison among the energy charge profile (dotted line), the active power profile at the PCC of the hybrid µG (solid line) and of the actual industrial facility (dashed line).
Comparison among the energy charge profile (dotted line), the active power profiles (solid line) related to: (a) the BESS converter at bus 3 and (b) to the AC/DC grids interfacing converter at bus 16.
Comparison among the energy charge profile (dotted line), the active power profile at the PCC of the industrial system with µG (solid line) and of the actual industrial system (dashed line).
The analysis of these last figures clearly evidences that:
to maximize the economic benefits, the maximum allowable discharge for the BESS arises in the hours of the maximum value of tariff;
the active power flowing from DC to AC µG is maximized in the hours of the maximum value of tariff, according to the day-ahead scheduling procedure that shifted the controllable loads far from the maximum tariff price and the PV generation available during those hours;
the active power at the PCC of hybrid µG increases respect to the value of the existing industrial facility when the tariff price is minimum, due to: (i) the BESS charge profile and (ii) the shifting of the controllable loads; on the other hand, the active power at the PCC of hybrid µG decreases when the tariff price assumes higher values, due to: (i) the shifting of the controllable loads; (ii) the PV system production; and (iii) the BESS discharge profile.
The aforesaid observations justify the reduction, shown in Figure 13, of the hourly costs to be sustained by the hybrid µG (solid line) respect to the ones sustained by the existing industrial facility (dotted line). In particular:
from 00:00 to 04:00 and from 22:00 to 23:00, the hybrid µG has costs slightly greater than the existing industrial system, cause of the BESS charge;
from 06:00 to 07:00 and from 21:00 to 22:00, the hybrid µG has costs slightly greater than the existing industrial system, cause of the shifting of controllable loads;
from 10:00 to 18:00, the hybrid µG has cost significantly lower than the existing industrial system, cause of both the PV system production and BESS discharge;
from 18:00 to 21:00, the hybrid µG has costs lower than the existing industrial system, mainly cause of the micro-turbine operation, whose production costs are lower than the considered energy tariff, and also due to the PV system production.
Hourly costs to be sustained by the industrial system with hybrid µG (solid line) and by the actual industrial system (dotted line).
Globally, the proposed procedure allows a daily reduction of the total costs for the energy purchase of about 26%.
With reference to the waveform distortion compensation, an analysis of the load currents in the existing industrial system revealed that main contributions to voltage distortions were due to the harmonic of order h = 5, 7, 11 and 13. So, the procedure described in Section 3.1.2 was applied only for the compensation of the aforesaid harmonic orders. The compensation action is applied in a working day using the AC/DC grids interfacing converter and the micro-turbine converter.
As illustrated in Section 3, the inputs needed to solve the minimization problem and to obtain the reference currents of converters (see Eq. (23)) are the estimated harmonic disturbances
The loads producing waveform distortion are marked with an asterisk in Table A. VI. As an example, Figures 14 and 15 report the harmonic currents amplitudes injected by disturbing loads at the buses 23 and 33 during the time slot from 16:20 to 16:40.
DFT estimation of harmonic currents amplitudes injected by disturbing loads at the bus 23 during the time slot from 16:20 to 16:40.
DFT estimation of harmonic currents amplitudes injected by disturbing loads at the bus 33 during the time slot from 16:20 to 16:40.
The terms of the weight matrix
The terms of weight matrix
Figures 16–18 show the comparison of total harmonic distortion (THD) with (solid bars) and without (empty bars) the proposed compensation procedure. Specifically, Figure 16 shows the THD comparison over the buses in the time slot corresponding to the time interval from 16:20 to 16:40, while Figures 17 and 18 show the THD comparison, in the whole day, for the buses 23 and 35, respectively. The benefits in term of THD reduction with the application of the proposed procedure are evident and significant, since, as observed in Figures 16–18, the THD values in all of the buses of the hybrid µG in time stayed always under the 0.6% with the waveform distortion compensation. Without compensation, the THD overcame the 4.5% in some buses for some time slots.
THD comparison with (solid bars) and without (empty bars) the proposed compensation procedure corresponding to the time interval from 16:20 to 16:40.
THD comparison with (solid bars) and without (empty bars) the proposed compensation procedure corresponding to the bus 23.
THD comparison with (solid bars) and without (empty bars) the proposed compensation procedure corresponding to the bus 35.
To simulate islanding mode of operation of the hybrid μG, an interruption in the MV distribution network supplying the industrial facility was simulated. The interruption determines the switching from grid-connected mode to islanding mode. The control strategy described in Section 3.2 is applied; the BESS converter switches from P-Q control mode to V-f control mode since the RMS voltage at PCC becomes lower than 5% of nominal voltage [26], that is the threshold for the switching of the BESS converter control strategy. The remaining inverters, on the contrary, continue to operate in P-Q mode. The interruption arises at 6 h 25′ 1″ of the working day analyzed in Section 4.1. As soon as the islanding condition arises, all loads of the hybrid μG are disconnected except the sensitive loads present in the DC μG. As illustrated in Figure 5, at 6 h 25′ all sensitive loads are working and absorbing 72 kW, micro-turbine and photovoltaic generators produce 30 and 31 kW, respectively. BESS is characterized by a SOC of about 100%.
In Figure 19, the voltage profile at bus 3 versus time is reported. In Figure 20, the active power injected by BESS and AC/DC grids interfacing converters are shown. Figures 19 show how the proposed approach allows to supply the hybrid μG during the interruption with obvious advantages in terms of continuity of the supplied energy.
Phase-A voltage at bus 3 versus time starting from 6 h 25′.
Active (a) and reactive (b) powers injected by BESS, micro-turbine and AC/DC grids interfacing converter starting from 6 h 25′.
Figure 20a shows the increasing of BESS active power from the value at 6 h 25′ in grid-connected mode (see also Figures 7 and 11) to the value needed to supply the hybrid μG in islanded mode. In particular, this value, in the considered application, is related to the grid active power losses and active power required by the sensitive loads connected to DCμG reduced by the active power furnished by micro-turbine and PV system. As expected, the active power flowing in the AC/DC grids interfacing converter remains constant to the value of grid-connected mode (see also Figures 8 and 11).
In Figure 20b, reactive power variations from the values at 6 h 25′ in grid-connected mode (see also Figures 7 and 8) to the values needed to provide the reactive power required by the hybrid μG in islanding mode is shown. In particular, since only the sensitive loads connected to DC μG are working, only reactive power losses are required and provided by BESS converter.
Note that the active and reactive power transients shown in Figure 20 are due to the switching of the BESS converter regulator from P-Q control mode to voltage control mode. Moreover, although the other converters in the hybrid µG continue to be controlled in P-Q mode, also their responses are influenced by the transient at bus 3.
Accordingly to the aim of the proposed procedure, all the distributed resources in the hybrid μG guarantee the continuity of supplying of sensitive loads.
A hybrid µG including two interconnected sections operating in DC and AC was considered starting from an existing Italian industrial facility. The DC section included non-dispatchable generation systems and controllable loads; the AC section included a BESS, a micro-turbine, non-controllable loads and non-linear loads.
A control strategy based on a real-time control, at both the fundamental frequency and harmonic frequencies, was applied. The control strategy required the solution of optimization problems and was aimed at minimizing the operation costs and improving the power quality levels.
The results of a numerical application relative to a typical working day are shown and demonstrated the effectiveness of the procedure in reducing the daily operating costs while guaranteeing higher values of the quality of the power supply.
This work was supported by Italian Ministero dell\'Istruzione dell\'Università e della Ricerca (MIUR) in the frame of PON03PE_00178_1: Microgrid Ibride in Corrente Continua e Corrente Alternata (MICCA) and by Università of Napoli Parthenope in the framework of “Bando per il Sostegno alla Ricerca individuale triennio 2015–2017”.
In the following, the data of the lines are provided. In particular, for each line, Tables A. I, A. II, A. III, and A. IV show the starting and ending buses, the length, resistance, and reactance. Tables A. V and A. VI report the transformers and loads data. Tables A. VII and A. VIII report the main data of the new components installed in the proposed hybrid μG.
Buses | ℓ [m] | |||
---|---|---|---|---|
From | To | |||
3 | 4 | 8 | 0.041 | 0.014 |
4 | 5 | 24 | 0.163 | 0.130 |
5 | 6 | 4 | 0.473 | 0.101 |
5 | 7 | 0.5 | 0.163 | 0.130 |
7 | 8 | 6 | 0.163 | 0.130 |
8 | 9 | 10 | 1.410 | 0.112 |
8 | 10 | 9.3 | 0.163 | 0.130 |
10 | 11 | 3 | 0.163 | 0.130 |
11 | 12 | 2.8 | 0.163 | 0.130 |
12 | 13 | 3.5 | 0.163 | 0.130 |
13 | 14 | 3.5 | 0.163 | 0.130 |
14 | 15 | 11 | 0.163 | 0.130 |
15 | 16 | 19.1 | 0.163 | 0.130 |
16 | 17 | 4 | 1.410 | 0.112 |
16 | 18 | 1.9 | 0.163 | 0.130 |
18 | 19 | 4 | 1.410 | 0.112 |
18 | 20 | 3 | 0.163 | 0.130 |
20 | 21 | 10 | 0.236 | 0.097 |
20 | 22 | 42 | 1.410 | 0.112 |
20 | 23 | 61 | 2.240 | 0.119 |
20 | 24 | 61 | 1.410 | 0.112 |
Line parameters for the feeder “Tanks and boxes manufactory”.
Buses | ℓ [m] | |||
---|---|---|---|---|
From | To | |||
4 | 25 | 31 | 0.041 | 0.014 |
25 | 26 | 3.5 | 0.163 | 0.130 |
26 | 27 | 16 | 2.240 | 0.119 |
26 | 28 | 9 | 0.163 | 0.130 |
28 | 29 | 12.5 | 0.163 | 0.130 |
29 | 30 | 10.5 | 0.163 | 0.130 |
30 | 31 | 8 | 0.641 | 0.101 |
30 | 32 | 1.5 | 0.163 | 0.130 |
32 | 33 | 10 | 0.641 | 0.101 |
32 | 34 | 12.5 | 0.163 | 0.130 |
34 | 35 | 8 | 0.328 | 0.096 |
34 | 36 | 13 | 0.163 | 0.130 |
36 | 37 | 1 | 0.163 | 0.130 |
37 | 38 | 35 | 0.665 | 0.260 |
38 | 39 | 0.5 | 0.665 | 0.260 |
39 | 40 | 0.5 | 0.665 | 0.260 |
40 | 41 | 20 | 1.410 | 0.112 |
Line parameters for the feeder “Assembly”.
Buses | ℓ [m] | |||
---|---|---|---|---|
From | To | |||
25 | 42 | 34 | 0.041 | 0.014 |
42 | 43 | 3.3 | 0.070 | 0.096 |
43 | 44 | 12 | 2.240 | 0.119 |
43 | 45 | 9.2 | 0.070 | 0.096 |
45 | 46 | 5.5 | 0.070 | 0.096 |
46 | 47 | 5.5 | 1.410 | 0.112 |
46 | 48 | 6.5 | 0.070 | 0.096 |
48 | 49 | 5.5 | 1.410 | 0.112 |
48 | 50 | 7.8 | 0.070 | 0.096 |
50 | 51 | 5.5 | 1.410 | 0.112 |
50 | 52 | 5.2 | 0.070 | 0.096 |
52 | 53 | 1.8 | 0.070 | 0.096 |
53 | 54 | 5.5 | 1.410 | 0.112 |
53 | 55 | 8.1 | 0.070 | 0.096 |
55 | 56 | 5.5 | 1.410 | 0.112 |
55 | 57 | 5.6 | 0.070 | 0.096 |
57 | 58 | 4.5 | 0.070 | 0.096 |
58 | 59 | 5.5 | 1.410 | 0.112 |
58 | 60 | 3 | 0.070 | 0.096 |
60 | 61 | 8 | 0.473 | 0.101 |
60 | 62 | 2.5 | 0.070 | 0.096 |
62 | 63 | 5.5 | 1.410 | 0.112 |
62 | 64 | 3.7 | 0.070 | 0.096 |
64 | 65 | 38 | 1.410 | 0.112 |
Line parameters for the feeder “Winding and coils”.
Buses | ℓ [m] | |||
---|---|---|---|---|
From | To | |||
2 | 66 | 30 | 0.094 | 0.090 |
66 | 67 | 87 | 0.163 | 0.130 |
67 | 68 | 7 | 1.410 | 0.112 |
67 | 69 | 0.5 | 0.163 | 0.130 |
69 | 70 | 7 | 0.473 | 0.101 |
Line parameters for the feeder “Test”.
20 kV/400 V | |
630 kVA | |
Dyn | |
11 | |
6.0% | |
5721 W | |
925 W | |
5% |
Transformer data.
Bus | Type | Rated power [kVA] | Power factor |
---|---|---|---|
3 | Heating and cooling system | 200 | 0.8 |
6 | Painting machine | 75 | 0.8 |
7 | Box overturning machine | 4 | 0.99 |
9* | Sandblasting machine | 55 | 0.75 |
11 | Welder aspirators | 11 | 0.99 |
15 | Manual bender | 8 | 0.99 |
17* | Folding walls island robot | 24 | 0.99 |
19* | Wave welding machine | 30 | 0.65 |
21 | Automated bending robot for corrugated panels | 122 | 0.65 |
22* | PLC + computer | 3 | 0.62 |
23* | Plasma cutting machine | 15 | 0.8 |
27 | Crane | 5.5 | 0.8 |
31*, 33* | Core cutting machine n. 1 | 60 | 0.8 |
35 | Autoclaves | 86 | 0.8 |
44 | Furnace | 5 | 0.99 |
47*, 49*, 51* | MT winder machine | 37 | 0.99 |
54*, 56* | Tuboly winder machine | 37 | 0.99 |
59*, 63* | BT winder machine | 37 | 0.99 |
61 | Offices | 36 | 0.99 |
68* | Automated bending robot for metal plates | 20 | 0.9 |
70 | Testing bench room | 50 | 0.7 |
Load data.
(a) | |
---|---|
PV generator | |
60 kWp | |
0.95 | |
DC v | 800 V |
(b) | ||||
---|---|---|---|---|
DCμG buses | ℓ [m] | |||
1 | 2 | 20 | 1.450 | 0.00032 |
1 | 3 | 30 | 0.660 | 0.00029 |
1 | 4 | 10 | 3.950 | 0.00038 |
1 | 5 | 35 | 0.660 | 0.00029 |
DCμG components: PV generator (a) and lines data (b).
BESS | Micro-turbine | AC/DC grids interfacing converter | |
---|---|---|---|
120 kVA | 40 kVA | 100 kVA | |
400 V | 400 V | 400 V | |
0.95 | 0.95 | 0.95 | |
800 V | – | 800 V | |
510 kWh | – | – | |
20% | – | – | |
102 kW | – | – |
ACμG components data.
Global mortality rate from diarrhea in children under 5 years in 2016. Data represent the analysis of diarrhea burden in 195 countries in 1990–2016, showing the regions most affected by the illness. Reprinted from Troeger et al. [
The treatment of
Assessing the antimicrobial resistance, virulence, and transmission dynamics of
Based on clinical manifestation, presence of specific virulence determinants and phylogenetic profiles, diarrheagenic
Enterotoxigenic
Dissemination and transmission routes of pathogenic
There is a great genetic diversity in ETEC as more than 100 different O antigens have been reported to be associated with clinical isolates. Among these, the O6 serogroup is most common, and geographically diverse among all ETEC serogroups, and has been implicated in multiple outbreaks in different countries [21]. Additionally, at least 34 H antigens are also associated with this pathotype. Among the serotypes as determined by the combination of O and H antigens, O6:H16 (heat-stable (ST) or heat-labile (LT) toxin), O148:H28 (ST), O167:H5, O153:H45 (ST), O169: H41 (ST only) are frequently isolated from humans, animals, environmental matrices, and from outbreaks in developing countries [22, 23, 24]. ETEC produces one or more colonization factors that facilitate its attachment to specific receptors on the mucosal layer of the small intestine of humans and animals, and secretes enterotoxins that cause electrolyte imbalance in the intestinal lumen resulting in dehydration, metabolic acidosis, and diarrheal [3, 18]. The ST toxin is a nonimmunogenic small protein molecule, but the LT toxin is structurally homologous and exhibits a similar mechanism of action to cholera toxin produced by
ETEC employs an array of genetic factors that are either chromosomal or plasmid-borne that mediate colonization and adherence to the intestinal epithelium, proliferation within the host, and evasion of host defense mechanisms (Table 1) [26].
Main reservoir | Clinical presentations | Virulence factors | |
---|---|---|---|
ETEC | Humans and animals | Watery noninflammatory diarrhea, adherence to small intestinal epithelium, nutrient malabsorption | Colonization factors (CFs), EatA, Tia, TibA, LeoA, ST, LT, EAST1 |
STEC | Animals | Watery bloody diarrhea, hemorrhagic colitis, Hemorrhagic Uremic syndrome, attaching-effacing lesions in the large intestine | LEE, Saa, Paa, EhaA, LpfA, OI-7, ST, EhxA, ToxB, EspP, KatP |
EPEC | Humans and animals | Shigella-like toxin, watery and/or bloody diarrhea, noninflammatory diarrhea, attaching-effacing lesions in the colon, nutrient malabsorption | LEE, pEAF, Bfp, OI-22, EAST1 |
EAEC | Humans and animals | Clump intestinal cells; mucoid watery diarrhea with persistent inflammation, nutrient malabsorption, postinfectious irritable bowel syndrome | AA, Afp, CapU, Air, Shf, AatA, Pic, EAST1, ShET1, Pet, SigA, SepA, pAA, HlyE |
EIEC | Humans and animals | Bacillary dysentery, watery diarrhea with or without blood and leukocytes, inflammation of the large intestinal epithelium | pINV, SepA, SigA, Sat, ShET2 |
DAEC | Humans | Watery diarrhea, persistent diarrhea, chronic inflammatory colon disease | Afa/Dr. Adhesins, Sat, Pet, SenB, HlyE |
Summary of the clinical characteristics and virulence factors of
Legend: ETEC: Enterotoxigenic
Colonization and adhesion are the primary and essential steps in the pathogenesis of pathogens. ETEC is not an exception as the colonization of the host intestinal epithelium by this pathotype is mediated by plasmid-borne genes that encode adhesins and one or more colonization factors (CFs) namely, pilus or pilus-related adhesins [23]. Pili are hair-like appendages on the cell surface of bacteria where they mediate the attachment of bacteria to surfaces.
They are composed of protein subunits (pilins) that are structurally polymeric and are almost exclusively plasmid-borne (Figure 3) [27]. ETEC CFs are designated as CS (coli surface antigens) followed by a number, except for CFA/I and PCFO71 [27]. Presently, at least 30 CFs have been reported in ETEC of human origin. It is estimated that about 50% of strains in this pathotype carry one or more CFs that are not detectable, suggesting that there could be more CFs that are yet to be discovered and characterized [18, 23]. The co-expression of one or more CFs with toxin-encoding genes has been described. For example, CFA/I + LT, CS7 with LT, CS5 + CS6 with LT + ST, CS2 + CS3 with LT + STh, among others [28, 29]. In the prototypical ETEC strain H10407, the production of CFA/I is mediated by
Colonization and adherence patterns of diarrheagenic
Other plasmid-encoded genetic factors that have been reported to play a significant role in the pathogenesis of ETEC include a class I SPATE (serine protease autotransporters of the Enterobacteriaceae) EatA that digests EtpA secreted by ETEC, thereby promoting the adhesion of flagella to the host receptor [30, 31]. ETEC can invade the host cell with two chromosomally encoded genes
One of the salient features that define ETEC is its ability to produce two types of enterotoxins, ST or LT [23]. STs are non-antigenic small enterotoxins that are frequent in human diseases, found in about 80% of ETEC either singly or in combination with LT [18, 33]. STs are classified into two different classes (STa and STb) based on their structure and function. STa is soluble in methanol and protease-resistant. It is frequent in human diseases and encoded by
Unlike, STs, LTs are hexameric and strongly immunogenic that are encoded by the
LTs promote the adherence of ETEC to host intestinal epithelial cells and evade the host defense mechanisms by inhibiting the expression of antimicrobial peptides produced by the hosts, in addition to the activation of host signaling pathways [3]. Another virulence factor encoding enterotoxin in ETEC strains is enteroaggregative heat-stable toxin (EAST1). EAST1 toxin is heat-stable and 38 amino acids long encoded by
Since the first isolation of ETEC in Kolkata about five decades ago [41], the emergence and increase in multidrug-resistant strains have been reported. A homogenous and high antibiotic susceptibility pattern was observed for ETEC strains at a time but the treatment of travelers’ diarrhea with different classes of antimicrobials such as macrolides (erythromycin and azithromycin), fluoroquinolones (norfloxacin, ofloxacin, ciprofloxacin), tetracycline (doxycycline), rifamycin and sulfamethoxazole-trimethoprim that are used to treat other types of infections [22] may have also contributed to the emergence of antimicrobial resistance in this pathotype [22]. Another contributor could be the indiscriminate use of antibiotics for the treatment of diarrheas caused by viral agents that are sometimes misdiagnosed because they present similar symptoms [3].
There are several studies from different countries assessing the antibiotic resistance profile and distribution of resistance determinants in ETEC. In a study, the antimicrobial resistance profile among patients with recent travel history to ETEC endemic regions between 2001 and 2004 reported that up to 60% of the ETEC isolates were resistant to sulfamethoxazole-trimethoprim, tetracycline, and/or ampicillin [42]. Ciprofloxacin resistance was reported to markedly increase from 1% to 8% within 10 years (1994–2004) in patients [42] which clearly suggests a rapid emergence of resistance with time in this pathotype. In a recent study on the WGS analyses of eight strains representing the major ETEC lineages that are causing diarrheal diseases in humans around the globe, all the strains showed resistance and carried resistance determinants to at least two of the 14 antibiotics tested, with resistance to penicillin, norfloxacin and chloramphenicol being the most common. In this study, two plasmids designated (pAvM_E1441_17 and pAvM_E2980_15) carried resistance determinants to mercury (
ETEC in animals, however, may be slightly different. In a study of 112 ETEC isolates recovered from pigs in Canada over a two-decade period (1978–2000),
ETEC strains are epidemiologically and phenotypically diverse and exhibit high genetic diversity. In addition to being polyphyletic, the distribution of ETEC lineages is not restricted by geography [45]. Several reports on the phylogenetic analyses of strains from the human origin using MLEE and MLST, and well as CF-toxin-based phylogeny showed that this pathotype might have evolved multiple times through clonal expansion and probably due to lack of common clonal lineage [46, 47]. In spite of the genomic diversity among strains in this pathotype, Turner and colleagues [48] reported ETEC to be associated with sequence type 10 (ST10). In a broader evolutionary study of a large collection of 1019 ETEC isolates from humans in 13 countries using MLST, 42 clonal groups were observed with evidence for horizontal gene exchange of plasmid-encoded CF genes between the lineages [46]. Since the advent of next-generation sequencing technologies, the study of the population structure of ETEC has improved the understanding of the genetic diversity and evolution of the pathotype [24, 49, 50].
A global collection of ETEC isolates from humans collected over a period of three decades (1980–2011) in 20 countries and representing four continents was assessed for genetic relatedness using WGS-based single nucleotide polymorphism (SNP) [49]. Indeed, ETEC strains are genetically diverse as they were reported to be distributed across different
Single nucleotide polymorphism based Phylogenetic tree of
Similarly, in a local study on the phylogenomic diversity of 94 ETEC isolates from Bangladesh [24], a polyphyletic scenario and a direct correlation between lineages and virulence profiles and CFs were noted. Using comparative genomic tools, the authors identified six novel CF variants. However, the experimental validation of these CFs would be important to decipher their association with other virulence determinants as well as their interaction with the host cells.
Shiga toxin-producing
The main reservoir for STEC strains causing infection in humans are known to be ruminant food production animals including cattle, sheep, and goats. These animals are asymptomatic carriers and shedders as the vascular receptor that facilitate the transportation of the Shiga toxins to organs are absent. This
STEC carries genes encoding adhesins and enterotoxins that are either chromosomal, on PAIs, or plasmid-borne. These determinants mediate colonization, attachments, and invasion of host cells (Table 1) [64].
Ingestion of contaminated food or direct contact with contaminated environmental matrices or infected persons or animals precedes STEC-related diseases. Colonization and attachment/adherence of STEC to intestinal epithelium is mediated by several genetic factors some of which are carried by the locus of enterocyte effacement (LEE) PAI [65]. LEE locus which is also often present in EPEC strains [66] encodes a type III secretion system (T3SS) that plays a role in the secretion and translocation of virulence-associated genetic factors into host cells [67]. One of these genetic factors is
LEE is unarguably important for the pathogenesis of STEC strains, but STEC LEE-negative strains/serotypes (e.g. O103:H21) have been implicated in infections [74]. This implies that there are several other virulence-associated factors that are carried on PAIs or mobile genetic elements mediating colonization and adherence to host cells in these strains [74]. For example, a gene (
STEC-related infections in humans are reported to be associated with the presence and expression of several virulence determinants, with the phage-encoded Shiga toxin genes Stx1 (Stx1a) and Stx2 (Stx2a) being the main virulence factors [64, 80]. Stx1 consists of 293 amino acids while the Stx2a is longer by only four amino acids. At least, 16 subtypes of these two toxins have been described based on amino acid differences and the level of cytotoxicity. Stx1 contains four variants encoded by
STEC strains can carry
Treatment of STEC infections with antibiotics is not encouraged as this might exacerbate the disease by activating the lytic cycle of the phage carrying Shiga toxin that could aggravate tissue damage in infected individuals. Antibiotics such as rifaximin, fosfomycin, azithromycin, and meropenem that do not encourage the release of Shiga toxin have been used for the treatment of early onset of STEC infection to prevent the progression of the diseases to HUS [90].
Several studies on the prevalence of antibiotic resistance in STEC from different countries and host or environments have reported that resistance to beta-lactams, sulfonamide, tetracycline, and trimethoprim are common STEC, while multidrug-resistance is more frequent in non-O157 than O157:H7 serotypes [91]. For STEC O157, resistance to ampicillin and cephalothin is common in strains of human origin, whereas tetracycline and sulphamethoxazole resistances are frequent in strains of animal origin [92]. In a study involving 54 STEC strains recovered from cattle and pigs, genetic determinants that encode resistance to trimethoprim (
Genetic relatedness of STEC isolates from different hosts and countries have been studied using different molecular tools that ranged from serotyping, PFGE, conventional MLST, and WGS. Evidence for transmission and dissemination of different STEC serogroups and clones have also been documented. Unlike ETEC that evolved multiple times through clonal expansion, STEC appears to have evolved by parallel evolution. Indeed, phylogenetic analyses of STEC strains have shown that isolates form multiple distinct clonal lineages, where strains with the same serotype and virulence content were nested together in the cluster [95]. STEC strains are spread across
In a recent study using WGS to understand the population dynamics of 757 STEC O157:H7 isolates from humans and animals from four continents, seven clades were reported and designated as A-G [101]. The most recent common ancestor of the isolates in this study was reported to have originated in the Netherlands in the late 19th century (1890) and then spread to other parts of the world. Although isolates were clustered on a geographical basis, there was an admixture of strains from different hosts suggesting transmission events between them [101]. The pangenome analyses of these isolates also showed that STEC O157:H7 from humans and animals differed in phage-related protein content. The molecular epidemiology of non-STEC O157:H7 is equally important especially considering their roles in outbreaks. From 1995 to 2017, a total of 674 outbreaks by non-O157 STEC strains were reported worldwide, where O26:H11 was predominant during this period [102]. Other serogroups implicated in these outbreaks include O26:H11, O45, O103:H25, O104:H4, O111:H8, O121, and O145:NM [102]. MLST-based phylogenetic analysis of 894 non-STEC isolates from patients over a period of 18 years (2001–2018) in Michigan revealed that the great majority of the isolates (95%) belonged to one clade [103]. Although the information on the evolutionary dynamics of STEC is inexhaustible, studies focusing on identifying new genetic factors associated with ecological adaptation of different lineages are elusive. Further studies should focus on this area.
Enteropathogenic
EPEC strains are previously classified solely based on the combination of the three immunogenic structures O, H, and K antigens but the diversity observed for these antigens rendered serotyping unreliable rapid diagnostic tool for this pathotype [17, 107]. However, as recommended in 1987 by World Health Organization, 12 serogroups; O26, O55, O86, O111, O114, O119, O125, O126, O127, O128, O142, and O158 belonged to EPEC pathotype. In addition to six others; O39, O88, O103, O145, O157, and O158 have been classified and belonged to this pathotype although some of these serogroups consist of
EPEC pathotype is defined based on the carriage of LEE locus that mediates the induction of A/E localized lesions [110], a feature that is shared with some STEC strains. However, the inability to produce Shiga toxins or other enterotoxins differentiate EPEC pathotype from EHEC/STEC strains [3, 10]. Additionally, based on the presence or absence of
Since EPEC does not produce Shiga toxin or other enterotoxins, the major feature the pathogenic strains in this pathotype employ is their ability to attach tightly to the host mucosal membrane, destroy microvilli, and induce the formation of lesions (Table 1) [115]. In addition, EPEC carries other genes encoding proteins that have been linked to colonization and adherence to host cells [116, 117].
The defining characteristic of EPEC is the carriage of LEE locus that is essential for inducing A/E lesions, causing localized lesions by attaching closely to the surface of the intestinal epithelial cells. Like some STEC strains, all the EPEC strains carry
The ~80 kb pEAF plasmid that defines tEPEC carries
Although EPEC-related infection could resolve itself or simply by oral rehydration therapy that replenishes the lost fluid, the persistence of this infection may necessitate the use of antibiotics. In this case, especially in adults, the recommended antimicrobial is trimethoprim/sulfamethoxazole, norfloxacin, or ciprofloxacin [124]. However, studies on antibiotic resistance of EPEC strains from different sources and countries have shown high resistance of this pathotype to ampicillin, cefpodoxime, nalidixic acid, trimethoprim, and tetracycline [125, 126]. While resistance to the great majority of these antibiotics is reported to be frequent in tEPEC, trimethoprim resistance is more common in aEPEC strains [127].
In a global study of 185 aEPEC isolates collected from healthy and diarrheal children living in seven sites in sub-Saharan Africa and South Asia, at least 55% of the isolates showed phenotypic resistance to ampicillin, trimethoprim, trimethoprim/sulphamethoxazole, and tetracycline, while streptomycin resistance was reported in 43% of the isolates. Shockingly, more than 50% of the isolates were resistant to three or more of the tested antibiotics [128]. The study also reported point mutations in genes that are associated with resistance to quinolone (
The acquisition of LEE and pEAF has been the defining evolutionary phenomenon for EPEC pathotypes [3, 129]. While tEPEC that carries pEAF plasmid is believed to be less diverse, aEPEC is greatly heterogeneous. The loss of pEAF plasmid in aEPEC and its close relatedness with LEE-positive STEC in serotypes, genetic characteristics, virulence properties, and reservoirs make serotype-based lineage definition unreliable [111, 130]. Based on the conventional MLEE and MLST, EPEC strains belong to six clonal lineages (EPEC1–EPEC6) that were represented among the EPEC strains worldwide [129, 131]. The whole genome-based phylogeny reported nine more EPEC lineages designated as EPEC7-EPEC15 [104, 132]. These phylogenomic EPEC lineages belonged to four
The close relatedness of aEPEC to other pathotypes could play a significant role in the diversity within this pathotype. This EPEC subtype can also include tEPEC that have lost the pEAF plasmid and LEE-positive STEC strains that have lost the Stx encoding bacteriophage during transmission events between hosts, within-host evolution, interaction with the host microbiota, or selective pressure in the environment [104, 130]. This could be a possible explanation why some aEPEC strains would cluster with other pathotypes. Indeed, phylogenomic analyses of 106 Brazilian and 221 global aEPEC genomes showed that isolates were clustered into the previously reported phylogroups for this pathotype and phylogroup D. Additionally, 42.5% of the isolates belonged to the four previously defined EPEC lineages [129, 131], while the remaining isolates were found in EPEC11-EPEC14 phylogenomic lineages, suggesting a gradual and continuous clonal expansion of this pathotype [132]. Of note, dissemination of the phylogenomic lineages of EPEC pathotype is not restricted by geography. Conversely, in a multicentre study involving seven sites in developing countries, EPEC isolates from sub-Saharan countries (The Gambia and Kenya) were clustered into two EPEC lineages (EPEC5 and EPEC10) in phylogroup A [104]. Overall, EPEC represents a pathotype that is still undergoing clonal expansion due to the occurrence of novel phylogenomic lineages with distinct accessory gene content and their pathogenic potential.
Enteroaggregative
EAEC strains are identified using a molecular probe AA that hybridizes with a region of pAA plasmid encoding an ATP binding cassette transporter apparatus which translocates dispersion across the bacterial cell membrane [136]. Isolates that carry the
Although some serotypes including O126:H27, O111:H21, O125, O44:H18 are frequently isolated from EAEC strains, the autoagglutinating phenotype by some EAEC strains complicates the serotyping of this pathotype [3, 138]. In several studies, EAEC strains are often described as nontypeable or as “O?” or O-rough. In a study of EAEC strains from children in Germany, 14 out of 16 isolates that were typeable belonged to different serotypes [139]. Likewise, in a study in the UK, 97 out of 143 EAEC strains that were typeable belonged to more than 40 different O-types [140]. While serotyping is no longer a dependable diagnostic tool for EAEC strains causing diarrheal illness [3, 138], a specific Shiga toxin producing EAEC serotype O104:H4 is associated with a series of outbreaks worldwide [141, 142].
EAEC strains that are implicated in diarrheal illness employ several virulence factors that initiate colonization, promote persistence through adherence to mucosal layers of the intestine, and enterotoxin and cytotoxin secretion (Table 1) [134].
EAEC colonizes the intestinal epithelium of the host using aggregative adhesion fimbriae (AAFs) that also activate the host inflammatory responses and afimbrial adhesins [143]. So far, five AAF variants have been described and are encoded by
EAEC produces enterotoxins and cytotoxins including EAST1 and colonization factors encoded by
The prevalence of the virulence determinants varies with studies and EAEC subtypes [144, 146]. For example, in a study,
EAEC-related diseases such as travelers’ diarrhea where antimicrobial therapy is proposed, fluoroquinolones, azithromycin, and rifaximin are often recommended. In immunocompromised patients that require chemoprophylaxis, fluoroquinolones are also considered [150]. For Shiga toxin producing EAEC O104: H4 related infections, azithromycin which has been shown to inhibit
Although a highly successful treatment rate is achieved with these antibiotics, EAEC strains that are resistant to multiple antibiotics have emerged in different regions [150]. Studies on the resistance of EAEC strains from Southeast Asia, India, Africa, and Latin America with travelers’ diarrhea showed that more than 50% of the isolates were resistant to ampicillin, sulphamethoxazole, and tetracycline [150, 151]. In a similar study in Iran, 78% and 60% of the extended-spectrum beta-lactamase (ESBL) producing EAEC strains carried the transposable
EAEC subtypes are defined based on the presence of virulence plasmid pAA that carries
In a recent study [146], of the 97 EAEC strains analyzed using MLST, 42% were reported to belong to phylogroup B1, while the majority of the few strains that belonged to phylogroup A lack the AAF-associated genes. Although serotype diversity is high in this pathotype, this study also noted that EAEC strains that belonged to phylogroup D were clustered into three serotype-specific lineages (lineage 1–3). All strains in lineage 1 were O166:H15 and belonged to ST349, lineage 2 consisted of serogroups O44, O73, and O17/O77 in combination with either H18 or H34 and ST130, while lineage 3 carried O153:H30 serotype and ST38 [146]. Contrarily, in India, EAEC strains implicated in diarrhea were more prevalent in phylogroup D [158] suggesting that the diversity in the pathotype is not limited by geography. While EAEC subtypes are believed to differ in their virulence determinants content, this hypothesis and comparative phylogenetic analysis of aEAEC and tEAEC are underexplored. Also, large-scale phylogenomic and phylogeographic analyses of this pathotype are scarce. Further studies should focus on this.
Enteroinvasive
At least, 20 serotypes have been assigned to this pathotype [159] among which some of the EIEC-associated O antigens including O28, O112ac, O121, O124, O143, O144, O152, and O167, are identical to O antigens present in
EIEC strains cause infection in humans by their ability to invade the colon mucosa layer with the expression of essential virulence determinants that mediate colonization, adherence, and invasion of the intestinal epithelial cells of the host (Table 1). These genes that are also shared with
The colonization, adherence, and invasion of intestinal epithelial cells by EIEC are mediated by genetic factors encoded by genes on a plasmid, pINV. pINV is a virulence plasmid found in EIEC that encodes the type III secretion system necessary for attachment, invasion of the host cell, and intercellular spread. This plasmid is structurally and functionally similar to those in
Also carried on pINV are genes that encode IcsA, a protein that facilitates the bacterial movement inside the cytoplasm, VirA, a GTPase-activating protein, and RnaG, a small RNA that negatively control the expression of
EIEC strains also carry a plasmid-borne gene,
EIEC-related infection is self-limiting that could be managed with rehydration to replenish the loss electrolyte. Zinc supplementation and nutritional therapy with iron-rich green plantain have also been shown to reduce the severity and the duration of diarrheal illness. However, in rare cases of severe symptoms antimicrobial treatment therapy has been reported to be effective [3, 10]. Since
Like other
EIEC pathotype is diverse and highly specialized due to the carriage of large virulence plasmid, a genetic element that is shared with
A phylogenetic analysis of 32 EIEC strains based on four housekeeping genes (
Insertion sequences were recently reported to contribute to the population structure of EIEC. A recent study [176] on the evolutionary dynamics of
In a comparative pangenome analysis of EIEC with
The EIEC lineages were reported to have distinct phenotypic and genotypic features. Lan et al. [173] reported that EIEC strains belonging to cluster 4 lack mucate fermentation ability, whereas strains in cluster 6 were able to utilize acetate and ferment mucate [173]. Likewise, Hazen et al. [175] identified up to 155 gene clusters that were exclusive in EIEC strains belonging to one phylogroup. Additionally, 12–155 gene clusters were also reported to be lineage-specific in the EIEC pathotype [175]. Protein-encoding genes that are linked to transcriptional regulation, metabolism, and transport, and a colicin were exclusive in EIEC lineage 1, whereas genes that encode membrane protein, the aerobactin siderophore receptor, and hypothetical proteins were exclusive for EIEC lineage 2. Genes encoding several transcriptional regulators and hypothetical proteins were limited to EIEC lineage 3 [175].
Diffusely adherent
DAEC is widespread and associated with diarrhea in both developing and industrialized countries around the world [3]. DAEC strains are associated with watery diarrhea in children under 5 years and can persist resulting in an increase in severity of disease in this age group [177]. DAEC has also been implicated in extraintestinal infections such as UTI, pregnancy complications [3, 177]. It has been speculated that the asymptomatic carriage of DAEC by this age group and adults can lead to chronic inflammatory colon disease such as Crohn’s disease [3, 177]. Meanwhile, there is no universal detection method for this pathotype but based on DNA hybridization of fimbrial encoding
While it is unclear how DAEC is transmitted or its reservoir, the fact that there are asymptomatic carriers could suggest humans as the main reservoir and the fecal-oral route as the primary means of transmission (Figure 2). Information regarding serotypes associated with DAEC is scarce. A study of 112 DAEC isolates from diarrheal and asymptomatic individuals in Brazil reported 45 different serotypes, of which 19 were exclusive in patients with diarrhea [179], whereas in another study [180] the serotypes were nontypeable.
DAEC strains infect the intestinal epithelium of the host by expressing surface-exposed adhesins that mediate colonization and attachment which allow them to resist host clearance mechanisms [177]. There are several virulence factors that mediate this process, and they include fimbria or afimbrial structures, adhesins, and secretion of cytotoxic toxins that promote the invasion of the host cells (Table 1) [177].
DAEC pathotype carries genes that encode Afa/Dr adhesins [3, 177]. Afa/Dr family includes Afa, Dr, and F1845 adhesins that are both afimbrial (such as AfaE-I and AfaE-III), and fimbrial (such as F1845 and Dr) adhesive structures on the bacterial surface encoded by the
The prevalence of the genes constituting the operons that encode the Afa/Dr adhesins have been reported to vary in DAEC pathotype. In a study, the prevalence of the Afa/Dr adhesin family encoding genes
DAEC secretes a class I SPATE toxin that is called secreted autotransporter toxin (Sat), encoded by
Oral rehydration solution therapy is the only recommended treatment for DAEC-related watery diarrhea. However, there are reports of antibiotic resistance in this pathotype. In a study of 112 DAEC strains isolated from children with watery diarrhea in Brazil [179], all DAEC isolates were susceptible to five antibiotics including gentamicin, ofloxacin, and nalidixic acid while 70% were resistant to three or more antibiotics and 50% showed resistance to either ampicillin, co-trimoxazole, streptomycin, sulfonamide, or tetracycline. Additionally, 20% of the strains were resistant to chloramphenicol [179]. A similar observation was noted in a study from Iran where 75–100% of DAEC strains from pediatric diarrhea were resistant to ampicillin, cefotaxime, and trimethoprim-sulfamethoxazole [182].
DAEC is a heterogeneous group that has also been implicated in extraintestinal infections such as UTI. Despite its implication in diarrhea in children, studies on its population structure are limited. The few studies available on the phylogenetic analysis of DAEC strains using MLEE reported that they are distributed among all of the phylogroups [47, 156, 189]. Conversely, a study of 31 DAEC strains from diarrhea and asymptomatic carriers in Peru reported that 87% of the isolates belonged to phylogroup D [190]. A large-scale genomic analysis of DAEC strains would be important to understand the population structure, determine dissemination and transmission dynamics of genetic lineages of this pathotype, as well as identify novel virulence determinants and other genetic factors that contribute to its pathoadaptation in the intestinal epithelium.
Much progress has been made on the biology and the genomic epidemiology of diarrheagenic
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',metaTitle:"Publication Agreement - Chapters",metaDescription:"IN TECH aims to guarantee that original material is published while at the same time giving significant freedom to our authors. For that matter, we uphold a flexible copyright policy meaning that there is no transfer of copyright to the publisher and authors retain exclusive copyright to their work.\n\nWhen submitting a manuscript the Corresponding Author is required to accept the terms and conditions set forth in our Publication Agreement as follows:",metaKeywords:null,canonicalURL:"/page/publication-agreement-chapters",contentRaw:'[{"type":"htmlEditorComponent","content":"The Corresponding Author (acting on behalf of all Authors) and INTECHOPEN LIMITED, incorporated and registered in England and Wales with company number 11086078 and a registered office at 5 Princes Gate Court, London, United Kingdom, SW7 2QJ conclude the following Agreement regarding the publication of a Book Chapter:
\\n\\n1. DEFINITIONS
\\n\\nCorresponding Author: The Author of the Chapter who serves as a Signatory to this Agreement. The Corresponding Author acts on behalf of any other Co-Author.
\\n\\nCo-Author: All other Authors of the Chapter besides the Corresponding Author.
\\n\\nIntechOpen: IntechOpen Ltd., the Publisher of the Book.
\\n\\nBook: The publication as a collection of chapters compiled by IntechOpen including the Chapter. Chapter: The original literary work created by Corresponding Author and any Co-Author that is the subject of this Agreement.
\\n\\n2. CORRESPONDING AUTHOR'S GRANT OF RIGHTS
\\n\\n2.1 Subject to the following Article, the Corresponding Author grants and shall ensure that each Co-Author grants, to IntechOpen, during the full term of copyright and any extensions or renewals of that term the following:
\\n\\nThe aforementioned licenses shall survive the expiry or termination of this Agreement for any reason.
\\n\\n2.2 The Corresponding Author (on their own behalf and on behalf of any Co-Author) reserves the following rights to the Chapter but agrees not to exercise them in such a way as to adversely affect IntechOpen's ability to utilize the full benefit of this Publication Agreement: (i) reprographic rights worldwide, other than those which subsist in the typographical arrangement of the Chapter as published by IntechOpen; and (ii) public lending rights arising under the Public Lending Right Act 1979, as amended from time to time, and any similar rights arising in any part of the world.
\\n\\nThe Corresponding Author confirms that they (and any Co-Author) are and will remain a member of any applicable licensing and collecting society and any successor to that body responsible for administering royalties for the reprographic reproduction of copyright works.
\\n\\nSubject to the license granted above, copyright in the Chapter and all versions of it created during IntechOpen's editing process (including the published version) is retained by the Corresponding Author and any Co-Author.
\\n\\nSubject to the license granted above, the Corresponding Author and any Co-Author retains patent, trademark and other intellectual property rights to the Chapter.
\\n\\n2.3 All rights granted to IntechOpen in this Article are assignable, sublicensable or otherwise transferrable to third parties without the Corresponding Author's or any Co-Author’s specific approval.
\\n\\n2.4 The Corresponding Author (on their own behalf and on behalf of each Co-Author) will not assert any rights under the Copyright, Designs and Patents Act 1988 to object to derogatory treatment of the Chapter as a consequence of IntechOpen's changes to the Chapter arising from translation of it, corrections and edits for house style, removal of problematic material and other reasonable edits.
\\n\\n3. CORRESPONDING AUTHOR'S DUTIES
\\n\\n3.1 When distributing or re-publishing the Chapter, the Corresponding Author agrees to credit the Book in which the Chapter has been published as the source of first publication, as well as IntechOpen. The Corresponding Author warrants that each Co-Author will also credit the Book in which the Chapter has been published as the source of first publication, as well as IntechOpen, when they are distributing or re-publishing the Chapter.
\\n\\n3.2 When submitting the Chapter, the Corresponding Author agrees to:
\\n\\nThe Corresponding Author will be held responsible for the payment of the Open Access Publishing Fees.
\\n\\nAll payments shall be due 30 days from the date of the issued invoice. The Corresponding Author or the payer on the Corresponding Author's and Co-Authors' behalf will bear all banking and similar charges incurred.
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\\n\\nThe Corresponding Author shall obtain written informed consent for publication from people who might recognize themselves or be identified by others (e.g. from case reports or photographs).
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\\n\\n4. CORRESPONDING AUTHOR'S WARRANTY
\\n\\n4.1 The Corresponding Author represents and warrants that the Chapter does not and will not breach any applicable law or the rights of any third party and, specifically, that the Chapter contains no matter that is defamatory or that infringes any literary or proprietary rights, intellectual property rights, or any rights of privacy. The Corresponding Author warrants and represents that: (i) the Chapter is the original work of themselves and any Co-Author and is not copied wholly or substantially from any other work or material or any other source; (ii) the Chapter has not been formally published in any other peer-reviewed journal or in a book or edited collection, and is not under consideration for any such publication; (iii) they themselves and any Co-Author are qualifying persons under section 154 of the Copyright, Designs and Patents Act 1988; (iv) they themselves and any Co-Author have not assigned and will not during the term of this Publication Agreement purport to assign any of the rights granted to IntechOpen under this Publication Agreement; and (v) the rights granted by this Publication Agreement are free from any security interest, option, mortgage, charge or lien.
\\n\\nThe Corresponding Author also warrants and represents that: (i) they have the full power to enter into this Publication Agreement on their own behalf and on behalf of each Co-Author; and (ii) they have the necessary rights and/or title in and to the Chapter to grant IntechOpen, on behalf of themselves and any Co-Author, the rights and licenses expressed to be granted in this Publication Agreement. If the Chapter was prepared jointly by the Corresponding Author and any Co-Author, the Corresponding Author warrants and represents that: (i) each Co-Author agrees to the submission, license and publication of the Chapter on the terms of this Publication Agreement; and (ii) they have the authority to enter into this Publication Agreement on behalf of and bind each Co-Author. The Corresponding Author shall: (i) ensure each Co-Author complies with all relevant provisions of this Publication Agreement, including those relating to confidentiality, performance and standards, as if a party to this Publication Agreement; and (ii) remain primarily liable for all acts and/or omissions of each such Co-Author.
\\n\\nThe Corresponding Author agrees to indemnify and hold IntechOpen harmless against all liabilities, costs, expenses, damages and losses and all reasonable legal costs and expenses suffered or incurred by IntechOpen arising out of or in connection with any breach of the aforementioned representations and warranties. This indemnity shall not cover IntechOpen to the extent that a claim under it results from IntechOpen's negligence or willful misconduct.
\\n\\n4.2 Nothing in this Publication Agreement shall have the effect of excluding or limiting any liability for death or personal injury caused by negligence or any other liability that cannot be excluded or limited by applicable law.
\\n\\n5. TERMINATION
\\n\\n5.1 IntechOpen has a right to terminate this Publication Agreement for quality, program, technical or other reasons with immediate effect, including without limitation (i) if the Corresponding Author or any Co-Author commits a material breach of this Publication Agreement; (ii) if the Corresponding Author or any Co-Author (being an individual) is the subject of a bankruptcy petition, application or order; or (iii) if the Corresponding Author or any Co-Author (being a company) commences negotiations with all or any class of its creditors with a view to rescheduling any of its debts, or makes a proposal for or enters into any compromise or arrangement with any of its creditors.
\\n\\nIn case of termination, IntechOpen will notify the Corresponding Author, in writing, of the decision.
\\n\\n6. INTECHOPEN’S DUTIES AND RIGHTS
\\n\\n6.1 Unless prevented from doing so by events outside its reasonable control, IntechOpen, in its discretion, agrees to publish the Chapter attributing it to the Corresponding Author and any Co-Author.
\\n\\n6.2 IntechOpen has the right to use the Corresponding Author’s and any Co-Author’s names and likeness in connection with scientific dissemination, retrieval, archiving, web hosting and promotion and marketing of the Chapter and has the right to contact the Corresponding Author and any Co-Author until the Chapter is publicly available on any platform owned and/or operated by IntechOpen.
\\n\\n6.3 IntechOpen is granted the authority to enforce the rights from this Publication Agreement, on behalf of the Corresponding Author and any Co-Author, against third parties (for example in cases of plagiarism or copyright infringements). In respect of any such infringement or suspected infringement of the copyright in the Chapter, IntechOpen shall have absolute discretion in addressing any such infringement which is likely to affect IntechOpen's rights under this Publication Agreement, including issuing and conducting proceedings against the suspected infringer.
\\n\\n7. MISCELLANEOUS
\\n\\n7.1 Further Assurance: The Corresponding Author shall and will ensure that any relevant third party (including any Co-Author) shall, execute and deliver whatever further documents or deeds and perform such acts as IntechOpen reasonably requires from time to time for the purpose of giving IntechOpen the full benefit of the provisions of this Publication Agreement.
\\n\\n7.2 Third Party Rights: A person who is not a party to this Publication Agreement may not enforce any of its provisions under the Contracts (Rights of Third Parties) Act 1999.
\\n\\n7.3 Entire Agreement: This Publication Agreement constitutes the entire agreement between the parties in relation to its subject matter. It replaces and extinguishes all prior agreements, draft agreements, arrangements, collateral warranties, collateral contracts, statements, assurances, representations and undertakings of any nature made by or on behalf of the parties, whether oral or written, in relation to that subject matter. Each party acknowledges that in entering into this Publication Agreement it has not relied upon any oral or written statements, collateral or other warranties, assurances, representations or undertakings which were made by or on behalf of the other party in relation to the subject matter of this Publication Agreement at any time before its signature (together "Pre-Contractual Statements"), other than those which are set out in this Publication Agreement. Each party hereby waives all rights and remedies which might otherwise be available to it in relation to such Pre-Contractual Statements. Nothing in this clause shall exclude or restrict the liability of either party arising out of its pre-contract fraudulent misrepresentation or fraudulent concealment.
\\n\\n7.4 Waiver: No failure or delay by a party to exercise any right or remedy provided under this Publication Agreement or by law shall constitute a waiver of that or any other right or remedy, nor shall it preclude or restrict the further exercise of that or any other right or remedy. No single or partial exercise of such right or remedy shall preclude or restrict the further exercise of that or any other right or remedy.
\\n\\n7.5 Variation: No variation of this Publication Agreement shall be effective unless it is in writing and signed by the parties (or their duly authorized representatives).
\\n\\n7.6 Severance: If any provision or part-provision of this Publication Agreement is or becomes invalid, illegal or unenforceable, it shall be deemed modified to the minimum extent necessary to make it valid, legal and enforceable. If such modification is not possible, the relevant provision or part-provision shall be deemed deleted.
\\n\\nAny modification to or deletion of a provision or part-provision under this clause shall not affect the validity and enforceability of the rest of this Publication Agreement.
\\n\\n7.7 No partnership: Nothing in this Publication Agreement is intended to, or shall be deemed to, establish or create any partnership or joint venture or the relationship of principal and agent or employer and employee between IntechOpen and the Corresponding Author or any Co-Author, nor authorize any party to make or enter into any commitments for or on behalf of any other party.
\\n\\n7.8 Governing law: This Publication Agreement and any dispute or claim (including non-contractual disputes or claims) arising out of or in connection with it or its subject matter or formation shall be governed by and construed in accordance with the law of England and Wales. The parties submit to the exclusive jurisdiction of the English courts to settle any dispute or claim arising out of or in connection with this Publication Agreement (including any non-contractual disputes or claims).
\\n\\nLast updated: 2020-11-27
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The Corresponding Author (acting on behalf of all Authors) and INTECHOPEN LIMITED, incorporated and registered in England and Wales with company number 11086078 and a registered office at 5 Princes Gate Court, London, United Kingdom, SW7 2QJ conclude the following Agreement regarding the publication of a Book Chapter:
\n\n1. DEFINITIONS
\n\nCorresponding Author: The Author of the Chapter who serves as a Signatory to this Agreement. The Corresponding Author acts on behalf of any other Co-Author.
\n\nCo-Author: All other Authors of the Chapter besides the Corresponding Author.
\n\nIntechOpen: IntechOpen Ltd., the Publisher of the Book.
\n\nBook: The publication as a collection of chapters compiled by IntechOpen including the Chapter. Chapter: The original literary work created by Corresponding Author and any Co-Author that is the subject of this Agreement.
\n\n2. CORRESPONDING AUTHOR'S GRANT OF RIGHTS
\n\n2.1 Subject to the following Article, the Corresponding Author grants and shall ensure that each Co-Author grants, to IntechOpen, during the full term of copyright and any extensions or renewals of that term the following:
\n\nThe aforementioned licenses shall survive the expiry or termination of this Agreement for any reason.
\n\n2.2 The Corresponding Author (on their own behalf and on behalf of any Co-Author) reserves the following rights to the Chapter but agrees not to exercise them in such a way as to adversely affect IntechOpen's ability to utilize the full benefit of this Publication Agreement: (i) reprographic rights worldwide, other than those which subsist in the typographical arrangement of the Chapter as published by IntechOpen; and (ii) public lending rights arising under the Public Lending Right Act 1979, as amended from time to time, and any similar rights arising in any part of the world.
\n\nThe Corresponding Author confirms that they (and any Co-Author) are and will remain a member of any applicable licensing and collecting society and any successor to that body responsible for administering royalties for the reprographic reproduction of copyright works.
\n\nSubject to the license granted above, copyright in the Chapter and all versions of it created during IntechOpen's editing process (including the published version) is retained by the Corresponding Author and any Co-Author.
\n\nSubject to the license granted above, the Corresponding Author and any Co-Author retains patent, trademark and other intellectual property rights to the Chapter.
\n\n2.3 All rights granted to IntechOpen in this Article are assignable, sublicensable or otherwise transferrable to third parties without the Corresponding Author's or any Co-Author’s specific approval.
\n\n2.4 The Corresponding Author (on their own behalf and on behalf of each Co-Author) will not assert any rights under the Copyright, Designs and Patents Act 1988 to object to derogatory treatment of the Chapter as a consequence of IntechOpen's changes to the Chapter arising from translation of it, corrections and edits for house style, removal of problematic material and other reasonable edits.
\n\n3. CORRESPONDING AUTHOR'S DUTIES
\n\n3.1 When distributing or re-publishing the Chapter, the Corresponding Author agrees to credit the Book in which the Chapter has been published as the source of first publication, as well as IntechOpen. The Corresponding Author warrants that each Co-Author will also credit the Book in which the Chapter has been published as the source of first publication, as well as IntechOpen, when they are distributing or re-publishing the Chapter.
\n\n3.2 When submitting the Chapter, the Corresponding Author agrees to:
\n\nThe Corresponding Author will be held responsible for the payment of the Open Access Publishing Fees.
\n\nAll payments shall be due 30 days from the date of the issued invoice. The Corresponding Author or the payer on the Corresponding Author's and Co-Authors' behalf will bear all banking and similar charges incurred.
\n\n3.3 The Corresponding Author shall obtain in writing all consents necessary for the reproduction of any material in which a third-party right exists, including quotations, photographs and illustrations, in all editions of the Chapter worldwide for the full term of the above licenses, and shall provide to IntechOpen upon request the original copies of such consents for inspection (at IntechOpen's option) or photocopies of such consents.
\n\nThe Corresponding Author shall obtain written informed consent for publication from people who might recognize themselves or be identified by others (e.g. from case reports or photographs).
\n\n3.4 The Corresponding Author and any Co-Author shall respect confidentiality rights during and after the termination of this Agreement. The information contained in all correspondence and documents as part of the publishing activity between IntechOpen and the Corresponding Author and any Co-Author are confidential and are intended only for the recipient. The contents may not be disclosed publicly and are not intended for unauthorized use or distribution. Any use, disclosure, copying, or distribution is prohibited and may be unlawful.
\n\n4. CORRESPONDING AUTHOR'S WARRANTY
\n\n4.1 The Corresponding Author represents and warrants that the Chapter does not and will not breach any applicable law or the rights of any third party and, specifically, that the Chapter contains no matter that is defamatory or that infringes any literary or proprietary rights, intellectual property rights, or any rights of privacy. The Corresponding Author warrants and represents that: (i) the Chapter is the original work of themselves and any Co-Author and is not copied wholly or substantially from any other work or material or any other source; (ii) the Chapter has not been formally published in any other peer-reviewed journal or in a book or edited collection, and is not under consideration for any such publication; (iii) they themselves and any Co-Author are qualifying persons under section 154 of the Copyright, Designs and Patents Act 1988; (iv) they themselves and any Co-Author have not assigned and will not during the term of this Publication Agreement purport to assign any of the rights granted to IntechOpen under this Publication Agreement; and (v) the rights granted by this Publication Agreement are free from any security interest, option, mortgage, charge or lien.
\n\nThe Corresponding Author also warrants and represents that: (i) they have the full power to enter into this Publication Agreement on their own behalf and on behalf of each Co-Author; and (ii) they have the necessary rights and/or title in and to the Chapter to grant IntechOpen, on behalf of themselves and any Co-Author, the rights and licenses expressed to be granted in this Publication Agreement. If the Chapter was prepared jointly by the Corresponding Author and any Co-Author, the Corresponding Author warrants and represents that: (i) each Co-Author agrees to the submission, license and publication of the Chapter on the terms of this Publication Agreement; and (ii) they have the authority to enter into this Publication Agreement on behalf of and bind each Co-Author. The Corresponding Author shall: (i) ensure each Co-Author complies with all relevant provisions of this Publication Agreement, including those relating to confidentiality, performance and standards, as if a party to this Publication Agreement; and (ii) remain primarily liable for all acts and/or omissions of each such Co-Author.
\n\nThe Corresponding Author agrees to indemnify and hold IntechOpen harmless against all liabilities, costs, expenses, damages and losses and all reasonable legal costs and expenses suffered or incurred by IntechOpen arising out of or in connection with any breach of the aforementioned representations and warranties. This indemnity shall not cover IntechOpen to the extent that a claim under it results from IntechOpen's negligence or willful misconduct.
\n\n4.2 Nothing in this Publication Agreement shall have the effect of excluding or limiting any liability for death or personal injury caused by negligence or any other liability that cannot be excluded or limited by applicable law.
\n\n5. TERMINATION
\n\n5.1 IntechOpen has a right to terminate this Publication Agreement for quality, program, technical or other reasons with immediate effect, including without limitation (i) if the Corresponding Author or any Co-Author commits a material breach of this Publication Agreement; (ii) if the Corresponding Author or any Co-Author (being an individual) is the subject of a bankruptcy petition, application or order; or (iii) if the Corresponding Author or any Co-Author (being a company) commences negotiations with all or any class of its creditors with a view to rescheduling any of its debts, or makes a proposal for or enters into any compromise or arrangement with any of its creditors.
\n\nIn case of termination, IntechOpen will notify the Corresponding Author, in writing, of the decision.
\n\n6. INTECHOPEN’S DUTIES AND RIGHTS
\n\n6.1 Unless prevented from doing so by events outside its reasonable control, IntechOpen, in its discretion, agrees to publish the Chapter attributing it to the Corresponding Author and any Co-Author.
\n\n6.2 IntechOpen has the right to use the Corresponding Author’s and any Co-Author’s names and likeness in connection with scientific dissemination, retrieval, archiving, web hosting and promotion and marketing of the Chapter and has the right to contact the Corresponding Author and any Co-Author until the Chapter is publicly available on any platform owned and/or operated by IntechOpen.
\n\n6.3 IntechOpen is granted the authority to enforce the rights from this Publication Agreement, on behalf of the Corresponding Author and any Co-Author, against third parties (for example in cases of plagiarism or copyright infringements). In respect of any such infringement or suspected infringement of the copyright in the Chapter, IntechOpen shall have absolute discretion in addressing any such infringement which is likely to affect IntechOpen's rights under this Publication Agreement, including issuing and conducting proceedings against the suspected infringer.
\n\n7. MISCELLANEOUS
\n\n7.1 Further Assurance: The Corresponding Author shall and will ensure that any relevant third party (including any Co-Author) shall, execute and deliver whatever further documents or deeds and perform such acts as IntechOpen reasonably requires from time to time for the purpose of giving IntechOpen the full benefit of the provisions of this Publication Agreement.
\n\n7.2 Third Party Rights: A person who is not a party to this Publication Agreement may not enforce any of its provisions under the Contracts (Rights of Third Parties) Act 1999.
\n\n7.3 Entire Agreement: This Publication Agreement constitutes the entire agreement between the parties in relation to its subject matter. It replaces and extinguishes all prior agreements, draft agreements, arrangements, collateral warranties, collateral contracts, statements, assurances, representations and undertakings of any nature made by or on behalf of the parties, whether oral or written, in relation to that subject matter. Each party acknowledges that in entering into this Publication Agreement it has not relied upon any oral or written statements, collateral or other warranties, assurances, representations or undertakings which were made by or on behalf of the other party in relation to the subject matter of this Publication Agreement at any time before its signature (together "Pre-Contractual Statements"), other than those which are set out in this Publication Agreement. Each party hereby waives all rights and remedies which might otherwise be available to it in relation to such Pre-Contractual Statements. Nothing in this clause shall exclude or restrict the liability of either party arising out of its pre-contract fraudulent misrepresentation or fraudulent concealment.
\n\n7.4 Waiver: No failure or delay by a party to exercise any right or remedy provided under this Publication Agreement or by law shall constitute a waiver of that or any other right or remedy, nor shall it preclude or restrict the further exercise of that or any other right or remedy. No single or partial exercise of such right or remedy shall preclude or restrict the further exercise of that or any other right or remedy.
\n\n7.5 Variation: No variation of this Publication Agreement shall be effective unless it is in writing and signed by the parties (or their duly authorized representatives).
\n\n7.6 Severance: If any provision or part-provision of this Publication Agreement is or becomes invalid, illegal or unenforceable, it shall be deemed modified to the minimum extent necessary to make it valid, legal and enforceable. If such modification is not possible, the relevant provision or part-provision shall be deemed deleted.
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\n\nLast updated: 2020-11-27
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Implications for parent education and prevention of risks for early and excessive exposure to digital technologies are discussed.",book:{id:"9043",slug:"parenting-studies-by-an-ecocultural-and-transactional-perspective",title:"Parenting",fullTitle:"Parenting - Studies by an Ecocultural and Transactional Perspective"},signatures:"Loredana Benedetto and Massimo Ingrassia",authors:[{id:"193200",title:"Prof.",name:"Loredana",middleName:null,surname:"Benedetto",slug:"loredana-benedetto",fullName:"Loredana Benedetto"},{id:"193901",title:"Prof.",name:"Massimo",middleName:null,surname:"Ingrassia",slug:"massimo-ingrassia",fullName:"Massimo Ingrassia"}]},{id:"45773",doi:"10.5772/57003",title:"Cultural Variations in Parenting Styles in the Majority World Evidences from Nigeria and Cameroon",slug:"cultural-variations-in-parenting-styles-in-the-majority-world-evidences-from-nigeria-and-cameroon",totalDownloads:5078,totalCrossrefCites:0,totalDimensionsCites:9,abstract:null,book:{id:"3440",slug:"parenting-in-south-american-and-african-contexts",title:"Parenting in South American and African Contexts",fullTitle:"Parenting in South American and African Contexts"},signatures:"Esther F. Akinsola",authors:[{id:"160225",title:"Dr.",name:"Esther",middleName:"Foluke",surname:"Akinsola",slug:"esther-akinsola",fullName:"Esther Akinsola"}]},{id:"45934",doi:"10.5772/57083",title:"Brazilian Mothers’ Cultural Models: Socialization for Autonomy and Relatedness",slug:"brazilian-mothers-cultural-models-socialization-for-autonomy-and-relatedness",totalDownloads:1687,totalCrossrefCites:2,totalDimensionsCites:8,abstract:null,book:{id:"3440",slug:"parenting-in-south-american-and-african-contexts",title:"Parenting in South American and African Contexts",fullTitle:"Parenting in South American and African Contexts"},signatures:"Maria Lucia Seidl-de-Moura, Rafael Vera Cruz de Carvalho and\nMauro Luís Vieira",authors:[{id:"108479",title:"Dr.",name:"Maria Lucia",middleName:null,surname:"Seidl-De-Moura",slug:"maria-lucia-seidl-de-moura",fullName:"Maria Lucia Seidl-De-Moura"}]},{id:"45939",doi:"10.5772/57242",title:"Child-Rearing Practices of Brazilian Mothers and Fathers: Predictors and Impact on Child Development",slug:"child-rearing-practices-of-brazilian-mothers-and-fathers-predictors-and-impact-on-child-development",totalDownloads:2977,totalCrossrefCites:1,totalDimensionsCites:4,abstract:null,book:{id:"3440",slug:"parenting-in-south-american-and-african-contexts",title:"Parenting in South American and African Contexts",fullTitle:"Parenting in South American and African Contexts"},signatures:"Cesar Augusto Piccinini, Patricia Alvarenga and Angela Helena\nMarin",authors:[{id:"161058",title:"Dr.",name:"Cesar",middleName:null,surname:"Piccinini",slug:"cesar-piccinini",fullName:"Cesar Piccinini"}]}],mostDownloadedChaptersLast30Days:[{id:"72249",title:"Digital Parenting: Raising and Protecting Children in Media World",slug:"digital-parenting-raising-and-protecting-children-in-media-world",totalDownloads:1577,totalCrossrefCites:4,totalDimensionsCites:10,abstract:"Digital media have quickly changed ways in which parents and children communicate, enjoy themselves, acquire information, and solve problems daily (both in ordinary and exceptional circumstances such as COVID-19 home confinement). Very young children are regular users of smartphones and tablet, so their early digital engagement poses new challenges to parent-child relationships and parental role. First, the chapter introduces the “digital parenting” construct, moving through the literature from “traditional” parenting styles to more recent studies on “parental mediation,” that is, the different behaviors parents adopt to regulate children’s engagement with the Internet and digital media. Second, the chapter reviews empirical researches on different parental mediation practices (active or restrictive behaviors) and how they are adjusted according to the child’s characteristics (age, digital competences, etc.) or parent’s media competence and beliefs. Finally, from a bidirectional perspective of parent-child relationships, the chapter discusses the role of youths’ social involvement, communication, self-disclosure, and digital skills on parent’s beliefs and practices. Implications for parent education and prevention of risks for early and excessive exposure to digital technologies are discussed.",book:{id:"9043",slug:"parenting-studies-by-an-ecocultural-and-transactional-perspective",title:"Parenting",fullTitle:"Parenting - Studies by an Ecocultural and Transactional Perspective"},signatures:"Loredana Benedetto and Massimo Ingrassia",authors:[{id:"193200",title:"Prof.",name:"Loredana",middleName:null,surname:"Benedetto",slug:"loredana-benedetto",fullName:"Loredana Benedetto"},{id:"193901",title:"Prof.",name:"Massimo",middleName:null,surname:"Ingrassia",slug:"massimo-ingrassia",fullName:"Massimo Ingrassia"}]},{id:"72823",title:"Helicopter Parenting and Adolescent Development: From the Perspective of Mental Health",slug:"helicopter-parenting-and-adolescent-development-from-the-perspective-of-mental-health",totalDownloads:951,totalCrossrefCites:1,totalDimensionsCites:2,abstract:"Helicopter parenting is a unique form of parenting style that is generally described as highly intensive and highly involved with the children. A particular parenting style influences all phases of development and life style of adolescent. Helicopter parents overly protect their children from the difficulties by setting some set of instructions without consideration of the uniqueness of their children. Recent literature has got huge attention on this parenting style and debating the pros and cons on the development of child. Higher life satisfaction and better psychological wellbeing have been found in the children of highly intrusive parents. When there are positive effects of helicopter parenting, there are negative outcome and impacts that have also been studied. The difficulties in emotional regulation, academic productivity, and social skills among children raised by helicopter parenting have been reported in the literature. Low self-efficacy, lack of trust on peers, and alienation from peers have also been associated with helicopter parenting. The chapter highlights the associated aspects of childhood and adolescence, raised by helicopter parenting. As parents have their own concern about raising their children in certain manner, it is important to understand the underlying mechanism of parenting style. Therefore, this chapter also describes the theoretical framework. The associated mental health issues and supportive psychological intervention to be also discussed.",book:{id:"9043",slug:"parenting-studies-by-an-ecocultural-and-transactional-perspective",title:"Parenting",fullTitle:"Parenting - Studies by an Ecocultural and Transactional Perspective"},signatures:"Deepika Srivastav and M.N. Lal Mathur",authors:[{id:"320545",title:"Ph.D.",name:"Deepika",middleName:null,surname:"Srivastav",slug:"deepika-srivastav",fullName:"Deepika Srivastav"},{id:"322605",title:"Dr.",name:"M.N.Lal",middleName:null,surname:"Mathur",slug:"m.n.lal-mathur",fullName:"M.N.Lal Mathur"}]},{id:"45760",title:"Parenting and Culture – Evidence from Some African Communities",slug:"parenting-and-culture-evidence-from-some-african-communities",totalDownloads:9583,totalCrossrefCites:10,totalDimensionsCites:25,abstract:null,book:{id:"3440",slug:"parenting-in-south-american-and-african-contexts",title:"Parenting in South American and African Contexts",fullTitle:"Parenting in South American and African Contexts"},signatures:"Patricia Mawusi Amos",authors:[{id:"162496",title:"Mrs.",name:"Patricia",middleName:"Mawusi",surname:"Amos",slug:"patricia-amos",fullName:"Patricia Amos"}]},{id:"72914",title:"Parent-Adolescent Relationship and the Impact of Substance Dependency within the Trajectory of Adolescent Substance Use Disorder",slug:"parent-adolescent-relationship-and-the-impact-of-substance-dependency-within-the-trajectory-of-adole",totalDownloads:645,totalCrossrefCites:0,totalDimensionsCites:0,abstract:"Adolescents strive for freedom and autonomy; thus, communication with their parents needs to be enhanced. Building solid healthy relationships at this stage of their lives is of utmost importance to help them cope with the changes and challenges they are experiencing. The purpose of this chapter is to explore the parent-adolescent relationship in the substance dependency field. The focus is on the relationship between parents and their adolescents who have a substance use disorder. Parenting adolescents poses its own set of challenges, making it difficult to build and maintain healthy parent-adolescent relationships. We argue that although adolescent substance use disorder has been extensively researched, the relationship between parents and adolescents with substance use disorder has surprisingly not received the same attention. It is this gap that this chapter seeks to address. With this in mind, the ecological systems theory was employed here to shed light on the importance and significance of developing healthy parent-adolescent relationships. The findings show that the parent-adolescent relationship primarily informs the daily living of both the parents and the adolescents. The parent-adolescent relationship is therefore very important as it represents whole-family functioning.",book:{id:"9043",slug:"parenting-studies-by-an-ecocultural-and-transactional-perspective",title:"Parenting",fullTitle:"Parenting - Studies by an Ecocultural and Transactional Perspective"},signatures:"Faith Mathibela and Rebecca Mmamoagi Skhosana",authors:[{id:"317920",title:"Mrs.",name:"Faith",middleName:null,surname:"Mathibela",slug:"faith-mathibela",fullName:"Faith Mathibela"}]},{id:"56390",title:"Introductory Chapter: Sociology of Knowledge and Epistemological Paradox of Globalization",slug:"introductory-chapter-sociology-of-knowledge-and-epistemological-paradox-of-globalization",totalDownloads:1823,totalCrossrefCites:0,totalDimensionsCites:0,abstract:null,book:{id:"5924",slug:"epistemology-and-transformation-of-knowledge-in-global-age",title:"Epistemology and Transformation of Knowledge in Global Age",fullTitle:"Epistemology and Transformation of Knowledge in Global Age"},signatures:"Zlatan Delić",authors:[{id:"31746",title:"Dr.",name:"Zlatan",middleName:null,surname:"Delic",slug:"zlatan-delic",fullName:"Zlatan Delic"}]}],onlineFirstChaptersFilter:{topicId:"276",limit:6,offset:0},onlineFirstChaptersCollection:[],onlineFirstChaptersTotal:0},preDownload:{success:null,errors:{}},subscriptionForm:{success:null,errors:{}},aboutIntechopen:{},privacyPolicy:{},peerReviewing:{},howOpenAccessPublishingWithIntechopenWorks:{},sponsorshipBooks:{sponsorshipBooks:[],offset:0,limit:8,total:null},allSeries:{pteSeriesList:[{id:"14",title:"Artificial Intelligence",numberOfPublishedBooks:9,numberOfPublishedChapters:89,numberOfOpenTopics:6,numberOfUpcomingTopics:0,issn:"2633-1403",doi:"10.5772/intechopen.79920",isOpenForSubmission:!0},{id:"7",title:"Biomedical Engineering",numberOfPublishedBooks:12,numberOfPublishedChapters:104,numberOfOpenTopics:3,numberOfUpcomingTopics:0,issn:"2631-5343",doi:"10.5772/intechopen.71985",isOpenForSubmission:!0}],lsSeriesList:[{id:"11",title:"Biochemistry",numberOfPublishedBooks:31,numberOfPublishedChapters:314,numberOfOpenTopics:4,numberOfUpcomingTopics:0,issn:"2632-0983",doi:"10.5772/intechopen.72877",isOpenForSubmission:!0},{id:"25",title:"Environmental Sciences",numberOfPublishedBooks:1,numberOfPublishedChapters:11,numberOfOpenTopics:4,numberOfUpcomingTopics:0,issn:"2754-6713",doi:"10.5772/intechopen.100362",isOpenForSubmission:!0},{id:"10",title:"Physiology",numberOfPublishedBooks:11,numberOfPublishedChapters:141,numberOfOpenTopics:4,numberOfUpcomingTopics:0,issn:"2631-8261",doi:"10.5772/intechopen.72796",isOpenForSubmission:!0}],hsSeriesList:[{id:"3",title:"Dentistry",numberOfPublishedBooks:8,numberOfPublishedChapters:129,numberOfOpenTopics:2,numberOfUpcomingTopics:0,issn:"2631-6218",doi:"10.5772/intechopen.71199",isOpenForSubmission:!0},{id:"6",title:"Infectious Diseases",numberOfPublishedBooks:13,numberOfPublishedChapters:113,numberOfOpenTopics:3,numberOfUpcomingTopics:1,issn:"2631-6188",doi:"10.5772/intechopen.71852",isOpenForSubmission:!0},{id:"13",title:"Veterinary Medicine and Science",numberOfPublishedBooks:11,numberOfPublishedChapters:105,numberOfOpenTopics:3,numberOfUpcomingTopics:0,issn:"2632-0517",doi:"10.5772/intechopen.73681",isOpenForSubmission:!0}],sshSeriesList:[{id:"22",title:"Business, Management and Economics",numberOfPublishedBooks:1,numberOfPublishedChapters:19,numberOfOpenTopics:2,numberOfUpcomingTopics:1,issn:"2753-894X",doi:"10.5772/intechopen.100359",isOpenForSubmission:!0},{id:"23",title:"Education and Human Development",numberOfPublishedBooks:0,numberOfPublishedChapters:5,numberOfOpenTopics:1,numberOfUpcomingTopics:1,issn:null,doi:"10.5772/intechopen.100360",isOpenForSubmission:!0},{id:"24",title:"Sustainable Development",numberOfPublishedBooks:0,numberOfPublishedChapters:14,numberOfOpenTopics:5,numberOfUpcomingTopics:0,issn:null,doi:"10.5772/intechopen.100361",isOpenForSubmission:!0}],testimonialsList:[{id:"6",text:"It is great to work with the IntechOpen to produce a worthwhile collection of research that also becomes a great educational resource and guide for future research endeavors.",author:{id:"259298",name:"Edward",surname:"Narayan",institutionString:null,profilePictureURL:"https://mts.intechopen.com/storage/users/259298/images/system/259298.jpeg",slug:"edward-narayan",institution:{id:"3",name:"University of Queensland",country:{id:null,name:"Australia"}}}},{id:"13",text:"The collaboration with and support of the technical staff of IntechOpen is fantastic. The whole process of submitting an article and editing of the submitted article goes extremely smooth and fast, the number of reads and downloads of chapters is high, and the contributions are also frequently cited.",author:{id:"55578",name:"Antonio",surname:"Jurado-Navas",institutionString:null,profilePictureURL:"https://s3.us-east-1.amazonaws.com/intech-files/0030O00002bRisIQAS/Profile_Picture_1626166543950",slug:"antonio-jurado-navas",institution:{id:"720",name:"University of Malaga",country:{id:null,name:"Spain"}}}}]},series:{item:{id:"14",title:"Artificial Intelligence",doi:"10.5772/intechopen.79920",issn:"2633-1403",scope:"Artificial Intelligence (AI) is a rapidly developing multidisciplinary research area that aims to solve increasingly complex problems. In today's highly integrated world, AI promises to become a robust and powerful means for obtaining solutions to previously unsolvable problems. 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Prior to his appointment at Stellenbosch University, he has been at the University of Pretoria, Department of Computer Science (1998-2018), where he was appointed as South Africa Research Chair in Artifical Intelligence (2007-2018), the head of the Department of Computer Science (2008-2017), and Director of the Institute for Big Data and Data Science (2017-2018). 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He is a full professor of signal processing and pattern recognition and is head of the Signals and Communications Department at ULPGC, teaching from 2001 on subjects on signal processing and learning theory. His research lines are biometrics, biomedical signals and images, data mining, classification system, signal and image processing, machine learning, and environmental intelligence. He has researched in 52 international and Spanish research projects, some of them as head researcher. He is co-author of 4 books, co-editor of 27 proceedings books, guest editor for 8 JCR-ISI international journals, and up to 24 book chapters. He has over 450 papers published in international journals and conferences (81 of them indexed on JCR – ISI - Web of Science). He has published seven patents in the Spanish Patent and Trademark Office. He has been a supervisor on 8 Ph.D. theses (11 more are under supervision), and 130 master theses. He is the founder of The IEEE IWOBI conference series and the president of its Steering Committee, as well as the founder of both the InnoEducaTIC and APPIS conference series. He is an evaluator of project proposals for the European Union (H2020), Medical Research Council (MRC, UK), Spanish Government (ANECA, Spain), Research National Agency (ANR, France), DAAD (Germany), Argentinian Government, and the Colombian Institutions. He has been a reviewer in different indexed international journals (<70) and conferences (<250) since 2001. He has been a member of the IASTED Technical Committee on Image Processing from 2007 and a member of the IASTED Technical Committee on Artificial Intelligence and Expert Systems from 2011. \n\nHe has held the general chair position for the following: ACM-APPIS (2020, 2021), IEEE-IWOBI (2019, 2020 and 2020), A PPIS (2018, 2019), IEEE-IWOBI (2014, 2015, 2017, 2018), InnoEducaTIC (2014, 2017), IEEE-INES (2013), NoLISP (2011), JRBP (2012), and IEEE-ICCST (2005)\n\nHe is an associate editor of the Computational Intelligence and Neuroscience Journal (Hindawi – Q2 JCR-ISI). He was vice dean from 2004 to 2010 in the Higher Technical School of Telecommunication Engineers at ULPGC and the vice dean of Graduate and Postgraduate Studies from March 2013 to November 2017. He won the “Catedra Telefonica” Awards in Modality of Knowledge Transfer, 2017, 2018, and 2019 editions, and awards in Modality of COVID Research in 2020.\n\nPublic References:\nResearcher ID http://www.researcherid.com/rid/N-5967-2014\nORCID https://orcid.org/0000-0002-4621-2768 \nScopus Author ID https://www.scopus.com/authid/detail.uri?authorId=6602376272\nScholar Google https://scholar.google.es/citations?user=G1ks9nIAAAAJ&hl=en \nResearchGate https://www.researchgate.net/profile/Carlos_Travieso",institutionString:null,institution:{name:"University of Las Palmas de Gran Canaria",institutionURL:null,country:{name:"Spain"}}},editorTwo:null,editorThree:null},{id:"23",title:"Computational Neuroscience",coverUrl:"https://cdn.intechopen.com/series_topics/covers/23.jpg",isOpenForSubmission:!0,editor:{id:"14004",title:"Dr.",name:"Magnus",middleName:null,surname:"Johnsson",slug:"magnus-johnsson",fullName:"Magnus Johnsson",profilePictureURL:"https://mts.intechopen.com/storage/users/14004/images/system/14004.png",biography:"Dr Magnus Johnsson is a cross-disciplinary scientist, lecturer, scientific editor and AI/machine learning consultant from Sweden. \n\nHe is currently at Malmö University in Sweden, but also held positions at Lund University in Sweden and at Moscow Engineering Physics Institute. \nHe holds editorial positions at several international scientific journals and has served as a scientific editor for books and special journal issues. \nHis research interests are wide and include, but are not limited to, autonomous systems, computer modeling, artificial neural networks, artificial intelligence, cognitive neuroscience, cognitive robotics, cognitive architectures, cognitive aids and the philosophy of mind. \n\nDr. Johnsson has experience from working in the industry and he has a keen interest in the application of neural networks and artificial intelligence to fields like industry, finance, and medicine. \n\nWeb page: www.magnusjohnsson.se",institutionString:null,institution:{name:"Malmö University",institutionURL:null,country:{name:"Sweden"}}},editorTwo:null,editorThree:null},{id:"24",title:"Computer Vision",coverUrl:"https://cdn.intechopen.com/series_topics/covers/24.jpg",isOpenForSubmission:!0,editor:{id:"294154",title:"Prof.",name:"George",middleName:null,surname:"Papakostas",slug:"george-papakostas",fullName:"George Papakostas",profilePictureURL:"https://s3.us-east-1.amazonaws.com/intech-files/0030O00002hYaGbQAK/Profile_Picture_1624519712088",biography:"George A. Papakostas has received a diploma in Electrical and Computer Engineering in 1999 and the M.Sc. and Ph.D. degrees in Electrical and Computer Engineering in 2002 and 2007, respectively, from the Democritus University of Thrace (DUTH), Greece. Dr. Papakostas serves as a Tenured Full Professor at the Department of Computer Science, International Hellenic University, Greece. Dr. Papakostas has 10 years of experience in large-scale systems design as a senior software engineer and technical manager, and 20 years of research experience in the field of Artificial Intelligence. Currently, he is the Head of the “Visual Computing” division of HUman-MAchines INteraction Laboratory (HUMAIN-Lab) and the Director of the MPhil program “Advanced Technologies in Informatics and Computers” hosted by the Department of Computer Science, International Hellenic University. He has (co)authored more than 150 publications in indexed journals, international conferences and book chapters, 1 book (in Greek), 3 edited books, and 5 journal special issues. His publications have more than 2100 citations with h-index 27 (GoogleScholar). His research interests include computer/machine vision, machine learning, pattern recognition, computational intelligence. \nDr. Papakostas served as a reviewer in numerous journals, as a program\ncommittee member in international conferences and he is a member of the IAENG, MIR Labs, EUCogIII, INSTICC and the Technical Chamber of Greece (TEE).",institutionString:null,institution:{name:"International Hellenic University",institutionURL:null,country:{name:"Greece"}}},editorTwo:null,editorThree:null},{id:"25",title:"Evolutionary Computation",coverUrl:"https://cdn.intechopen.com/series_topics/covers/25.jpg",isOpenForSubmission:!0,editor:{id:"136112",title:"Dr.",name:"Sebastian",middleName:null,surname:"Ventura Soto",slug:"sebastian-ventura-soto",fullName:"Sebastian Ventura Soto",profilePictureURL:"https://mts.intechopen.com/storage/users/136112/images/system/136112.png",biography:"Sebastian Ventura is a Spanish researcher, a full professor with the Department of Computer Science and Numerical Analysis, University of Córdoba. Dr Ventura also holds the positions of Affiliated Professor at Virginia Commonwealth University (Richmond, USA) and Distinguished Adjunct Professor at King Abdulaziz University (Jeddah, Saudi Arabia). Additionally, he is deputy director of the Andalusian Research Institute in Data Science and Computational Intelligence (DaSCI) and heads the Knowledge Discovery and Intelligent Systems Research Laboratory. He has published more than ten books and over 300 articles in journals and scientific conferences. Currently, his work has received over 18,000 citations according to Google Scholar, including more than 2200 citations in 2020. In the last five years, he has published more than 60 papers in international journals indexed in the JCR (around 70% of them belonging to first quartile journals) and he has edited some Springer books “Supervised Descriptive Pattern Mining” (2018), “Multiple Instance Learning - Foundations and Algorithms” (2016), and “Pattern Mining with Evolutionary Algorithms” (2016). He has also been involved in more than 20 research projects supported by the Spanish and Andalusian governments and the European Union. He currently belongs to the editorial board of PeerJ Computer Science, Information Fusion and Engineering Applications of Artificial Intelligence journals, being also associate editor of Applied Computational Intelligence and Soft Computing and IEEE Transactions on Cybernetics. Finally, he is editor-in-chief of Progress in Artificial Intelligence. He is a Senior Member of the IEEE Computer, the IEEE Computational Intelligence, and the IEEE Systems, Man, and Cybernetics Societies, and the Association of Computing Machinery (ACM). Finally, his main research interests include data science, computational intelligence, and their applications.",institutionString:null,institution:{name:"University of Córdoba",institutionURL:null,country:{name:"Spain"}}},editorTwo:null,editorThree:null},{id:"26",title:"Machine Learning and Data Mining",coverUrl:"https://cdn.intechopen.com/series_topics/covers/26.jpg",isOpenForSubmission:!0,editor:{id:"24555",title:"Dr.",name:"Marco Antonio",middleName:null,surname:"Aceves Fernandez",slug:"marco-antonio-aceves-fernandez",fullName:"Marco Antonio Aceves Fernandez",profilePictureURL:"https://mts.intechopen.com/storage/users/24555/images/system/24555.jpg",biography:"Dr. Marco Antonio Aceves Fernandez obtained his B.Sc. (Eng.) in Telematics from the Universidad de Colima, Mexico. He obtained both his M.Sc. and Ph.D. from the University of Liverpool, England, in the field of Intelligent Systems. He is a full professor at the Universidad Autonoma de Queretaro, Mexico, and a member of the National System of Researchers (SNI) since 2009. Dr. Aceves Fernandez has published more than 80 research papers as well as a number of book chapters and congress papers. He has contributed in more than 20 funded research projects, both academic and industrial, in the area of artificial intelligence, ranging from environmental, biomedical, automotive, aviation, consumer, and robotics to other applications. He is also a honorary president at the National Association of Embedded Systems (AMESE), a senior member of the IEEE, and a board member of many institutions. His research interests include intelligent and embedded systems.",institutionString:"Universidad Autonoma de Queretaro",institution:{name:"Autonomous University of Queretaro",institutionURL:null,country:{name:"Mexico"}}},editorTwo:null,editorThree:null},{id:"27",title:"Multi-Agent Systems",coverUrl:"https://cdn.intechopen.com/series_topics/covers/27.jpg",isOpenForSubmission:!0,editor:{id:"148497",title:"Dr.",name:"Mehmet",middleName:"Emin",surname:"Aydin",slug:"mehmet-aydin",fullName:"Mehmet Aydin",profilePictureURL:"https://mts.intechopen.com/storage/users/148497/images/system/148497.jpg",biography:"Dr. Mehmet Emin Aydin is a Senior Lecturer with the Department of Computer Science and Creative Technology, the University of the West of England, Bristol, UK. His research interests include swarm intelligence, parallel and distributed metaheuristics, machine learning, intelligent agents and multi-agent systems, resource planning, scheduling and optimization, combinatorial optimization. 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She is now a lecturer at the University of Witwatersrand, South Africa, and a principal researcher at the Health Economics and Epidemiology Research Office (HE2RO), South Africa. Dr. Moolla holds a Ph.D. in Psychology with her research being focused on mental health and resilience. In her professional work capacity, her research has further expanded into the fields of early childhood development, mental health, the HIV and TB care cascades, as well as COVID. She is also a UNESCO-trained International Bioethics Facilitator.",institutionString:"University of the Witwatersrand",institution:{name:"University of the Witwatersrand",country:{name:"South Africa"}}},{id:"342152",title:"Dr.",name:"Santo",middleName:null,surname:"Grace Umesh",slug:"santo-grace-umesh",fullName:"Santo Grace Umesh",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/342152/images/16311_n.jpg",biography:null,institutionString:null,institution:{name:"SRM Dental College",country:{name:"India"}}},{id:"333647",title:"Dr.",name:"Shreya",middleName:null,surname:"Kishore",slug:"shreya-kishore",fullName:"Shreya Kishore",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/333647/images/14701_n.jpg",biography:"Dr. Shreya Kishore completed her Bachelor in Dental Surgery in Chettinad Dental College and Research Institute, Chennai, and her Master of Dental Surgery (Orthodontics) in Saveetha Dental College, Chennai. She is also Invisalign certified. She’s working as a Senior Lecturer in the Department of Orthodontics, SRM Dental College since November 2019. She is actively involved in teaching orthodontics to the undergraduates and the postgraduates. Her clinical research topics include new orthodontic brackets, fixed appliances and TADs. She’s published 4 articles in well renowned indexed journals and has a published patency of her own. Her private practice is currently limited to orthodontics and works as a consultant in various clinics.",institutionString:null,institution:{name:"SRM Dental College",country:{name:"India"}}},{id:"323731",title:"Prof.",name:"Deepak M.",middleName:"Macchindra",surname:"Vikhe",slug:"deepak-m.-vikhe",fullName:"Deepak M. Vikhe",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/323731/images/13613_n.jpg",biography:"Dr Deepak M.Vikhe .\n\n\t\n\tDr Deepak M.Vikhe , completed his Masters & PhD in Prosthodontics from Rural Dental College, Loni securing third rank in the Pravara Institute of Medical Sciences Deemed University. He was awarded Dr.G.C.DAS Memorial Award for Research on Implants at 39th IPS conference Dubai (U A E).He has two patents under his name. He has received Dr.Saraswati medal award for best research for implant study in 2017.He has received Fully funded scholarship to Spain ,university of Santiago de Compostela. He has completed fellowship in Implantlogy from Noble Biocare. \nHe has attended various conferences and CDE programmes and has national publications to his credit. His field of interest is in Implant supported prosthesis. Presently he is working as a associate professor in the Dept of Prosthodontics, Rural Dental College, Loni and maintains a successful private practice specialising in Implantology at Rahata.\n\nEmail: drdeepak_mvikhe@yahoo.com..................",institutionString:null,institution:{name:"Pravara Institute of Medical Sciences",country:{name:"India"}}},{id:"204110",title:"Dr.",name:"Ahmed A.",middleName:null,surname:"Madfa",slug:"ahmed-a.-madfa",fullName:"Ahmed A. Madfa",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/204110/images/system/204110.jpg",biography:"Dr. Madfa is currently Associate Professor of Endodontics at Thamar University and a visiting lecturer at Sana'a University and University of Sciences and Technology. He has more than 6 years of experience in teaching. His research interests include root canal morphology, functionally graded concept, dental biomaterials, epidemiology and dental education, biomimetic restoration, finite element analysis and endodontic regeneration. Dr. Madfa has numerous international publications, full articles, two patents, a book and a book chapter. Furthermore, he won 14 international scientific awards. Furthermore, he is involved in many academic activities ranging from editorial board member, reviewer for many international journals and postgraduate students' supervisor. Besides, I deliver many courses and training workshops at various scientific events. Dr. Madfa also regularly attends international conferences and holds administrative positions (Deputy Dean of the Faculty for Students’ & Academic Affairs and Deputy Head of Research Unit).",institutionString:"Thamar University",institution:null},{id:"210472",title:"Dr.",name:"Nermin",middleName:"Mohammed Ahmed",surname:"Yussif",slug:"nermin-yussif",fullName:"Nermin Yussif",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/210472/images/system/210472.jpg",biography:"Dr. Nermin Mohammed Ahmed Yussif is working at the Faculty of dentistry, University for October university for modern sciences and arts (MSA). Her areas of expertise include: periodontology, dental laserology, oral implantology, periodontal plastic surgeries, oral mesotherapy, nutrition, dental pharmacology. She is an editor and reviewer in numerous international journals.",institutionString:"MSA University",institution:null},{id:"204606",title:"Dr.",name:"Serdar",middleName:null,surname:"Gözler",slug:"serdar-gozler",fullName:"Serdar Gözler",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/204606/images/system/204606.jpeg",biography:"Dr. Serdar Gözler has completed his undergraduate studies at the Marmara University Faculty of Dentistry in 1978, followed by an assistantship in the Prosthesis Department of Dicle University Faculty of Dentistry. Starting his PhD work on non-resilient overdentures with Assoc. Prof. Hüsnü Yavuzyılmaz, he continued his studies with Prof. Dr. Gürbüz Öztürk of Istanbul University Faculty of Dentistry Department of Prosthodontics, this time on Gnatology. He attended training programs on occlusion, neurology, neurophysiology, EMG, radiology and biostatistics. In 1982, he presented his PhD thesis \\Gerber and Lauritzen Occlusion Analysis Techniques: Diagnosis Values,\\ at Istanbul University School of Dentistry, Department of Prosthodontics. As he was also working with Prof. Senih Çalıkkocaoğlu on The Physiology of Chewing at the same time, Gözler has written a chapter in Çalıkkocaoğlu\\'s book \\Complete Prostheses\\ entitled \\The Place of Neuromuscular Mechanism in Prosthetic Dentistry.\\ The book was published five times since by the Istanbul University Publications. Having presented in various conferences about occlusion analysis until 1998, Dr. Gözler has also decided to use the T-Scan II occlusion analysis method. Having been personally trained by Dr. Robert Kerstein on this method, Dr. Gözler has been lecturing on the T-Scan Occlusion Analysis Method in conferences both in Turkey and abroad. Dr. Gözler has various articles and presentations on Digital Occlusion Analysis methods. He is now Head of the TMD Clinic at Prosthodontic Department of Faculty of Dentistry , Istanbul Aydın University , Turkey.",institutionString:"Istanbul Aydin University",institution:{name:"Istanbul Aydın University",country:{name:"Turkey"}}},{id:"240870",title:"Ph.D.",name:"Alaa Eddin Omar",middleName:null,surname:"Al Ostwani",slug:"alaa-eddin-omar-al-ostwani",fullName:"Alaa Eddin Omar Al Ostwani",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/240870/images/system/240870.jpeg",biography:"Dr. Al Ostwani Alaa Eddin Omar received his Master in dentistry from Damascus University in 2010, and his Ph.D. in Pediatric Dentistry from Damascus University in 2014. Dr. Al Ostwani is an assistant professor and faculty member at IUST University since 2014. \nDuring his academic experience, he has received several awards including the scientific research award from the Union of Arab Universities, the Syrian gold medal and the international gold medal for invention and creativity. Dr. Al Ostwani is a Member of the International Association of Dental Traumatology and the Syrian Society for Research and Preventive Dentistry since 2017. He is also a Member of the Reviewer Board of International Journal of Dental Medicine (IJDM), and the Indian Journal of Conservative and Endodontics since 2016.",institutionString:"International University for Science and Technology.",institution:{name:"Islamic University of Science and Technology",country:{name:"India"}}},{id:"42847",title:"Dr.",name:"Belma",middleName:null,surname:"Işik Aslan",slug:"belma-isik-aslan",fullName:"Belma Işik Aslan",position:null,profilePictureURL:"https://mts.intechopen.com/storage/users/42847/images/system/42847.jpg",biography:"Dr. Belma IşIk Aslan was born in 1976 in Ankara-TURKEY. After graduating from TED Ankara College in 1994, she attended to Gazi University, Faculty of Dentistry in Ankara. She completed her PhD in orthodontic education at Gazi University between 1999-2005. Dr. Işık Aslan stayed at the Providence Hospital Craniofacial Institude and Reconstructive Surgery in Michigan, USA for three months as an observer. She worked as a specialist doctor at Gazi University, Dentistry Faculty, Department of Orthodontics between 2005-2014. She was appointed as associate professor in January, 2014 and as professor in 2021. Dr.